Levels of soluble endothelium-derived adhesion molecules in patients with sickle cell disease are associated with pulmonary hypertension, organ dysfunction, and mortality.
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Biomarkers and recent advances in the management and therapy of sickle cell diseaseHemorheological alterations in sickle cell anemia and their clinical consequences - The role of genetic modulators.Inhibition of cell adhesion by anti-P-selectin aptamer: a new potential therapeutic agent for sickle cell disease.Angiogenic and inflammatory markers of cardiopulmonary changes in children and adolescents with sickle cell disease.Cardiovascular changes in patients with non-severe Plasmodium vivax malariaPlacenta growth factor in sickle cell disease: association with hemolysis and inflammation.Heme oxygenase-1 gene delivery by Sleeping Beauty inhibits vascular stasis in a murine model of sickle cell disease.Increased levels of the inflammatory biomarker C-reactive protein at baseline are associated with childhood sickle cell vasocclusive crises.Endogenous nitric oxide synthase inhibitors in sickle cell disease: abnormal levels and correlations with pulmonary hypertension, desaturation, haemolysis, organ dysfunction and deathGMI-1070, a novel pan-selectin antagonist, reverses acute vascular occlusions in sickle cell mice.Deconstructing sickle cell disease: reappraisal of the role of hemolysis in the development of clinical subphenotypes.Pulmonary hypertension and cor pulmonale during severe acute chest syndrome in sickle cell disease.Severe sickle cell anemia is associated with increased plasma levels of TNF-R1 and VCAM-1Evolution of novel small-molecule therapeutics targeting sickle cell vasculopathy.Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.Peptide-directed highly selective targeting of pulmonary arterial hypertension.Vasculopathy and pulmonary hypertension in sickle cell disease.Pulmonary endothelial cell activation during experimental acute kidney injury.Non-congenital heart disease associated pediatric pulmonary arterial hypertensionIncreased pulmonary pressures and myocardial wall stress in children with severe malariaPleiotropic effects of intravascular haemolysis on vascular homeostasis.Relations of a marker of endothelial activation (s-VCAM) to function and mortality in community-dwelling older adults.Lipid levels in sickle-cell disease associated with haemolytic severity, vascular dysfunction and pulmonary hypertensionNitric oxide and arginine dysregulation: a novel pathway to pulmonary hypertension in hemolytic disordersPriapism in sickle-cell disease: a hematologist's perspective.Elevated Steady State WBC and Platelet Counts Are Associated with Frequent Emergency Room Use in Adults with Sickle Cell Anemia.Lactate dehydrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease.Platelet activation in patients with sickle disease, hemolysis-associated pulmonary hypertension, and nitric oxide scavenging by cell-free hemoglobinGenetic diminution of circulating prothrombin ameliorates multiorgan pathologies in sickle cell disease miceLongitudinal Analysis of Patient Specific Predictors for Mortality in Sickle Cell Disease.Atorvastatin reduces serum cholesterol and triglycerides with limited improvement in vascular function in adults with sickle cell anemia.Erythrocyte glutamine depletion, altered redox environment, and pulmonary hypertension in sickle cell disease.Cerebrovascular disease associated with sickle cell pulmonary hypertension.The acute phase inflammatory response to maximal exercise testing in children and young adults with sickle cell anaemia.Beyond hydroxyurea: new and old drugs in the pipeline for sickle cell disease.Vascular dysfunction in a murine model of severe hemolysis.Nonhematopoietic Nrf2 dominantly impedes adult progression of sickle cell anemia in mice.Hospital admission for acute painful episode following methacholine challenge in an adolescent with sickle cell disease.Endothelial Nitric Oxide Synthase (-786T>C) and Endothelin-1 (5665G>T) Gene Polymorphisms as Vascular Dysfunction Risk Factors in Sickle Cell Anemia.Longitudinal study of echocardiography-derived tricuspid regurgitant jet velocity in sickle cell disease
P2860
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P2860
Levels of soluble endothelium-derived adhesion molecules in patients with sickle cell disease are associated with pulmonary hypertension, organ dysfunction, and mortality.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@ast
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@en
type
label
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@ast
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@en
prefLabel
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@ast
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@en
P2093
P2860
P1476
Levels of soluble endothelium- ...... an dysfunction, and mortality.
@en
P2093
James S Nichols
Lori A Hunter
Marie-Luise Brennan
Sabrina Martyr
Stanley L Hazen
William C Blackwelder
Wynona A Coles
P2860
P304
P356
10.1111/J.1365-2141.2005.05701.X
P407
P577
2005-09-01T00:00:00Z