Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
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Indoleamine 2,3 Dioxygenase as a Potential Therapeutic Target in Huntington's DiseaseHigh Times for Painful Blues: The Endocannabinoid System in Pain-Depression ComorbidityGlia in the cytokine-mediated onset of depression: fine tuning the immune responseIndoleamine 2,3 dioxygenase in intestinal diseaseChronic pain assessment from bench to bedside: lessons along the translation continuumVirus Infections Incite Pain Hypersensitivity by Inducing Indoleamine 2,3 DioxygenaseMicrobiota alteration is associated with the development of stress-induced despair behaviorDepression with pain co morbidity effect on quality of life among HIV positive patients in Uganda: a cross sectional study.Alterations in the inflammatory cytokines and brain-derived neurotrophic factor contribute to depression-like phenotype after spared nerve injury: improvement by ketamine.Spared Nerve Injury Increases the Expression of Microglia M1 Markers in the Prefrontal Cortex of Rats and Provokes Depression-Like Behaviors.Constitutive IDO expression in human cancer is sustained by an autocrine signaling loop involving IL-6, STAT3 and the AHR.The antidepressant-like effect of trans-astaxanthin involves the serotonergic system.Epstein-Barr virus infection induces indoleamine 2,3-dioxygenase expression in human monocyte-derived macrophages through p38/mitogen-activated protein kinase and NF-κB pathways: impairment in T cell functions.Monoamine oxidase A upregulated by chronic intermittent hypoxia activates indoleamine 2,3-dioxygenase and neurodegeneration.Activation of the STING adaptor attenuates experimental autoimmune encephalitis.The role of brain-derived neurotrophic factor in comorbid depression: possible linkage with steroid hormones, cytokines, and nutritionIndoleamine 2,3 dioxygenase and metabolic control of immune responses.AMPAkines have novel analgesic properties in rat models of persistent neuropathic and inflammatory pain.Pain and depression comorbidity: a preclinical perspective.Human osteoclasts are inducible immunosuppressive cells in response to T cell-derived IFN-γ and CD40 ligand in vitroFluoxetine prevents the development of depressive-like behavior in a mouse model of cancer related fatigueSynergistic effects of celecoxib and bupropion in a model of chronic inflammation-related depression in mice.Increased levels of IL-6 in the cerebrospinal fluid of patients with chronic schizophrenia--significance for activation of the kynurenine pathway.Neuroplasticity underlying the comorbidity of pain and depressionActivation of corticostriatal circuitry relieves chronic neuropathic pain.Peripheral indoleamine 2,3-dioxygenase 1 is required for comorbid depression-like behavior but does not contribute to neuropathic pain in mice.Putative neuroprotective and neurotoxic kynurenine pathway metabolites are associated with hippocampal and amygdalar volumes in subjects with major depressive disorderCost-utility and biological underpinnings of Mindfulness-Based Stress Reduction (MBSR) versus a psychoeducational programme (FibroQoL) for fibromyalgia: a 12-month randomised controlled trial (EUDAIMON study).Persistent pain alters AMPA receptor subunit levels in the nucleus accumbensDifferential Transcriptome Networks between IDO1-Knockout and Wild-Type Mice in Brain Microglia and Macrophages.M30 Antagonizes Indoleamine 2,3-Dioxygenase Activation and Neurodegeneration Induced by Corticosterone in the HippocampusCurrent challenges in translational pain research.Persistent neuropathic pain increases synaptic GluA1 subunit levels in core and shell subregions of the nucleus accumbensIbuprofen ameliorates fatigue- and depressive-like behavior in tumor-bearing mice.Noncanonical NF-κB activation mediates STAT3-stimulated IDO upregulation in myeloid-derived suppressor cells in breast cancerA dialogue between the immune system and brain, spoken in the language of serotonin.Electroconvulsive therapy suppresses the neurotoxic branch of the kynurenine pathway in treatment-resistant depressed patientsThe rapid antidepressant effect of ketamine in rats is associated with down-regulation of pro-inflammatory cytokines in the hippocampusGlucocorticoid Receptors, Brain-Derived Neurotrophic Factor, Serotonin and Dopamine Neurotransmission are Associated with Interferon-Induced Depression.Tryptophan-kynurenine pathway is dysregulated in inflammation, and immune activation.
P2860
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P2860
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@ast
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@en
type
label
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@ast
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@en
prefLabel
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@ast
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@en
P2093
P2860
P356
P1476
Brain indoleamine 2,3-dioxygenase contributes to the comorbidity of pain and depression
@en
P2093
Backil Sung
Gabriel Rusanescu
Hyangin Kim
Jianren Mao
Liling Yang
Michael F McCabe
Shuxing Wang
Yinghong Tian
P2860
P304
P356
10.1172/JCI61884
P407
P577
2012-07-02T00:00:00Z