Deletion of TLR5 results in spontaneous colitis in mice - Wikidata - awgldk - TriplyDB

Deletion of TLR5 results in spontaneous colitis in mice

Deletion of TLR5 results in spontaneous colitis in mice

http://www.wikidata.org/entity/Q36138027

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Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human genetics Enteric infection meets intestinal function: how bacterial pathogens cause diarrhoea Unexpected protective role for Toll-like receptor 3 in the arterial wall Mucosal Interactions between Genetics, Diet, and Microbiome in Inflammatory Bowel Disease Use of Metatranscriptomics in Microbiome Research Immunity and Tolerance Induced by Intestinal Mucosal Dendritic Cells Genetically engineered mouse models for studying inflammatory bowel disease Gut microbes and host physiology: what happens when you host billions of guests?Exploring host-microbiota interactions in animal models and humans Multiple hits, including oxidative stress, as pathogenesis and treatment target in non-alcoholic steatohepatitis (NASH)Role of commensal gut bacteria in inflammatory bowel diseases The role of innate immunity receptors in the pathogenesis of inflammatory bowel disease Gut Microbiome and Obesity: A Plausible Explanation for Obesity Overexpression of Ste20-related proline/alanine-rich kinase exacerbates experimental colitis in mice Changes in intestinal Toll-like receptors and cytokines precede histological injury in a rat model of necrotizing enterocolitis Reptile Toll-like receptor 5 unveils adaptive evolution of bacterial flagellin recognition TLR5 risk-associated haplotype for canine inflammatory bowel disease confers hyper-responsiveness to flagellin Polymorphisms in the TLR4 and TLR5 gene are significantly associated with inflammatory bowel disease in German shepherd dogs Metabolic syndrome and altered gut microbiota in mice lacking Toll-like receptor 5 Inflammatory bowel disease Inflammatory bowel disease A2B adenosine receptor gene deletion attenuates murine colitis.Enteric commensal bacteria potentiate epithelial restitution via reactive oxygen species-mediated inactivation of focal adhesion kinase phosphatases.Epithelial IL-23R Signaling Licenses Protective IL-22 Responses in Intestinal Inflammation Patterns and scales in gastrointestinal microbial ecology.The induction of colitis and ileitis in mice is associated with marked increases in intestinal concentrations of stimulants of TLRs 2, 4, and 5 Interactions between the host innate immune system and microbes in inflammatory bowel disease.UNC93B1 is essential for the plasma membrane localization and signaling of Toll-like receptor 5 Pathogen recognition receptors, cancer and inflammation in the gut.Microbial control of regulatory and effector T cell responses in the gut.Uncovering Pathogenic Mechanisms of Inflammatory Bowel Disease Using Mouse Models of Crohn's Disease-Like Ileitis: What is the Right Model?Maternal obesity induces gut inflammation and impairs gut epithelial barrier function in nonobese diabetic mice.Serum miRNA signature diagnoses and discriminates murine colitis subtypes and predicts ulcerative colitis in humans.Molecular mechanisms and new treatment strategies for non-alcoholic steatohepatitis (NASH)Genetic and Chemical Models of Colorectal Cancer in Mice.Role of gut microbiota and Toll-like receptors in nonalcoholic fatty liver disease The DNA Sensor AIM2 Maintains Intestinal Homeostasis via Regulation of Epithelial Antimicrobial Host Defense.Evaluation of Toll-like, chemokine, and integrin receptors on monocytes and neutrophils from peripheral blood of septic patients and their correlation with clinical outcomes.AIEC pathobiont instigates chronic colitis in susceptible hosts by altering microbiota composition.TLR5-deficient mice lack basal inflammatory and metabolic defects but exhibit impaired CD4 T cell responses to a flagellated pathogen

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P2860

Deletion of TLR5 results in spontaneous colitis in mice

http://www.wikidata.org/entity/Q36138027

description

2007 nî lūn-bûn

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name

Deletion of TLR5 results in spontaneous colitis in mice

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Deletion of TLR5 results in spontaneous colitis in mice

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Deletion of TLR5 results in spontaneous colitis in mice

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Deletion of TLR5 results in spontaneous colitis in mice

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Deletion of TLR5 results in spontaneous colitis in mice

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Deletion of TLR5 results in spontaneous colitis in mice

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Journal of Clinical Investigation

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Deletion of TLR5 results in spontaneous colitis in mice

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P2093

Amrita Kumar

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Andrew S Neish

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Andrew T Gewirtz

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Catherine J Sanders

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Ifor R Williams

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Jesse D Aitken

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Matam Vijay-Kumar

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Rebekah T Taylor

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Satoshi Uematsu

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Shanthi V Sitaraman

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P2860

Angiogenins: a new class of microbicidal proteins involved in innate immunity

Maintenance of colonic homeostasis by distinctive apical TLR9 signalling in intestinal epithelial cells

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

Cutting edge: bacterial flagellin activates basolaterally expressed TLR5 to induce epithelial proinflammatory gene expression

Toll-like receptor 2 enhances ZO-1-associated intestinal epithelial barrier integrity via protein kinase C

NOD2 is a negative regulator of Toll-like receptor 2-mediated T helper type 1 responses

Cytosolic flagellin requires Ipaf for activation of caspase-1 and interleukin 1beta in salmonella-infected macrophages

Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis

Interleukin-10-deficient mice develop chronic enterocolitis

Rapid development of colitis in NSAID-treated IL-10-deficient mice.

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3909-3921

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12

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117

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18008007

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Toll-like receptor

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2075480

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