Symptomatic response to antiarrhythmic drug therapy is modulated by a common single nucleotide polymorphism in atrial fibrillation. - Wikidata - awgldk - TriplyDB

Symptomatic response to antiarrhythmic drug therapy is modulated by a common single nucleotide polymorphism in atrial fibrillation.

Symptomatic response to antiarrhythmic drug therapy is modulated by a common single nucleotide polymorphism in atrial fibrillation.

http://www.wikidata.org/entity/Q36139922

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Cardiovascular pharmacogenomics: current status and future directions Emerging directions in the genetics of atrial fibrillation Highlights of the year in JACC 2012 Genome-wide association studies of late-onset cardiovascular disease The Genetic Basis of Coronary Artery Disease and Atrial Fibrillation: A Search for Disease Mechanisms and Therapeutic Targets Open access integrated therapeutic and diagnostic platforms for personalized cardiovascular medicine Severity of obstructive sleep apnea influences the effect of genotype on response to anti-arrhythmic drug therapy for atrial fibrillation.PITX2-dependent gene regulation in atrial fibrillation and rhythm control.A genome-wide association study to identify genomic modulators of rate control therapy in patients with atrial fibrillation G protein-coupled receptor kinase 5 gene polymorphisms are associated with postoperative atrial fibrillation after coronary artery bypass grafting in patients receiving β-blockers The role of transcription factors in atrial fibrillation Korean Atrial Fibrillation (AF) Network: Genetic Variants for AF Do Not Predict Ablation Success The Role of Pharmacogenetics in Atrial Fibrillation Therapeutics: Is Personalized Therapy in Sight?Gene-guided therapy for catheter-ablation of atrial fibrillation: are we there yet?The value of basic research insights into atrial fibrillation mechanisms as a guide to therapeutic innovation: a critical analysis Common variation in atrial fibrillation: navigating the path from genetic association to mechanism.Common atrial fibrillation risk alleles at 4q25 predict recurrence after catheter-based atrial fibrillation ablation Evaluating the HATCH score for predicting progression to sustained atrial fibrillation in ED patients with new atrial fibrillation.Genetic mechanisms of atrial fibrillation: impact on response to treatment Impact of genetic variants on the upstream efficacy of renin-angiotensin system inhibitors for the prevention of atrial fibrillation.Common genetic polymorphism at 4q25 locus predicts atrial fibrillation recurrence after successful cardioversion Pleiotropic effects of antiarrhythmic agents: dronedarone in the treatment of atrial fibrillation Gene signatures of postoperative atrial fibrillation in atrial tissue after coronary artery bypass grafting surgery in patients receiving β-blockers.Paired-like homeodomain 2: a novel therapeutic target for atrial fibrillation?A common angiotensin-converting enzyme polymorphism and preoperative angiotensin-converting enzyme inhibition modify risk of tachyarrhythmias after congenital heart surgery.Genetic determinants of response to cardiovascular drugs.New directions in antiarrhythmic drug therapy for atrial fibrillation.Personalized medicine to treat arrhythmias.Pharmacokinetic and pharmacodynamic profile of dronedarone , a new antiarrhythmic agent for the treatment of atrial fibrillation.Expert consensus document: Defining the major health modifiers causing atrial fibrillation: a roadmap to underpin personalized prevention and treatment.Genetic Discoveries in Atrial Fibrillation and Implications for Clinical Practice.Tailored treatment strategies: a new approach for modern management of atrial fibrillation.Atrial Fibrillation Genetics: Is There a Practical Clinical Value Now or in the Future?Genotype influence in responses to therapy for atrial fibrillation.Association of the angiotensinogen M235T polymorphism with recurrence after catheter ablation of acquired atrial fibrillation.The burden of proof: The current state of atrial fibrillation prevention and treatment trials.Investigational antiarrhythmic agents: promising drugs in early clinical development.Korean atrial fibrillation network genome-wide association study for early-onset atrial fibrillation identifies novel susceptibility loci.Can polymorphisms predict response to antiarrhythmic drugs in atrial fibrillation?Dronedarone: is it time to turn it down?

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Symptomatic response to antiarrhythmic drug therapy is modulated by a common single nucleotide polymorphism in atrial fibrillation.

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Dan Roden

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Gayle Kucera

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Joseph Vaglio

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Raafia Muhammad

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Shane Rowan

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P2860

A genome-wide association study identifies novel and functionally related susceptibility Loci for Kawasaki disease

Somatic mutations in the connexin 40 gene (GJA5) in atrial fibrillation

Augmented potassium current is a shared phenotype for two genetic defects associated with familial atrial fibrillation

Mutation in nuclear pore component NUP155 leads to atrial fibrillation and early sudden cardiac death

KCNQ1 gain-of-function mutation in familial atrial fibrillation

Frequent somatic mutations of the transcription factor ATBF1 in human prostate cancer

Kv1.5 channelopathy due to KCNA5 loss-of-function mutation causes human atrial fibrillation

Paroxysmal atrial fibrillation: a disorder of autonomic tone?

Pitx2c and Nkx2-5 are required for the formation and identity of the pulmonary myocardium

Spontaneous initiation of atrial fibrillation by ectopic beats originating in the pulmonary veins

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