Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
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Neuroprotective actions of PIKE-L by inhibition of SET proteolytic degradation by asparagine endopeptidaseIs protein phosphatase inhibition responsible for the toxic effects of okadaic Acid in animals?Kainic Acid-Induced Excitotoxicity Experimental Model: Protective Merits of Natural Products and Plant ExtractsTau clearance mechanisms and their possible role in the pathogenesis of Alzheimer diseaseInvolvement of autophagy in hypoxic-excitotoxic neuronal deathMyoinositol Attenuates the Cell Loss and Biochemical Changes Induced by Kainic Acid Status Epilepticus.Knockdown and overexpression of NR1 modulates NMDA receptor function.Altered mRNA editing and expression of ionotropic glutamate receptors after kainic acid exposure in cyclooxygenase-2 deficient mice.Activin A is essential for neurogenesis following neurodegenerationActivation of Retinoid X Receptor increases dopamine cell survival in models for Parkinson's disease.Comparative analysis of cis-regulation following stroke and seizures in subspaces of conserved eigensystems.Marked calpastatin (CAST) depletion in Alzheimer's disease accelerates cytoskeleton disruption and neurodegeneration: neuroprotection by CAST overexpression.Dysregulation of tau phosphorylation in mouse brain during excitotoxic damageA Decade of Research on TLR2 Discovering Its Pivotal Role in Glial Activation and Neuroinflammation in Neurodegenerative Diseases.Ginseng Rb fraction protects glia, neurons and cognitive function in a rat model of neurodegenerationInsulin blocks glutamate-induced neurotoxicity in differentiated SH-SY5Y neuronal cellsFosb gene products contribute to excitotoxic microglial activation by regulating the expression of complement C5a receptors in microgliaSpatial memory is enhanced in long-living Ames dwarf mice and maintained following kainic acid induced neurodegenerationA new generation fatty acid amide hydrolase inhibitor protects against kainate-induced excitotoxicity.The prostaglandin EP1 receptor potentiates kainate receptor activation via a protein kinase C pathway and exacerbates status epilepticusMechanism for the protective effect of resveratrol against oxidative stress-induced neuronal death.Kainic acid-induced neurodegenerative model: potentials and limitationsMicroglial Toll-like receptor 2 contributes to kainic acid-induced glial activation and hippocampal neuronal cell death.Selective androgen receptor modulator RAD140 is neuroprotective in cultured neurons and kainate-lesioned male rats.A New Transgenic Mouse Model for Studying the Neurotoxicity of Spermine Oxidase Dosage in the Response to Excitotoxic Injury.NMDA-induced seizure intensity is enhanced in COX-2 deficient mice.Overexpression of C-terminal fragment of glutamate receptor 6 prevents neuronal injury in kainate-induced seizure via disassembly of GluR6-PSD-95-MLK3 signaling module.Smaller cornu ammonis 2-3/dentate gyrus volumes and elevated cortisol in multiple sclerosis patients with depressive symptoms.Adiponectin protects rat hippocampal neurons against excitotoxicity.Kainic Acid-induced neurotoxicity: targeting glial responses and glia-derived cytokines.Neuroprotection requires the functions of the RNA-binding protein HuR.Prosaposin overexpression following kainic acid-induced neurotoxicity.Bioluminescence imaging of Smad signaling in living mice shows correlation with excitotoxic neurodegeneration.Naringin Attenuates Autophagic Stress and Neuroinflammation in Kainic Acid-Treated Hippocampus In Vivo.HIV-Tat Induces the Nrf2/ARE Pathway through NMDA Receptor-Elicited Spermine Oxidase Activation in Human Neuroblastoma Cells.MicroRNA-34b mediates hippocampal astrocyte apoptosis in a rat model of recurrent seizuresThe Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System DiseasesActivating mitochondrial function and haemoglobin expression with EH-201, an inducer of erythropoietin in neuronal cells, reverses memory impairmentIL-1RAcPb signaling regulates adaptive mechanisms in neurons that promote their long-term survival following excitotoxic insults.Mechanisms of tau and Aβ-induced excitotoxicity.
P2860
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P2860
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@ast
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@en
type
label
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@ast
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@en
prefLabel
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@ast
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@en
P2093
P356
P1476
Kainic acid-mediated excitotoxicity as a model for neurodegeneration.
@en
P2093
Agnes Simonyi
Albert Y Sun
Grace Y Sun
P2888
P356
10.1385/MN:31:1-3:003
P577
2005-01-01T00:00:00Z
P6179
1035197908