Clonality and altered behavior of endothelial cells from hemangiomas. - Wikidata - awgldk - TriplyDB

Clonality and altered behavior of endothelial cells from hemangiomas.

Clonality and altered behavior of endothelial cells from hemangiomas.

http://www.wikidata.org/entity/Q36163041

about

Down-regulation of mir-424 contributes to the abnormal angiogenesis via MEK1 and cyclin E1 in senile hemangioma: its implications to therapy Evidence by molecular profiling for a placental origin of infantile hemangioma.Modeling human endothelial cell transformation in vascular neoplasias Somatic mutation of vascular endothelial growth factor receptors in juvenile hemangioma Endothelial cell heterogeneity Mesenchymal stem cells and adipogenesis in hemangioma involution.Hemangiomas of infancy: clinical and biological characteristics.Identification of signaling systems in proliferating and involuting phase infantile hemangiomas by genome-wide transcriptional profiling Human pulmonary valve progenitor cells exhibit endothelial/mesenchymal plasticity in response to vascular endothelial growth factor-A and transforming growth factor-beta2.Vasculogenesis in infantile hemangioma.Human pulmonary valve endothelial cells express functional adhesion molecules for leukocytes Corticosteroid suppression of VEGF-A in infantile hemangioma-derived stem cells A switch in Notch gene expression parallels stem cell to endothelial transition in infantile hemangioma.Beta-blockers as therapy for infantile hemangiomas Effects of propranolol and isoproterenol on infantile hemangioma endothelial cells in vitro Signaling mechanisms in infantile hemangioma Insulin-like growth factor 2 and potential regulators of hemangioma growth and involution identified by large-scale expression analysis Nox-4-dependent nuclear H2O2 drives DNA oxidation resulting in 8-OHdG as urinary biomarker and hemangioendothelioma formation Pathogenesis of hemangioma Rapamycin inhibits proliferation of hemangioma endothelial cells by reducing HIF-1-dependent expression of VEGF.Infantile hemangioma is a proliferation of LYVE-1-negative blood endothelial cells without lymphatic competence.Pathogenesis of vascular anomalies.Imaging and percutaneous treatment of vascular anomalies.Glucose transporter 1-positive endothelial cells in infantile hemangioma exhibit features of facultative stem cells.One Possible Mechanism of Pulsed Dye Laser Treatment on Infantile Hemangioma: Induction of Endothelial Apoptosis and Serum vascular endothelial growth factor (VEGF) Level Changes Myeloid cells in infantile hemangioma.Conversion of vascular endothelial cells into multipotent stem-like cells.Prospective study of infantile haemangiomas: incidence, clinical characteristics and association with placental anomalies.Transforming growth factor-β2 promotes Snail-mediated endothelial-mesenchymal transition through convergence of Smad-dependent and Smad-independent signalling.Increased Tie2 expression, enhanced response to angiopoietin-1, and dysregulated angiopoietin-2 expression in hemangioma-derived endothelial cells.Endothelial progenitor cells from infantile hemangioma and umbilical cord blood display unique cellular responses to endostatin.Propranolol is an effective treatment for airway haemangiomas: a critical analysis and meta-analysis of published interventional studies Propranolol treatment of infantile hemangioma endothelial cells: A molecular analysis E-selectin mediates stem cell adhesion and formation of blood vessels in a murine model of infantile hemangioma The role of β-adrenergic receptor signaling in the proliferation of hemangioma-derived endothelial cells Multipotential stem cells recapitulate human infantile hemangioma in immunodeficient mice.Gene expression analysis reveals marked differences in the transcriptome of infantile hemangioma endothelial cells compared to normal dermal microvascular endothelial cells.Altered ratios of pro- and anti-angiogenic VEGF-A variants and pericyte expression of DLL4 disrupt vascular maturation in infantile haemangioma Suppressed NFAT-dependent VEGFR1 expression and constitutive VEGFR2 signaling in infantile hemangioma Pathogenesis of infantile haemangioma.

P2860

Q21091084-326E68BC-86F7-47FE-8992-E4E68C98C7F7 Q24535837-DE80B0C9-631B-419D-A134-AE8A5042C5D7 Q26860203-91C93B31-7B41-4CD8-AE97-176644BD704A Q28216461-3DFC7D93-E00F-4A2D-9DA0-8D5A252EA666 Q28740296-6D4A053B-5F04-4947-9428-5C32FF95757F Q30159926-A731B82B-8D6F-482B-B175-702B7DDC82F0 Q33228482-C259CDC6-A6A1-403F-A644-93B12316B363 Q33427340-51BE6898-8E01-47EC-AD56-58A2CC4C75EC Q33606662-1985436B-3188-42D5-9231-4013CE1A4CB5 Q33607327-DC3C91E4-253B-4EF7-A2C7-D456E1DB7E60 Q33607348-38466F65-4FFB-4D71-9C8E-98B3F295FE20 Q33754759-3647AA07-9C01-44FD-B4E3-B85FE3D93A97 Q33756156-AEC9A189-79A3-42F1-9D80-37D1C993D1EE Q33832594-A445A0C4-1D25-4D84-BEAE-0662FF6169F2 Q33837509-EEA72FCB-78C9-4433-A5CF-C7C52FFF3298 Q33954701-90F4CE78-5328-455D-91A8-7E4601E52EC1 Q34064067-2D2DC63B-D200-408C-B996-A9ABCAC3C31E Q34110134-5E07A2E5-8B00-4C97-B8E2-9B67BC61534A Q34187184-E806AF36-0612-4DBC-8FB1-5AD0541A1D05 Q34382663-090D2B5C-3E6F-43AA-B174-AA7C06C0E07D Q34486352-E55AC6AE-4BFE-4789-BD3A-87B260170BAC Q34540738-9AF88DF3-92B9-477A-A5E2-FD93C2D9452B Q34563659-7F716844-3705-4E81-8E7E-7B4ADCDDC0C6 Q34734440-CB4A7129-9992-434A-88D0-04F53E0B852A Q34927424-10B9300C-F6AB-42C4-A157-D502ACF94B8C Q35087931-7DD67675-8FC4-4404-9361-579A0BABA4BF Q35532554-B03D5FFA-8B4A-4E2E-8E61-0DE0EE0DDD42 Q35539585-A2F757AF-EAAC-4CFC-AAA7-DB60416D57A3 Q35688456-91FDC6E1-9394-4BA3-80C7-C16DC216971D Q35747092-6EAEDDCF-50D3-41CC-B136-8AC0D655ABED Q35849864-34314000-9B81-4943-9B3E-32B51BE81AD8 Q36310708-7B18A765-7A72-4C12-9C2E-429EE0DE833E Q36409111-17A6A92D-3756-4E23-9DF7-95E761685E57 Q36433043-85E00723-3F15-478A-BE2E-9AD9EF0E81F1 Q36614361-A8FBB157-922E-4882-9671-6118793B10CA Q36700275-5743B498-0541-4C0B-A7C1-B7DC780DBC3B Q36849376-F6708832-4839-453E-9338-9DE0476E1A4B Q36908121-9F2B3E0E-1701-4EC0-BE7B-74C1369FF3D7 Q36995485-6DCA43CA-3DF1-4E72-B65A-ED10E11E00C0 Q37001304-16B68201-7D70-4280-AC94-D9EABFC4382C

P2860

Clonality and altered behavior of endothelial cells from hemangiomas.

http://www.wikidata.org/entity/Q36163041

description

2001 nî lūn-bûn

@nan

2001年の論文

@ja

2001年論文

@yue

2001年論文

@zh-hant

2001年論文

@zh-hk

2001年論文

@zh-mo

2001年論文

@zh-tw

2001年论文

@wuu

2001年论文

@zh

2001年论文

@zh-cn

name

Clonality and altered behavior of endothelial cells from hemangiomas.

@ast

Clonality and altered behavior of endothelial cells from hemangiomas.

@en

type

label

Clonality and altered behavior of endothelial cells from hemangiomas.

@ast

Clonality and altered behavior of endothelial cells from hemangiomas.

@en

prefLabel

Clonality and altered behavior of endothelial cells from hemangiomas.

@ast

Clonality and altered behavior of endothelial cells from hemangiomas.

@en

P1433

Q36163041-100F7341-7485-4137-93C2-BEF7B202AC4B

P1476

Q36163041-3337D113-B7B2-4EA4-A450-45248CB193BA

P2093

Q36163041-033B2D59-85BE-42CC-96A4-AC2A1060BBBD

Q36163041-17155643-0EB1-4933-8274-CBD8AC74F50C

Q36163041-18B8E3B3-F158-420F-9C9B-6B7CB17964BC

Q36163041-A66C501F-29EB-4FA5-92AA-5CE624C6C310

Q36163041-D05F82CE-72A3-463A-BCE2-E97B6158B42E

P2860

Q36163041-1DC62236-00FB-491D-A666-59460C0B941D

Q36163041-1EBAD86F-EFBD-4992-B1DC-FF92CCB1FECA

Q36163041-2998E54C-2499-4CCD-93A0-EA633DB8D749

Q36163041-320E8F13-A52E-447C-BF40-7958E75A11BB

Q36163041-375BB59B-1A66-4812-AA28-AACA0A65960C

Q36163041-38E5FEC6-94A0-4808-A923-EEDA078A177D

Q36163041-3D584D8B-BE7D-4912-9CAB-34A8A4882F62

Q36163041-477F0E27-7813-4390-951B-81C5ED37895C

Q36163041-4DD83926-196B-44D2-9452-CF5A63D3E0BC

Q36163041-68851A0F-81EF-457F-A874-D656DF6C70BE

P304

Q36163041-3E895F0D-F285-4C57-A012-1A2D9AE645FC

P31

Q36163041-8CFE6DF7-E6D7-4B84-8CBB-3A6A5CC1E958

P356

Q36163041-8F3CC390-1465-4B84-8ECD-F3A85A90ECE1

P407

Q36163041-DA841E88-E5B9-493E-BC2C-CA3900B09C25

P433

Q36163041-82DB5C23-FE51-4BD3-AEF5-4C0A544F529E

P478

Q36163041-AD172BE9-A1AB-47C6-B3E2-54B51AE84B30

P50

Q36163041-AA06D0F9-E4AE-4E61-8A6D-1EFF80DFF649

P577

Q36163041-A2074278-113A-46A2-B6DD-C4BCB1F615E8

P5875

Q36163041-39A43AA4-E672-4418-A3D2-B1DCF9180109

P698

Q36163041-2A5B540D-E114-48B2-BEEA-EB7C9C927B6C

P921

Q36163041-4A93678C-73FD-4D73-90EC-60C0FF70D7C0

P932

Q36163041-1395DD62-AB70-4DCA-B593-C0DFF73EF50E

P356

P1433

Journal of Clinical Investigation

P1476

Clonality and altered behavior of endothelial cells from hemangiomas

@en

P2093

B R Olsen

http://www.w3.org/2001/XMLSchema#string

E Boye

http://www.w3.org/2001/XMLSchema#string

G Paranya

http://www.w3.org/2001/XMLSchema#string

J B Mulliken

http://www.w3.org/2001/XMLSchema#string

Y Yu

http://www.w3.org/2001/XMLSchema#string

P2860

GLUT1: a newly discovered immunohistochemical marker for juvenile hemangiomas

Current status of antiangiogenic factors.

Methylation of HpaII and HhaI sites near the polymorphic CAG repeat in the human androgen-receptor gene correlates with X chromosome inactivation.

Genetic variation at five trimeric and tetrameric tandem repeat loci in four human population groups.

Monoclonality of smooth muscle cells in human atherosclerosis.

Vascular endothelial growth factor confers a growth advantage in vitro and in vivo to stromal cells cultured from neonatal hemangiomas.

Clonal architecture of normal and atherosclerotic aorta: implications for atherogenesis and vascular development.

Disseminated peritoneal leiomyomatosis. Clonality analysis by X chromosome inactivation and cytogenetics of a clinically benign smooth muscle proliferation.

E-selectin is present in proliferating endothelial cells in human hemangiomas.

P304

745-752

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1172/JCI11432

http://www.w3.org/2001/XMLSchema#string

P407

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

107

http://www.w3.org/2001/XMLSchema#string

P50

P577

2001-03-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

24590755

http://www.w3.org/2001/XMLSchema#string

P698

11254674

http://www.w3.org/2001/XMLSchema#string

P921

P932

208946

http://www.w3.org/2001/XMLSchema#string