Deletion of interleukin (IL)-12p35 induces liver fibrosis in dominant-negative TGFβ receptor type II mice. - Wikidata - awgldk - TriplyDB

Deletion of interleukin (IL)-12p35 induces liver fibrosis in dominant-negative TGFβ receptor type II mice.

Deletion of interleukin (IL)-12p35 induces liver fibrosis in dominant-negative TGFβ receptor type II mice.

http://www.wikidata.org/entity/Q36179395

about

Restoring homeostasis of CD4⁺ T cells in hepatitis-B-virus-related liver fibrosis The immunogenetics of primary biliary cirrhosis: A comprehensive review The diagnosis of primary biliary cirrhosis The genetics of complex cholestatic disorders Identification of potential cytokine pathways for therapeutic intervention in murine primary biliary cirrhosis Implications of genome-wide association studies in novel therapeutics in primary biliary cirrhosis Mest attenuates CCl4-induced liver fibrosis in rats by inhibiting the Wnt/β-catenin signaling pathway STAT4 knockout mice are more susceptible to concanavalin A-induced T-cell hepatitis.Murine autoimmune cholangitis requires two hits: cytotoxic KLRG1(+) CD8 effector cells and defective T regulatory cells.Activated rat hepatic stellate cells influence Th1/Th2 profile in vitro.Genome-Wide Association Studies in Primary Biliary Cirrhosis.Overexpression of microRNA-21 is associated with elevated pro-inflammatory cytokines in dominant-negative TGF-β receptor type II mouse.Successful treatment of murine autoimmune cholangitis by parabiosis: Implications for hematopoietic therapy Association of IL12A Expression Quantitative Trait Loci (eQTL) With Primary Biliary Cirrhosis in a Chinese Han Population.The Distribution and the Fibrotic Role of Elevated Inflammatory Th17 Cells in Patients With Primary Biliary Cirrhosis Clonality, activated antigen-specific CD8(+) T cells, and development of autoimmune cholangitis in dnTGFβRII mice A contemporary perspective on the molecular characteristics of mitochondrial autoantigens and diagnosis in primary biliary cholangitis.Effects of an intravitreal injection of interleukin-35-expressing plasmid on pro-inflammatory and anti-inflammatory cytokines.IL-12/Th1 and IL-23/Th17 biliary microenvironment in primary biliary cirrhosis: implications for therapy.Interleukin 35: a key mediator of suppression and the propagation of infectious tolerance.Extrahepatic autoimmune conditions associated with primary biliary cirrhosis.Animal models in primary biliary cirrhosis and primary sclerosing cholangitis.Women and primary biliary cirrhosis.IL-35: a potential therapeutic target for controlling hepatitis B virus infection.IL-35 and Autoimmunity: a Comprehensive Perspective.T-helper cell-mediated factors in drug-induced liver injury.Epigenetics and Primary Biliary Cirrhosis: a Comprehensive Review and Implications for Autoimmunity.Endogenous interleukin-22 protects against inflammatory bowel disease but not autoimmune cholangitis in dominant negative form of transforming growth factor beta receptor type II mice.Animal models of primary biliary cirrhosis.Immunological potential of cytotoxic T lymphocyte antigen 4 immunoglobulin in murine autoimmune cholangitis.Anti-CD40 ligand monoclonal antibody delays the progression of murine autoimmune cholangitis.New therapeutics in primary biliary cirrhosis: will there ever be light?Mouse model of primary biliary cirrhosis with progressive fibrosis: are we there yet?Successful immunotherapy of autoimmune cholangitis by adoptive transfer of forkhead box protein 3(+) regulatory T cells.Mesenchymal Stem Cells Overexpressing Interleukin-35 Propagate Immunosuppressive Effects in Mice.Gene Therapy for Autoimmune Disease.Toward solving the etiological mystery of primary biliary cholangitis.Cholangiopathies - Towards a molecular understanding

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Deletion of interleukin (IL)-12p35 induces liver fibrosis in dominant-negative TGFβ receptor type II mice.

http://www.wikidata.org/entity/Q36179395

description

2013 nî lūn-bûn

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2013年论文

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name

Deletion of interleukin (IL)-1 ...... ve TGFβ receptor type II mice.

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Deletion of interleukin (IL)-1 ...... ve TGFβ receptor type II mice.

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Deletion of interleukin (IL)-1 ...... ve TGFβ receptor type II mice.

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Deletion of interleukin (IL)-1 ...... ve TGFβ receptor type II mice.

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Deletion of interleukin (IL)-1 ...... ve TGFβ receptor type II mice.

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Bin Gao

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Guo-Xiang Yang

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Kazuhito Kawata

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Koichi Tsuneyama

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Masanobu Tsuda

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Ogyi Park

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Patrick S C Leung

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Richard Flavell

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Weici Zhang

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William M Ridgway

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P2860

Chemokine and chemokine receptor interactions provide a mechanism for selective T cell recruitment to specific liver compartments within hepatitis C-infected liver

Primary biliary cirrhosis associated with HLA, IL12A, and IL12RB2 variants

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The inhibitory cytokine IL-35 contributes to regulatory T-cell function

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

Interleukin-12 and the regulation of innate resistance and adaptive immunity

IL-17 family cytokines and the expanding diversity of effector T cell lineages

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

Isolation of murine hepatic lymphocytes using mechanical dissection for phenotypic and functional analysis of NK1.1+ cells.

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10.1002/HEP.25829

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2

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57

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M. Eric Gershwin

Aftab A. Ansari

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2013-02-01T00:00:00Z

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235901392

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22576253

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3424295

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