Inflammatory conditions induce gap junctional communication between rat Kupffer cells both in vivo and in vitro
about
Connexins and pannexins in liver damageConnexin and pannexin signaling in gastrointestinal and liver diseaseRoles of connexins and pannexins in digestive homeostasisRole of connexin/pannexin containing channels in infectious diseasesReciprocal regulation between proinflammatory cytokine-induced inducible NO synthase (iNOS) and connexin43 in bladder smooth muscle cellsInvolvement of connexin43 in acetaminophen-induced liver injury.Connexins, Pannexins, and Their Channels in Fibroproliferative DiseasesThe role of gap junction channels during physiologic and pathologic conditions of the human central nervous systemA quantitative model of thermal injury-induced acute inflammation.A role for connexin43 in macrophage phagocytosis and host survival after bacterial peritoneal infection.Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegenerationMechanisms of HIV Neuropathogenesis: Role of Cellular Communication Systems.Structure, Regulation and Function of Gap Junctions in Liver.Gap junctional intercellular communication as a target for liver toxicity and carcinogenicity.Modulation of connexin signaling by bacterial pathogens and their toxins.Regulation of hemichannels and gap junction channels by cytokines in antigen-presenting cells.The role of connexin and pannexin containing channels in the innate and acquired immune response.Connexins and their channels in inflammation.Inhibitors of connexin and pannexin channels as potential therapeutics.Connexin-based signaling and drug-induced hepatotoxicity.C(6)-ceramide enhances phagocytic activity of Kupffer cells through the production of endogenous ceramidesPMNs facilitate translocation of platelets across human and mouse epithelium and together alter fluid homeostasis via epithelial cell-expressed ecto-NTPDases.Role of the JNK signaling pathway in downregulation of connexin43 by TNF-α in human corneal fibroblasts.TAT-Gap19 and Carbenoxolone Alleviate Liver Fibrosis in Mice.Distinct behavior of myelomonocytic cells and CD8 T cells underlies the hepatic response to Listeria monocytogenes.
P2860
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P2860
Inflammatory conditions induce gap junctional communication between rat Kupffer cells both in vivo and in vitro
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Inflammatory conditions induce ...... ells both in vivo and in vitro
@ast
Inflammatory conditions induce ...... ells both in vivo and in vitro
@en
type
label
Inflammatory conditions induce ...... ells both in vivo and in vitro
@ast
Inflammatory conditions induce ...... ells both in vivo and in vitro
@en
prefLabel
Inflammatory conditions induce ...... ells both in vivo and in vitro
@ast
Inflammatory conditions induce ...... ells both in vivo and in vitro
@en
P2093
P2860
P1433
P1476
Inflammatory conditions induce ...... ells both in vivo and in vitro
@en
P2093
Eliseo A Eugenín
Helmuth A Sánchez
Hernán E González
Juan C Sáez
María C Brañes
P2860
P304
P356
10.1016/J.CELLIMM.2007.08.001
P577
2007-06-01T00:00:00Z