ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
about
Suppression of ATAD2 inhibits hepatocellular carcinoma progression through activation of p53- and p38-mediated apoptotic signalingATAD2 is an epigenetic reader of newly synthesized histone marks during DNA replication.Low expression of DCXR protein indicates a poor prognosis for hepatocellular carcinoma patients.ATAD2 is overexpressed in gastric cancer and serves as an independent poor prognostic biomarker.Human germ/stem cell-specific gene TEX19 influences cancer cell proliferation and cancer prognosis.ATAD2 overexpression is associated with progression and prognosis in colorectal cancer.Decreased Expression of ZWINT is Associated With Poor Prognosis in Patients With HCC After Surgery
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ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@ast
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@en
type
label
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@ast
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@en
prefLabel
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@ast
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@en
P2093
P2860
P356
P1476
ATAD2 as a Poor Prognostic Marker for Hepatocellular Carcinoma after Curative Resection
@en
P2093
Cheol-Keun Park
Heejin Bang
Hye Won Hwang
Sang Yun Ha
P2860
P304
P356
10.4143/CRT.2014.177
P577
2015-02-16T00:00:00Z