FANCD2, FANCJ and BRCA2 cooperate to promote replication fork recovery independently of the Fanconi Anemia core complex.
about
Getting Ready for the Dance: FANCJ Irons Out DNA WrinklesCellular response to DNA interstrand crosslinks: the Fanconi anemia pathwayInteraction with PALB2 Is Essential for Maintenance of Genomic Integrity by BRCA2Recovery from the DNA Replication CheckpointMolding BRCA2 function through its interacting partnersChromosomal Integrity after UV Irradiation Requires FANCD2-Mediated Repair of Double Strand Breaks.Primary Ovarian Insufficiency Induced by Fanconi Anemia E Mutation in a Mouse Model.Replication Protein A (RPA) deficiency activates the Fanconi anemia DNA repair pathway.Mutated Fanconi anemia pathway in non-Fanconi anemia cancersRad51 recombinase prevents Mre11 nuclease-dependent degradation and excessive PrimPol-mediated elongation of nascent DNA after UV irradiation.FANCJ at the FORK.Nuclear alpha spectrin: Critical roles in DNA interstrand cross-link repair and genomic stability.FANCJ is essential to maintain microsatellite structure genome-wide during replication stress.Nuclear α Spectrin Differentially Affects Monoubiquitinated Versus Non-Ubiquitinated FANCD2 Function After DNA Interstrand Cross-Link Damage.The Emerging Role of Non-traditional Ubiquitination in Oncogenic Pathways.FANCD2 Facilitates Replication through Common Fragile Sites.The USP1-UAF1 complex interacts with RAD51AP1 to promote homologous recombination repair.BLM promotes the activation of Fanconi Anemia signaling pathway.Proliferation of Double-Strand Break-Resistant Polyploid Cells Requires Drosophila FANCD2.BRCA2 antagonizes classical and alternative nonhomologous end-joining to prevent gross genomic instability.FANCI and FANCD2 have common as well as independent functions during the cellular replication stress response.Fanconi Anemia FANCM/FNCM-1 and FANCD2/FCD-2 Are Required for Maintaining Histone Methylation Levels and Interact with the Histone Demethylase LSD1/SPR-5 in Caenorhabditis elegans.Fork Protection and Therapy Resistance in Hereditary Breast Cancer.A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair
P2860
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P2860
FANCD2, FANCJ and BRCA2 cooperate to promote replication fork recovery independently of the Fanconi Anemia core complex.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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name
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@ast
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@en
type
label
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@ast
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@en
prefLabel
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@ast
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@en
P2093
P2860
P1433
P1476
FANCD2, FANCJ and BRCA2 cooper ...... e Fanconi Anemia core complex.
@en
P2093
Alexandra Sobeck
Helmut Hanenberg
Indrajit Chaudhury
Stephanie L Kelich
P2860
P304
P356
10.4161/15384101.2014.987614
P577
2015-01-01T00:00:00Z