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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

http://www.wikidata.org/entity/Q36190811

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System for Rapid, Precise Modulation of Intraocular Pressure, toward Minimally-Invasive In Vivo Measurement of Intracranial Pressure Animal models of glucocorticoid-induced glaucoma What do mechanotransduction, Hippo, Wnt, and TGFβ have in common? YAP and TAZ as key orchestrating molecules in ocular health and disease.Localized and Controlled Delivery of Nitric Oxide to the Conventional Outflow Pathway via Enzyme Biocatalysis: Toward Therapy for Glaucoma.The structure of the trabecular meshwork, its connections to the ciliary muscle, and the effect of pilocarpine on outflow facility in mice.Pilocarpine-induced dilation of Schlemm's canal and prevention of lumen collapse at elevated intraocular pressures in living mice visualized by OCT.ATP-sensitive potassium (KATP) channel openers diazoxide and nicorandil lower intraocular pressure by activating the Erk1/2 signaling pathway Ultrastructural changes associated with dexamethasone-induced ocular hypertension in mice.The formation of cortical actin arrays in human trabecular meshwork cells in response to cytoskeletal disruption Measurement of Outflow Facility Using iPerfusion Role of nitric oxide in murine conventional outflow physiology.Using genetic mouse models to gain insight into glaucoma: Past results and future possibilities.High-throughput screening for modulators of cellular contractile force.Physical Factors Affecting Outflow Facility Measurements in Mice.Dose- and time-dependent effects of actomyosin inhibition on live mouse outflow resistance and aqueous drainage tissues The Soluble Guanylate Cyclase Stimulator IWP-953 Increases Conventional Outflow Facility in Mouse Eyes.Differential effects of prostaglandin E2-sensitive receptors on contractility of human ocular cells that regulate conventional outflow ATP-sensitive potassium (K(ATP)) channel openers diazoxide and nicorandil lower intraocular pressure in vivo Transplantation of iPSC-derived TM cells rescues glaucoma phenotypes in vivo.Pigment epithelium-derived factor decreases outflow facility.Suppression of Acid Sphingomyelinase Protects the Retina from Ischemic Injury.The influence of genetic background on conventional outflow facility in mice.Feedback-controlled constant-pressure anterior chamber perfusion in live mice.VEGF as a Paracrine Regulator of Conventional Outflow Facility.Bioactive lysophospholipids: role in regulation of aqueous humor outflow and intraocular pressure in the context of pathobiology and therapy of glaucoma.Biomechanics of Schlemm's canal endothelium and intraocular pressure reduction.Unconventional aqueous humor outflow: A review.Model systems for the study of steroid-induced IOP elevation.Optimizing gene transfer to conventional outflow cells in living mouse eyes.Visualization of conventional outflow tissue responses to netarsudil in living mouse eyes.Endogenous production of extracellular adenosine by trabecular meshwork cells: potential role in outflow regulation Differential Intraocular Pressure Measurements by Tonometry and Direct Cannulation After Treatment with Soluble Adenylyl Cyclase Inhibitors.The relationship between outflow resistance and trabecular meshwork stiffness in mice.Aqueous Humor Dynamics of the Brown-Norway Rat.

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

http://www.wikidata.org/entity/Q36190811

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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Investigative Ophthalmology & Visual Science

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Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes

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P2093

Alexandra Boussommier-Calleja

http://www.w3.org/2001/XMLSchema#string

C Ross Ethier

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David F Woodward

http://www.w3.org/2001/XMLSchema#string

Jacques Bertrand

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W Daniel Stamer

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P2860

Aquaporin deletion in mice reduces intraocular pressure and aqueous fluid production

Sphingosine-1-phosphate: an enigmatic signalling lipid

Expression of cyclooxygenase-1 and -2 in normal and glaucomatous human eyes.

Role of lysophospholipid growth factors in the modulation of aqueous humor outflow facility.

Prostaglandin E2 receptor subtypes, EP1, EP2, EP3 and EP4 in human and mouse ocular tissues--a comparative immunohistochemical study.

Enhanced inflow and outflow rates despite lower IOP in bestrophin-2-deficient mice.

Mouse genetics: a tool to help unlock the mechanisms of glaucoma.

International Union of Basic and Clinical Pharmacology. LXXXIII: classification of prostanoid receptors, updating 15 years of progress

Effects of prostaglandins on the aqueous humor outflow pathways.

S1P₂ receptor regulation of sphingosine-1-phosphate effects on conventional outflow physiology

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5838-5845

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10.1167/IOVS.12-9923

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9

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Darryl R. Overby

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229161379

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3428113

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