RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease. - Wikidata - awgldk - TriplyDB

RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

http://www.wikidata.org/entity/Q36193793

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Plausible Roles for RAGE in Conditions Exacerbated by Direct and Indirect (Secondhand) Smoke Exposure.Sex-specific lung functional changes in adult mice exposed only to second-hand smoke in utero.Antenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.Nontypeable Haemophilus Influenzae Infection Upregulates the NLRP3 Inflammasome and Leads to Caspase-1-Dependent Secretion of Interleukin-1β - A Possible Pathway of Exacerbations in COPD Effect of hospital use of oral nutritional supplementation on length of stay, hospital cost, and 30-day readmissions among Medicare patients with COPD.Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous Spermidine Association of polymorphisms of the receptor for advanced glycation end products gene with COPD in the Chinese population.Blockade of RAGE ameliorates elastase-induced emphysema development and progression via RAGE-DAMP signaling.Second hand smoke and COPD: lessons from animal studies.Conditional over-expression of RAGE by embryonic alveolar epithelium compromises the respiratory membrane and impairs endothelial cell differentiation.Conditionally induced RAGE expression by proximal airway epithelial cells in transgenic mice causes lung inflammation.Inhibition of the receptor for advanced glycation end-products (RAGE) protects from secondhand smoke (SHS)-induced intrauterine growth restriction IUGR in mice.Effect of long-term maternal smoking on the offspring's lung health.Cigarette smoke extract induces oral squamous cell carcinoma cell invasion in a receptor for advanced glycation end-products-dependent manner.Cardiomyocyte mitochondrial respiration is reduced by receptor for advanced glycation end-product signaling in a ceramide-dependent manner.Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice.COPD as an endothelial disorder: endothelial injury linking lesions in the lungs and other organs? (2017 Grover Conference Series).Receptor for advanced glycation end products and soluble receptor for advanced glycation end products: a balancing act in chronic obstructive pulmonary disease?

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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

http://www.wikidata.org/entity/Q36193793

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2012 nî lūn-bûn

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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

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FPHYS.2012.00301

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Frontiers in Physiology

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RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.

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Adam B Robinson

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Jeffrey A Stogsdill

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Joshua B Lewis

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Paul R Reynolds

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Tyler T Wood

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P2860

S100A14 stimulates cell proliferation and induces cell apoptosis at different concentrations via receptor for advanced glycation end products (RAGE)

Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Cloning and characterization of a unique elastolytic metalloproteinase produced by human alveolar macrophages

Interaction of the RAGE cytoplasmic domain with diaphanous-1 is required for ligand-stimulated cellular migration through activation of Rac1 and Cdc42

Tobacco smoke is a source of toxic reactive glycation products

The natural history of chronic airflow obstruction

Hazardous compounds in tobacco smoke

Update in environmental and occupational medicine 2010

Receptor for advanced glycation end-products is a marker of type I cell injury in acute lung injury

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301

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10.3389/FPHYS.2012.00301

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3

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2012-07-25T00:00:00Z

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230763838

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22934052

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chronic obstructive pulmonary disease

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3429072

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