Chronic valproate treatment enhances postischemic angiogenesis and promotes functional recovery in a rat model of ischemic stroke - Wikidata - awgldk - TriplyDB

Chronic valproate treatment enhances postischemic angiogenesis and promotes functional recovery in a rat model of ischemic stroke

Chronic valproate treatment enhances postischemic angiogenesis and promotes functional recovery in a rat model of ischemic stroke

http://www.wikidata.org/entity/Q36195297

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Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorder HDAC and HDAC Inhibitor: From Cancer to Cardiovascular Diseases The power of using functional fMRI on small rodents to study brain pharmacology and disease Histone Deacetylases Exert Class-Specific Roles in Conditioning the Brain and Heart Against Acute Ischemic Injury Novel protective effects of histone deacetylase inhibition on stroke and white matter ischemic injury Visualizing epigenetics: current advances and advantages in HDAC PET imaging techniques A transient ischemic environment induces reversible compaction of chromatin Histone methylation patterns in astrocytes are influenced by age following ischemia HDAC inhibitors mitigate ischemia-induced oligodendrocyte damage: potential roles of oligodendrogenesis, VEGF, and anti-inflammation The role of citicoline in neuroprotection and neurorepair in ischemic stroke.A new avenue for lithium: intervention in traumatic brain injury.Hypoxia-inducible factor-1 alpha is involved in RIP-induced necroptosis caused by in vitro and in vivo ischemic brain injury Post-insult valproate treatment potentially improved functional recovery in patients with acute middle cerebral artery infarction.Promoting endothelial function by S-nitrosoglutathione through the HIF-1α/VEGF pathway stimulates neurorepair and functional recovery following experimental stroke in rats.Progesterone protects endothelial cells after cerebrovascular occlusion by decreasing MCP-1- and CXCL1-mediated macrophage infiltration.Post-insult valproic acid-regulated microRNAs: potential targets for cerebral ischemia.Tubastatin A, an HDAC6 inhibitor, alleviates stroke-induced brain infarction and functional deficits: potential roles of α-tubulin acetylation and FGF-21 up-regulation Novel molecular triggers underlie valproate-induced liver injury and its alleviation by the omega-3 fatty acid DHA: role of inflammation and apoptosis Epigenetic mechanisms in cerebral ischemia.Valproic Acid and Other HDAC Inhibitors Upregulate FGF21 Gene Expression and Promote Process Elongation in Glia by Inhibiting HDAC2 and 3 Inhibition of Histone Deacetylase 3 (HDAC3) Mediates Ischemic Preconditioning and Protects Cortical Neurons against Ischemia in Rats Chronic cerebral hypoperfusion causes decrease of O-GlcNAcylation, hyperphosphorylation of tau and behavioral deficits in mice.Microglia preconditioned by oxygen-glucose deprivation promote functional recovery in ischemic rats.Valproic acid: a new candidate of therapeutic application for the acute central nervous system injuries.Cerebral neovascularization in diabetes: implications for stroke recovery and beyond.From chronic cerebral hypoperfusion to Alzheimer-like brain pathology and neurodegeneration.The relevance of epigenetics to occlusive cerebral and peripheral arterial disease.Astrocytic response to cerebral ischemia is influenced by sex differences and impaired by aging The Emerging Role of Epigenetics in Cerebral Ischemia.Concordance of Several Subcellular Interactions Initiates Alzheimer's Dementia: Their Reversal Requires Combination Treatment.Rodent Gymnastics: Neurobehavioral Assays in Ischemic Stroke.Implications of Epigenetic Mechanisms and their Targets in Cerebral Ischemia Models.GSNO promotes functional recovery in experimental TBI by stabilizing HIF-1α.Valproic Acid Prevents Renal Dysfunction and Inflammation in the Ischemia-Reperfusion Injury Model.Impact of trichostatin A and sodium valproate treatment on post-stroke neurogenesis and behavioral outcomes in immature mice.Synergistic Association of Valproate and Resveratrol Reduces Brain Injury in Ischemic Stroke.Physiologically based microenvironment for in vitro neural differentiation of adipose-derived stem cells.LncRNA ameliorates brain microvascular endothelial cell injury by targeting miR-199a

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Chronic valproate treatment enhances postischemic angiogenesis and promotes functional recovery in a rat model of ischemic stroke

http://www.wikidata.org/entity/Q36195297

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Chronic valproate treatment en ...... a rat model of ischemic stroke

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De-Maw Chuang

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Emily Bame Fessler

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Fairouz Chibane

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Jeeva Munasinghe

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Peter Leeds

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Yan Leng

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Zhifei Wang

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Protein kinase D-dependent phosphorylation and nuclear export of histone deacetylase 5 mediates vascular endothelial growth factor-induced gene expression and angiogenesis

HDAC5 is a repressor of angiogenesis and determines the angiogenic gene expression pattern of endothelial cells

General involvement of hypoxia-inducible factor 1 in transcriptional response to hypoxia

Increased gelatinase A (MMP-2) and gelatinase B (MMP-9) activities in human brain after focal ischemia

Histone deacetylase 7 associates with hypoxia-inducible factor 1alpha and increases transcriptional activity

VEGF enhances angiogenesis and promotes blood-brain barrier leakage in the ischemic brain.

Beneficial effects of mood stabilizers lithium, valproate and lamotrigine in experimental stroke models.

Neurotrophins promote revascularization by local recruitment of TrkB+ endothelial cells and systemic mobilization of hematopoietic progenitors.

Vascular endothelial growth factor receptor-2 inhibition promotes cell death and limits endothelial cell proliferation in a neonatal rodent model of stroke

Effects of the histone deacetylase inhibitor valproic acid on human pericytes in vitro.

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