PPARγ isoforms differentially regulate metabolic networks to mediate mouse prostatic epithelial differentiation.
about
Oxidative stress in prostate hyperplasia and carcinogenesisA Novel Splicing Variant of Peroxisome Proliferator-Activated Receptor-γ (Pparγ1sv) Cooperatively Regulates Adipocyte Differentiation with Pparγ2Surgical intervention for symptomatic benign prostatic hyperplasia is correlated with expression of the AP-1 transcription factor networkInhibition of cancer cell proliferation by PPARγ is mediated by a metabolic switch that increases reactive oxygen species levelsEPI-001 is a selective peroxisome proliferator-activated receptor-gamma modulator with inhibitory effects on androgen receptor expression and activity in prostate cancerTRAMP prostate tumor growth is slowed by walnut diets through altered IGF-1 levels, energy pathways, and cholesterol metabolism.Evaluation of the synuclein-γ (SNCG) gene as a PPARγ target in murine adipocytes, dorsal root ganglia somatosensory neurons, and human adipose tissue.Molecular subtypes of urothelial carcinoma are defined by specific gene regulatory systemsDeficiency in metabolic regulators PPARγ and PTEN cooperates to drive keratinizing squamous metaplasia in novel models of human tissue regeneration.NF-κB and androgen receptor variant expression correlate with human BPH progressionTaurine protects against As2O3-induced autophagy in livers of rat offsprings through PPARγ pathwayThe Androgen Receptor Regulates PPARγ Expression and Activity in Human Prostate Cancer Cells.Schlafen 12 expression modulates prostate cancer cell differentiation.Bone marrow adipocytes promote tumor growth in bone via FABP4-dependent mechanisms.The role of peroxisome proliferator-activated receptor gamma in prostate cancer.Differential ontogenetic exposure to obesogenic environment induces hyperproliferative status and nuclear receptors imbalance in the rat prostate at adulthood.Crosstalk between the Androgen Receptor and PPAR Gamma Signaling Pathways in the Prostate.Association of genetic variants and expression levels of porcine FABP4 and FABP5 genes.Fatty acid binding protein 4 enhances prostate cancer progression by upregulating matrix metalloproteinases and stromal cell cytokine production.Estrogen receptor-beta is a potential target for triple negative breast cancer treatment
P2860
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P2860
PPARγ isoforms differentially regulate metabolic networks to mediate mouse prostatic epithelial differentiation.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
PPARγ isoforms differentially ...... ic epithelial differentiation.
@ast
PPARγ isoforms differentially ...... ic epithelial differentiation.
@en
type
label
PPARγ isoforms differentially ...... ic epithelial differentiation.
@ast
PPARγ isoforms differentially ...... ic epithelial differentiation.
@en
prefLabel
PPARγ isoforms differentially ...... ic epithelial differentiation.
@ast
PPARγ isoforms differentially ...... ic epithelial differentiation.
@en
P2093
P2860
P50
P356
P1476
PPARγ isoforms differentially ...... tic epithelial differentiation
@en
P2093
P2860
P356
10.1038/CDDIS.2012.99
P577
2012-08-09T00:00:00Z