Mammalian alteration/deficiency in activation 3 (Ada3) is essential for embryonic development and cell cycle progression.
about
Tale of a multifaceted co-activator, hADA3: from embryogenesis to cancer and beyondA Novel Interaction of Ecdysoneless (ECD) Protein with R2TP Complex Component RUVBL1 Is Required for the Functional Role of ECD in Cell Cycle ProgressionCo-expression analysis of fetal weight-related genes in ovine skeletal muscle during mid and late fetal development stages.Alteration/Deficiency in Activation 3 (ADA3) Protein, a Cell Cycle Regulator, Associates with the Centromere through CENP-B and Regulates Chromosome Segregation.Cytoplasmic localization of alteration/deficiency in activation 3 (ADA3) predicts poor clinical outcome in breast cancer patients.ADA3 regulates normal and tumor mammary epithelial cell proliferation through c-MYC.A functional genomics screen identifies PCAF and ADA3 as regulators of human granzyme B-mediated apoptosis and Bid cleavage.Acetylation of Mammalian ADA3 Is Required for Its Functional Roles in Histone Acetylation and Cell Proliferation.Alteration/deficiency in activation-3 (Ada3) plays a critical role in maintaining genomic stability.Epidermal Growth Factor Receptor activation promotes ADA3 acetylation through the AKT-p300 pathway.Genes conserved in bilaterians but jointly lost with Myc during nematode evolution are enriched in cell proliferation and cell migration functionsEnzymatic modules of the SAGA chromatin-modifying complex play distinct roles in Drosophila gene expression and development.High-risk HPV16E6 stimulates hADA3 degradation by enhancing its SUMOylation.Genomic instability: Ada3 and HPV E6-acetyltransferase connections?Evidence for differential alternative splicing in blood of young boys with autism spectrum disorders.Alteration/deficiency in activation 3 (ADA3): Regulator of DNA repair and genome stability.Loss of the nuclear pool of ubiquitin ligase CHIP/STUB1 in breast cancer unleashes the MZF1-cathepsin pro-oncogenic program.
P2860
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P2860
Mammalian alteration/deficiency in activation 3 (Ada3) is essential for embryonic development and cell cycle progression.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@ast
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@en
type
label
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@ast
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@en
prefLabel
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@ast
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@en
P2093
P2860
P356
P1476
Mammalian alteration/deficienc ...... nt and cell cycle progression.
@en
P2093
Bryan Katafiasz
Channabasavaiah Basavaraju Gurumurthy
Chittibabu Guda
Hamid Band
James Eudy
Mayumi Naramura
Meghan Quinn
Sameer Mirza
P2860
P304
29442-29456
P356
10.1074/JBC.M112.378901
P407
P577
2012-06-26T00:00:00Z