Receptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss - Wikidata - awgldk - TriplyDB

Receptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss

Receptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss

http://www.wikidata.org/entity/Q36216058

about

The Modulatory Effects of Mesenchymal Stem Cells on Osteoclastogenesis High Cholesterol Deteriorates Bone Health: New Insights into Molecular Mechanisms The role of estrogen and androgen receptors in bone health and disease Immune cells link obesity-associated type 2 diabetes and periodontitis Rib fractures and death from deletion of osteoblast βcatenin in adult mice is rescued by corticosteroids Osteocytes, not Osteoblasts or Lining Cells, are the Main Source of the RANKL Required for Osteoclast Formation in Remodeling Bone Identification of B cells participated in the mechanism of postmenopausal women osteoporosis using microarray analysis Biologic therapies and bone loss in rheumatoid arthritis.Bone remodelling markers in rheumatoid arthritis.Activation of Toll‐like receptor 9 inhibits lipopolysaccharide‐induced receptor activator of nuclear factor kappa‐ B ligand expression in rat B lymphocytes.FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation.Osteocyte-derived RANKL is a critical mediator of the increased bone resorption caused by dietary calcium deficiency.SRT2104 extends survival of male mice on a standard diet and preserves bone and muscle mass Biology of the RANKL-RANK-OPG System in Immunity, Bone, and Beyond.Physiological functions of osteoblast lineage and T cell-derived RANKL in bone homeostasis.Sex steroid actions in male bone Dysregulated B cell expression of RANKL and OPG correlates with loss of bone mineral density in HIV infection Estrogens and Androgens in Skeletal Physiology and Pathophysiology.Denosumab: targeting the RANKL pathway to treat rheumatoid arthritis.T-cells and B-cells in osteoporosis Minireview: nuclear receptor regulation of osteoclast and bone remodeling.Bone-immune cell crosstalk: bone diseases.B Cell Production of Both OPG and RANKL is Significantly Increased in Aged Mice Mechanical environment change in root, periodontal ligament, and alveolar bone in response to two canine retraction treatment strategies Osteoclasts: New Insights Trauma-induced heterotopic bone formation and the role of the immune system: A review Unique Distal Enhancers Linked to the Mouse Tnfsf11 Gene Direct Tissue-Specific and Inflammation-Induced Expression of RANKL.New Insights Into Androgen and Estrogen Receptor Regulation of the Male Skeleton.Non-nuclear-initiated actions of the estrogen receptor protect cortical bone mass.Low bone mass and changes in the osteocyte network in mice lacking autophagy in the osteoblast lineage.Deletion of the Distal Tnfsf11 RL-D2 Enhancer That Contributes to PTH-Mediated RANKL Expression in Osteoblast Lineage Cells Results in a High Bone Mass Phenotype in Mice RANKL cytokine: from pioneer of the osteoimmunology era to cure for a rare disease Osteoimmunology: from mice to humans The Effects of Androgens on Murine Cortical Bone Do Not Require AR or ERα Signaling in Osteoblasts and Osteoclasts Postmenopausal osteoporosis: the role of immune system cells Osteoclasts-Key Players in Skeletal Health and Disease.The Interplay between the bone and the immune system Production of RANKL by Memory B Cells: A Link Between B Cells and Bone Erosion in Rheumatoid Arthritis.Basic biology of skeletal aging: role of stress response pathways.The Role of Inflammatory Cytokines, the RANKL/OPG Axis, and the Immunoskeletal Interface in Physiological Bone Turnover and Osteoporosis.

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Receptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone loss

http://www.wikidata.org/entity/Q36216058

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2012年论文

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2012年论文

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Receptor activator of nuclear ...... ovariectomy-induced bone loss

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Receptor activator of nuclear ...... ovariectomy-induced bone loss

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Receptor activator of nuclear ...... ovariectomy-induced bone loss

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Receptor activator of nuclear ...... ovariectomy-induced bone loss

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JBC.M112.377945

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Journal of Biological Chemistry

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Receptor activator of nuclear ...... ovariectomy-induced bone loss

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P2093

Charles A O'Brien

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Jeff D Thostenson

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Jinhu Xiong

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Maria Almeida

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Melda Onal

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Stavros C Manolagas

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Xinrong Chen

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P2860

Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Matrix-embedded cells control osteoclast formation

Evidence for osteocyte regulation of bone homeostasis through RANKL expression

Attenuation of the self-renewal of transit-amplifying osteoblast progenitors in the murine bone marrow by 17 beta-estradiol

The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone

Orchiectomy upregulates free soluble RANKL in bone marrow of aged rats

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35

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3436192

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