An integrated transcriptional regulatory circuit that reinforces the breast cancer stem cell state - Wikidata - awgldk - TriplyDB

An integrated transcriptional regulatory circuit that reinforces the breast cancer stem cell state

An integrated transcriptional regulatory circuit that reinforces the breast cancer stem cell state

http://www.wikidata.org/entity/Q36221885

about

TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance BMI1: A Biomarker of Hematologic Malignancies Common stemness regulators of embryonic and cancer stem cells Implications of miR cluster 143/145 as universal anti-oncomiRs and their dysregulation during tumorigenesis MicroRNAs: New Biomarkers for Diagnosis, Prognosis, Therapy Prediction and Therapeutic Tools for Breast Cancer Tumor suppressor microRNAs: a novel non-coding alliance against cancer Large oncosomes mediate intercellular transfer of functional microRNA.Epithelial-to-mesenchymal transition and the cancer stem cell phenotype: insights from cancer biology with therapeutic implications for colorectal cancer.Computational analysis identifies a sponge interaction network between long non-coding RNAs and messenger RNAs in human breast cancer.The nutritional phenome of EMT-induced cancer stem-like cells miR-107 activates ATR/Chk1 pathway and suppress cervical cancer invasion by targeting MCL1.Stability of the hybrid epithelial/mesenchymal phenotype.MicroRNA-145: a potent tumour suppressor that regulates multiple cellular pathways.MiR-200a inhibits epithelial-mesenchymal transition of pancreatic cancer stem cell.MicroRNA100 inhibits self-renewal of breast cancer stem-like cells and breast tumor development By promoting cell differentiation, miR-100 sensitizes basal-like breast cancer stem cells to hormonal therapy.Lgr5 Methylation in Cancer Stem Cell Differentiation and Prognosis-Prediction in Colorectal Cancer Post-transcriptional regulation of BRCA1 through its coding sequence by the miR-15/107 group of miRNAs.MicroRNA networks regulated by all-trans retinoic acid and Lapatinib control the growth, survival and motility of breast cancer cells WNT7A Regulation by miR-15b in Ovarian Cancer.The miR-200 family and its targets regulate type II cell differentiation in human fetal lung.Identification of MicroRNAs as Breast Cancer Prognosis Markers through the Cancer Genome Atlas.How interacting pathways are regulated by miRNAs in breast cancer subtypes.Leptin-STAT3-G9a Signaling Promotes Obesity-Mediated Breast Cancer Progression.Metformin inhibits the inflammatory response associated with cellular transformation and cancer stem cell growth.Nuclear reprogramming of luminal-like breast cancer cells generates Sox2-overexpressing cancer stem-like cellular states harboring transcriptional activation of the mTOR pathway.A feed-forward loop between lncARSR and YAP activity promotes expansion of renal tumour-initiating cells.Downregulation of Smurf2, a tumor-suppressive ubiquitin ligase, in triple-negative breast cancers: involvement of the RB-microRNA axis.A novel panel of serum miR-21/miR-155/miR-365 as a potential diagnostic biomarker for breast cancer.MiR-24 induces chemotherapy resistance and hypoxic advantage in breast cancer.Roles of microRNAs during prostatic tumorigenesis and tumor progression.Aptamers and their potential to selectively target aspects of EGF, Wnt/β-catenin and TGFβ-smad family signaling.Tumor heterogeneity and cancer stem cell paradigm: updates in concept, controversies and clinical relevance.The cancer stem cell hypothesis: a guide to potential molecular targets.Metabostemness: a new cancer hallmark.Role of microRNAs in maintaining cancer stem cells.Xenopatients 2.0: reprogramming the epigenetic landscapes of patient-derived cancer genomes.Recurrence and metastasis of breast cancer is influenced by ovarian hormone's effect on breast cancer stem cells.Aberrant regulation of miR-15b in human malignant tumors and its effects on the hallmarks of cancer.Breast cancer stem cells programs: enter the (non)-code.

P2860

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P2860

An integrated transcriptional regulatory circuit that reinforces the breast cancer stem cell state

http://www.wikidata.org/entity/Q36221885

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

@zh

2012年论文

@zh-cn

name

An integrated transcriptional ...... breast cancer stem cell state

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An integrated transcriptional ...... breast cancer stem cell state

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type

label

An integrated transcriptional ...... breast cancer stem cell state

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An integrated transcriptional ...... breast cancer stem cell state

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prefLabel

An integrated transcriptional ...... breast cancer stem cell state

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An integrated transcriptional ...... breast cancer stem cell state

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Proceedings of the National Academy of Sciences of the United States of America

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An integrated transcriptional ...... breast cancer stem cell state

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Christos Polytarchou

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Dimitrios Iliopoulos

http://www.w3.org/2001/XMLSchema#string

Kevin Struhl

http://www.w3.org/2001/XMLSchema#string

P2860

The miR-200 family inhibits epithelial-mesenchymal transition and cancer cell migration by direct targeting of E-cadherin transcriptional repressors ZEB1 and ZEB2

Loss of miR-200 inhibition of Suz12 leads to polycomb-mediated repression required for the formation and maintenance of cancer stem cells

p53 represses c-Myc through induction of the tumor suppressor miR-145

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

An epigenetic switch involving NF-kappaB, Lin28, Let-7 MicroRNA, and IL6 links inflammation to cell transformation

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

Control of developmental regulators by Polycomb in human embryonic stem cells

Transitions between epithelial and mesenchymal states: acquisition of malignant and stem cell traits

miR-15a and miR-16 are implicated in cell cycle regulation in a Rb-dependent manner and are frequently deleted or down-regulated in non-small cell lung cancer

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14470-14475

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10.1073/PNAS.1212811109

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