Polymorphism in signal transduction is a major route through which osteoarthritis susceptibility is acting.
about
Lack of association of a variable number of aspartic acid residues in the asporin gene with osteoarthritis susceptibility: case-control studies in Spanish CaucasiansCrystals in hand.Monoclinic form of isopentenyl diphosphate isomerase: a case of polymorphism in biomolecular crystalsCrystallographic analysis of metal-ion binding to human ubiquitinNovel markers for differentiation of lobular and ductal invasive breast carcinomas by laser microdissection and microarray analysis.Differential expression of GADD45beta in normal and osteoarthritic cartilage: potential role in homeostasis of articular chondrocytes.Lack of association between the CALM1 core promoter polymorphism (-16C/T) and susceptibility to knee osteoarthritis in a Chinese Han population.Prevalence and concomitants of arthritis in the elderly in Rio Grande do Sul, Brazil.Strong linkage on 2q33.3 to familial early-onset generalized osteoarthritis and a consideration of two positional candidate genes.Morphologic differences between the hips of Chinese women and white women: could they account for the ethnic difference in the prevalence of hip osteoarthritis?Comparative analysis of gene expression profiles in normal hip human cartilage and cartilage from patients with necrosis of the femoral headAsporin, a susceptibility gene in osteoarthritis, is expressed at higher levels in the more degenerate human intervertebral discGene expression profile analysis of human intervertebral disc degeneration.Cartilage homeostasis in health and rheumatic diseases.Roles of inflammatory and anabolic cytokines in cartilage metabolism: signals and multiple effectors converge upon MMP-13 regulation in osteoarthritis.Beta-catenin, cartilage, and osteoarthritis.Chondropenia: current concept review.HIF-1α:CRAT:miR-144-3p axis dysregulation promotes osteoarthritis chondrocyte apoptosis and VLCFA accumulation.Molecular mechanisms of cartilage destruction: mechanics, inflammatory mediators, and aging collide.The retinoic acid binding protein CRABP2 is increased in murine models of degenerative joint disease.
P2860
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P2860
Polymorphism in signal transduction is a major route through which osteoarthritis susceptibility is acting.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Polymorphism in signal transdu ...... itis susceptibility is acting.
@ast
Polymorphism in signal transdu ...... itis susceptibility is acting.
@en
type
label
Polymorphism in signal transdu ...... itis susceptibility is acting.
@ast
Polymorphism in signal transdu ...... itis susceptibility is acting.
@en
prefLabel
Polymorphism in signal transdu ...... itis susceptibility is acting.
@ast
Polymorphism in signal transdu ...... itis susceptibility is acting.
@en
P1476
Polymorphism in signal transdu ...... ritis susceptibility is acting
@en
P2093
John Loughlin
P304
P356
10.1097/01.BOR.0000176687.85198.49
P577
2005-09-01T00:00:00Z