Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes - Wikidata - awgldk - TriplyDB

Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

http://www.wikidata.org/entity/Q36226158

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Human immunodeficiency virus infection of human astrocytes disrupts blood-brain barrier integrity by a gap junction-dependent mechanism Acquired immunodeficiency syndrome and the blood-brain barrier Endothelin-1 and its role in the pathogenesis of infectious diseases Role of connexin/pannexin containing channels in infectious diseases Synergistic cooperation between methamphetamine and HIV-1 gsp120 through the P13K/Akt pathway induces IL-6 but not IL-8 expression in astrocytes Dopamine receptor activation increases HIV entry into primary human macrophages CCL2 disrupts the adherens junction: implications for neuroinflammation Flaviviruses, an expanding threat in public health: focus on dengue, West Nile, and Japanese encephalitis virus.Transcriptome analyses identify key cellular factors associated with HIV-1-associated neuropathogenesis in infected men.Regulation of gap junction channels by infectious agents and inflammation in the CNS.Vorinostat positively regulates synaptic plasticity genes expression and spine density in HIV infected neurons: role of nicotine in progression of HIV-associated neurocognitive disorder.HIV-1 neuroimmunity in the era of antiretroviral therapy.Glutamate metabolism and HIV-associated neurocognitive disorders.Gap junctions and blood-tissue barriers PrPC, the cellular isoform of the human prion protein, is a novel biomarker of HIV-associated neurocognitive impairment and mediates neuroinflammation The role of gap junction channels during physiologic and pathologic conditions of the human central nervous system Cytochrome C dysregulation induced by HIV infection of astrocytes results in bystander apoptosis of uninfected astrocytes by an IP3 and calcium-dependent mechanism.Human synaptic plasticity gene expression profile and dendritic spine density changes in HIV-infected human CNS cells: role in HIV-associated neurocognitive disorders (HAND).Mechanisms of HIV entry into the CNS: increased sensitivity of HIV infected CD14+CD16+ monocytes to CCL2 and key roles of CCR2, JAM-A, and ALCAM in diapedesis Drug induced increases in CNS dopamine alter monocyte, macrophage and T cell functions: implications for HAND.Characterization of monocyte maturation/differentiation that facilitates their transmigration across the blood-brain barrier and infection by HIV: implications for NeuroAIDS.Breaking down the barrier: the effects of HIV-1 on the blood-brain barrier.HIV increases the release of dickkopf-1 protein from human astrocytes by a Cx43 hemichannel-dependent mechanism Monocytes mediate HIV neuropathogenesis: mechanisms that contribute to HIV associated neurocognitive disorders Novel mechanisms of central nervous system damage in HIV infection.Effect of human immunodeficiency virus on blood-brain barrier integrity and function: an update.Dynasore disrupts trafficking of herpes simplex virus proteins Monocyte maturation, HIV susceptibility, and transmigration across the blood brain barrier are critical in HIV neuropathogenesis DJ1 expression downregulates in neuroblastoma cells (SK-N-MC) chronically exposed to HIV-1 and cocaine.HIV-Tat regulates macrophage gene expression in the context of neuroAIDS.HIV-1 gp120 induces type-1 programmed cell death through ER stress employing IRE1α, JNK and AP-1 pathway.Human immunodeficiency virus (HIV) infects human arterial smooth muscle cells in vivo and in vitro: implications for the pathogenesis of HIV-mediated vascular disease HIV-1, methamphetamine and astrocyte glutamate regulation: combined excitotoxic implications for neuro-AIDS.HIV-1 Tat-Mediated Calcium Dysregulation and Neuronal Dysfunction in Vulnerable Brain Regions.HIV-tat alters Connexin43 expression and trafficking in human astrocytes: role in NeuroAIDS.Targeting the glutamatergic system for the treatment of HIV-associated neurocognitive disorders Taurine suppresses the spread of cell death in electrically coupled RPE cells Pannexin1 hemichannels are critical for HIV infection of human primary CD4+ T lymphocytes.Glial connexins and gap junctions in CNS inflammation and disease.Neurological manifestations of oculodentodigital dysplasia: a Cx43 channelopathy of the central nervous system?

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P2860

Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

http://www.wikidata.org/entity/Q36226158

description

2007 nî lūn-bûn

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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2007年學術文章

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name

Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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type

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Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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prefLabel

Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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JNEUROSCI.4154-07.2007

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Journal of Neuroscience

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Gap junctions mediate human immunodeficiency virus-bystander killing in astrocytes

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Eliseo A Eugenin

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Joan W Berman

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P2860

MCP-1 (CCL2) protects human neurons and astrocytes from NMDA or HIV-tat-induced apoptosis

DC-SIGN, a dendritic cell-specific HIV-1-binding protein that enhances trans-infection of T cells

The neuropathogenesis of AIDS

Astrocytic gap junctional communication decreases neuronal vulnerability to oxidative stress-induced disruption of Ca2+ homeostasis and cell death.

Brain-immune connection: immuno-regulatory properties of CNS-resident cells.

Gap junction uncoupler heptanol prevents cell-to-cell progression of hypercontracture and limits necrosis during myocardial reperfusion.

Dementia and neurocognitive disorders due to HIV-1 infection.

Prospects for rational development of pharmacological gap junction channel blockers.

Gap junctions and connexin expression in the normal and pathological central nervous system.

Neuroimmunity and the blood-brain barrier: molecular regulation of leukocyte transmigration and viral entry into the nervous system with a focus on neuroAIDS.

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