A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells
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HLA-B*35-Px-mediated acceleration of HIV-1 infection by increased inhibitory immunoregulatory impulsesLeukocyte Ig-Like Receptors - A Model for MHC Class I Disease AssociationsHLA-dependent tumour development: a role for tumour associate macrophages?Loss of viral fitness and cross-recognition by CD8+ T cells limit HCV escape from a protective HLA-B27–restricted human immune responseCrystal structures of the two membrane-proximal Ig-like domains (D3D4) of LILRB1/B2: alternative models for their involvement in peptide-HLA bindingThe inhibitory receptor LILRB4 (ILT3) modulates antigen presenting cell phenotype and, along with LILRB2 (ILT4), is upregulated in response to Salmonella infection.NK cells in HIV-1 infection: evidence for their role in the control of HIV-1 infection.Mutational escape in HIV-1 CTL epitopes leads to increased binding to inhibitory myelomonocytic MHC class I receptors.Signaling and ligand interaction of ILT7: receptor-mediated regulatory mechanisms for plasmacytoid dendritic cellsLeukocyte immunoglobulin-like receptors maintain unique antigen-presenting properties of circulating myeloid dendritic cells in HIV-1-infected elite controllersPediatric HIV Long-Term Nonprogressors.Prolonged control of an HIV type 1 escape variant following treatment interruption in an HLA-B*27-positive patientRapid antigen processing and presentation of a protective and immunodominant HLA-B*27-restricted hepatitis C virus-specific CD8+ T-cell epitope.Transcriptional profiling of CD4 T cells identifies distinct subgroups of HIV-1 elite controllersEscape is a more common mechanism than avidity reduction for evasion of CD8+ T cell responses in primary human immunodeficiency virus type 1 infection.LILRB2 interaction with HLA class I correlates with control of HIV-1 infection.Escape from highly effective public CD8+ T-cell clonotypes by HIVHigh-functional-avidity cytotoxic T lymphocyte responses to HLA-B-restricted Gag-derived epitopes associated with relative HIV controlProtective effect of human leukocyte antigen B27 in hepatitis C virus infection requires the presence of a genotype-specific immunodominant CD8+ T-cell epitopeSelection and accumulation of an HIV-1 escape mutant by three types of HIV-1-specific cytotoxic T lymphocytes recognizing wild-type and/or escape mutant epitopes.Systemic inhibition of myeloid dendritic cells by circulating HLA class I molecules in HIV-1 infection.Escape from the dominant HLA-B27-restricted cytotoxic T-lymphocyte response in Gag is associated with a dramatic reduction in human immunodeficiency virus type 1 replicationStructural insights into activation of antiviral NK cell responsesAnalysis of the Expression and Function of Immunoglobulin-Like Transcript 4 (ILT4, LILRB2) in Dendritic Cells from Patients with Systemic Lupus Erythematosus.Structural and functional constraints limit options for cytotoxic T-lymphocyte escape in the immunodominant HLA-B27-restricted epitope in human immunodeficiency virus type 1 capsid.Comprehensive immunological evaluation reveals surprisingly few differences between elite controller and progressor Mamu-B*17-positive simian immunodeficiency virus-infected rhesus macaques.Evidence for natural selection on leukocyte immunoglobulin-like receptors for HLA class I in Northeast Asians.Immune-driven recombination and loss of control after HIV superinfection.Immunogenetics of HIV disease.Immunogenetics of spontaneous control of HIVTransmission and long-term stability of compensated CD8 escape mutations.The first T cell response to transmitted/founder virus contributes to the control of acute viremia in HIV-1 infection.Susceptibility to CD8 T-cell-mediated killing influences the reservoir of latently HIV-1-infected CD4 T cellsCD8+ T cells from HLA-B*57 elite suppressors effectively suppress replication of HIV-1 escape mutants.Regulation of T-cell immunity by leucocyte immunoglobulin-like receptors: innate immune receptors for self on antigen-presenting cells.Effect of multiple infection of cells on the evolutionary dynamics of HIV in vivo: implications for host adaptation mechanisms.The emerging role of leukocyte immunoglobulin-like receptors (LILRs) in HIV-1 infection.The nature of activatory and tolerogenic dendritic cell-derived signal II.Molecular signatures of T-cell inhibition in HIV-1 infection.Regulation of neutrophil functions through inhibitory receptors: an emerging paradigm in health and disease.
P2860
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P2860
A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@ast
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@en
type
label
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@ast
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@en
prefLabel
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@ast
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@en
P2093
P2860
P50
P356
P1476
A viral CTL escape mutation le ...... bition of myelomonocytic cells
@en
P2093
Beenu Moza
Daniel G Kavanagh
Eric J Sundberg
Katie L Williams
Margaret Feeney
Mathias Lichterfeld
Rachel Allen
Rachel Allgaier
Rajesh T Gandhi
Rohini Sivamurthy
P2860
P304
P356
10.1084/JEM.20061865
P407
P50
P577
2007-11-19T00:00:00Z