Tumor necrosis factor is a critical mediator in hapten induced irritant and contact hypersensitivity reactions.
about
Epoxomicin, a potent and selective proteasome inhibitor, exhibits in vivo antiinflammatory activityT cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine productionp85alpha acts as a novel signal transducer for mediation of cellular apoptotic response to UV radiationImmune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune reBroad-spectrum sunscreens prevent the secretion of proinflammatory cytokines in human keratinocytes exposed to ultraviolet A and phototoxic lomefloxacin.MMP19 is essential for T cell development and T cell-mediated cutaneous immune responsesIn vivo maturation and migration of dendritic cells.Epidermal cytokines in murine cutaneous irritant responses.Analysis of tumor necrosis factor promoter responses to ultraviolet light.N-acetylcysteine attenuates hexavalent chromium-induced hypersensitivity through inhibition of cell death, ROS-related signaling and cytokine expression.Effects of tumor necrosis factor alpha on dendritic cell accumulation in lymph nodes draining the immunization site and the impact on the anticryptococcal cell-mediated immune responseIrritant dermatitis, irritancy and its role in allergic contact dermatitis.Biological response modifiers and their potential use in the treatment of inflammatory skin diseases.Hapten application to the skin induces an inflammatory program directing hapten-primed effector CD8 T cell interaction with hapten-presenting endothelial cellsCutaneous barrier perturbation stimulates cytokine production in the epidermis of mice.Novel predictive assay for contact allergens using human skin explant cultures.Treatment of irritant and allergic contact dermatitis.Neutrophil expression of Fas ligand and perforin directs effector CD8 T cell infiltration into antigen-challenged skin.TNFSF15 Modulates Neovascularization and InflammationRegulation of contact hypersensitivity by interleukin 10.Mast cells control neutrophil recruitment during T cell-mediated delayed-type hypersensitivity reactions through tumor necrosis factor and macrophage inflammatory protein 2.Required early complement activation in contact sensitivity with generation of local C5-dependent chemotactic activity, and late T cell interferon gamma: a possible initiating role of B cells.CD40-CD40 ligand interactions in vivo regulate migration of antigen-bearing dendritic cells from the skin to draining lymph nodes.Mechanisms of drug-induced delayed-type hypersensitivity reactions in the skinEffects of the tumor microenvironment on the efficacy of tumor immunotherapy.Early molecular events in the induction phase of contact sensitivity.Pharmacotherapy for allergic contact dermatitis.Regulation of inflammation by collagen-binding integrins alpha1beta1 and alpha2beta1 in models of hypersensitivity and arthritis.Immunological mechanisms of contact hypersensitivity in mice.Cytokines and chemokines in irritant contact dermatitisIL-12 is produced by antigen-presenting cells stimulated with soluble alphabeta TCR and restores impaired T(h)1 responses.Effects of immunological responsiveness on Langerhans cell behavior in contact sensitization.Tumour necrosis factor-alpha mediates ultraviolet light B-enhanced expression of contact hypersensitivity.Immunology and genetics of tumour necrosis factor in allergic contact dermatitis.Tumor necrosis factor expression during mouse hepatitis virus-induced demyelinating encephalomyelitis.The 55-kD tumor necrosis factor receptor on human keratinocytes is regulated by tumor necrosis factor-alpha and by ultraviolet B radiation.The role of cytokines and neuroendocrine hormones in cutaneous immunity and inflammation.Chemical and biological evaluation of endotoxin contamination on natural rubber latex products.Mechanism in allergic contact dermatitis.Keratinocyte-derived cytokines and UVB-induced immunosuppression.
P2860
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P2860
Tumor necrosis factor is a critical mediator in hapten induced irritant and contact hypersensitivity reactions.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@ast
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@en
type
label
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@ast
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@en
prefLabel
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@ast
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@en
P2093
P2860
P356
P1476
Tumor necrosis factor is a cri ...... ct hypersensitivity reactions.
@en
P2093
P2860
P304
P356
10.1084/JEM.173.3.673
P407
P577
1991-03-01T00:00:00Z