Neutrophil-mediated dissolution of infected host cells as a defense strategy against a facultative intracellular bacterium.
about
In vivo involvement of polymorphonuclear neutrophils in Leishmania infantum infectionSurface proteins of gram-positive bacteria and mechanisms of their targeting to the cell wall envelope.Administration of superantigens protects mice from lethal Listeria monocytogenes infection by enhancing cytotoxic T cells.An anti-CD3 monoclonal antibody protects mice against a lethal infection with Listeria monocytogenes through induction of endogenous cytokinesNeutrophils are involved in acute, nonspecific resistance to Listeria monocytogenes in mice.Effector role of blood monocytes in experimental visceral leishmaniasisDisruption of the cellular inflammatory response to Listeria monocytogenes infection in mice with disruptions in targeted genes.Role of polymorphonuclear neutrophils in a murine model of Chlamydia psittaci-induced abortion.Infection-stimulated fibrin deposition controls hemorrhage and limits hepatic bacterial growth during listeriosisIndependent protective effects for tumor necrosis factor and lymphotoxin alpha in the host response to Listeria monocytogenes infection.Listeria pathogenesis and molecular virulence determinantsPolymorphonuclear neutrophils are necessary for the recruitment of CD8(+) T cells in the liver in a pregnant mouse model of Chlamydophila abortus (Chlamydia psittaci serotype 1) infection.Sex-dependent susceptibility to Listeria monocytogenes infection is mediated by differential interleukin-10 production.Listeria monocytogenes is resistant to lysozyme through the regulation, not the acquisition, of cell wall-modifying enzymes.Impaired antibacterial host defense in mice lacking the N-formylpeptide receptor.Differential inlA and inlB expression and interaction with human intestinal and liver cells by Listeria monocytogenes strains of different origins.Experimental validation of low virulence in field strains of Listeria monocytogenesA recombinant minigene vaccine containing a nonameric cytotoxic-T-lymphocyte epitope confers limited protection against Listeria monocytogenes infectionIncreased susceptibility to primary infection with Listeria monocytogenes in germfree mice may be due to lack of accumulation of L-selectin+ CD44+ T cells in sites of inflammation.Minimal requirements for murine resistance to infection with Francisella tularensis LVS.Role of neutrophils in controlling early stages of a Chlamydia trachomatis infection.Elimination of resident macrophages from the livers and spleens of immune mice impairs acquired resistance against a secondary Listeria monocytogenes infection.CD95 (Fas) may control the expansion of activated T cells after elimination of bacteria in murine listeriosis.Listeriolysin and IrpA are major protein targets of the human humoral response against Listeria monocytogenesCritical roles of neutrophils in host defense against experimental systemic infections of mice by Listeria monocytogenes, Salmonella typhimurium, and Yersinia enterocoliticaA comparison of T cell memory against the same antigen induced by virus versus intracellular bacteria.Compartmentalization of the broad-range phospholipase C activity to the spreading vacuole is critical for Listeria monocytogenes virulence.Improved antibacterial host defense and altered peripheral granulocyte homeostasis in mice lacking the adhesion class G protein receptor CD97.B Lymphocytes provide an infection niche for intracellular bacterium Brucella abortus.The inlAB locus mediates the entry of Listeria monocytogenes into hepatocytes in vivoCrucial role of interferon consensus sequence binding protein, but neither of interferon regulatory factor 1 nor of nitric oxide synthesis for protection against murine listeriosisInducible nitric oxide is essential for host control of persistent but not acute infection with the intracellular pathogen Toxoplasma gondiiImpact of the Listeria monocytogenes protein InlC on infection in mice.Protective immunity and granuloma formation are mediated by two distinct tumor necrosis factor alpha- and gamma interferon-dependent T cell-phagocyte interactions in murine listeriosis: dissociation on the basis of phagocyte adhesion mechanisms.Roles of Listeria monocytogenes virulence factors in survival: virulence factors distinct from listeriolysin are needed for the organism to survive an early neutrophil-mediated host defense mechanism.Regulation of Apoptosis by Gram-Positive Bacteria: Mechanistic Diversity and Consequences for Immunity.Use of animal models in the development of human vaccines.Testing human biologicals in animal host resistance models.Identification of an IFN-gamma-producing neutrophil early in the response to Listeria monocytogenesA novel IL-17-dependent mechanism of cross protection: respiratory infection with mycoplasma protects against a secondary listeria infection.
P2860
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P2860
Neutrophil-mediated dissolution of infected host cells as a defense strategy against a facultative intracellular bacterium.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@ast
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@en
type
label
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@ast
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@en
prefLabel
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@ast
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@en
P2860
P356
P1476
Neutrophil-mediated dissolutio ...... ative intracellular bacterium.
@en
P2093
P2860
P304
P356
10.1084/JEM.174.3.741
P407
P577
1991-09-01T00:00:00Z