Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection. - Wikidata - awgldk - TriplyDB

Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection.

Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection.

http://www.wikidata.org/entity/Q36230778

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Sensitivity of Dendritic Cells to Microenvironment Signals Antigenicity and immunogenicity of allogeneic retinal transplants NK cell patrolling and elimination of donor-derived dendritic cells favor indirect alloreactivity Immunogenicity of unprocessed and photooxidized bovine and human osteochondral grafts in collagen-sensitive mice Roles of Host Nonhematopoietic Cells in Autoimmunity and Donor Cell Engraftment in Graft-versus-host Disease.Immunology of naturally transmissible tumours.Recent developments in the pharmacological treatment and prevention of corneal graft rejection.Antigen location contributes to the pathological features of a transplanted heart graft.The indirect alloresponse impairs the induction but not maintenance of tolerance to MHC class I-disparate allografts.Autoantibodies to vimentin cause accelerated rejection of cardiac allografts.Mixed chimerism and split tolerance: mechanisms and clinical correlations.Bidirectional alloreactivity: A proposed microchimerism-based solution to the NIMA paradox.Contribution of direct and indirect recognition pathways to T cell alloreactivity.Immunologically nonspecific mechanisms of tissue destruction in the rejection of skin grafts.Efficient presentation of phagocytosed cellular fragments on the major histocompatibility complex class II products of dendritic cells.Dendritic cells as promoters of transplant tolerance.Extracellular matrix-based biomaterial scaffolds and the host response.Interleukin-23 secretion by donor antigen-presenting cells is critical for organ-specific pathology in graft-versus-host disease.Differential roles of direct and indirect allorecognition pathways in the rejection of skin and corneal transplants.Exosomes: The missing link between microchimerism and acquired tolerance?T cell receptor biases and clonal proliferations among lung transplant recipients with obliterative bronchiolitis.Indirect recognition of donor HLA-DR peptides in organ allograft rejection.T cells reactive to a single immunodominant self-restricted allopeptide induce skin graft rejection in mice.Mechanism of cellular rejection in transplantation Donor exosomes rather than passenger leukocytes initiate alloreactive T cell responses after transplantation Autologous and Allogenous Antibodies in Lung and Islet Cell Transplantation.Natural killer cells in rejection and tolerance of solid organ allografts Loss of tolerance to self after transplant.Immune recognition and rejection of allogeneic skin grafts.Contributions of direct and indirect alloresponses to chronic rejection of kidney allografts in nonhuman primates.A Paradigm Shift on the Question of B Cells in Transplantation? Recent Insights on Regulating the Alloresponse.The role of MHC genes in contagious cancer: the story of Tasmanian devils.Mechanisms of oral tolerance by MHC peptides.New approaches to specific immunomodulation in transplantation.T cell receptor (TCR) repertoire in alloimmune responses.Role of indirect allorecognition in allograft rejection.The contribution of peptides to T cell allorecognition and allograft rejection.Indirect T cell recognition in allograft rejection.The role of polymorphic donor peptides in allograft recognition and rejection.Suppression of the indirect pathway of T cell reactivity by high doses of allopeptide.

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P2860

Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection.

http://www.wikidata.org/entity/Q36230778

description

1992 nî lūn-bûn

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1992年の論文

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1992年論文

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1992年論文

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1992年論文

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1992年論文

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1992年論文

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1992年论文

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1992年论文

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1992年论文

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name

Donor major histocompatibility ...... ecules during graft rejection.

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Donor major histocompatibility ...... ecules during graft rejection.

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type

label

Donor major histocompatibility ...... ecules during graft rejection.

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Donor major histocompatibility ...... ecules during graft rejection.

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prefLabel

Donor major histocompatibility ...... ecules during graft rejection.

@ast

Donor major histocompatibility ...... ecules during graft rejection.

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P1433

Journal of Experimental Medicine

P1476

Donor major histocompatibility ...... ecules during graft rejection.

@en

P2093

C A Olson

http://www.w3.org/2001/XMLSchema#string

E E Sercarz

http://www.w3.org/2001/XMLSchema#string

G Benichou

http://www.w3.org/2001/XMLSchema#string

M McMillan

http://www.w3.org/2001/XMLSchema#string

P A Takizawa

http://www.w3.org/2001/XMLSchema#string

P2860

Creation of H-2 class I epitopes using synthetic peptides: recognition by alloreactive cytotoxic T lymphocytes.

Allorecognition of DR1 by T cells from a DR4/DRw13 responder mimics self-restricted recognition of endogenous peptides.

Working principles in the immune system implied by the "peptidic self" model.

Immunogenicity and tolerogenicity of self-major histocompatibility complex peptides.

I-A alpha polymorphic residues that determine alloreactive T cell recognition.

Transgenic mice expressing a soluble foreign H-2 class I antigen are tolerant to allogeneic fragments presented by self class I but not to the whole membrane-bound alloantigen.

Studies in histocompatibility.

Corecognition of HLA-A1 and HLA-DPw3 by a human CD4+ alloreactive T lymphocyte clone

T cells sensitized to synthetic HLA-DR3 peptide give evidence of continuous presentation of denatured HLA-DR3 molecules by HLA-DP.

Differential involvement of CD4+ cells in mediating skin graft rejection against different amounts of transgenic H-2K(b) antigen.

P304

305-308

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scholarly article

P356

10.1084/JEM.175.1.305

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1

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175

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1992-01-01T00:00:00Z

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1730925

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2119070

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