N-type calcium channel inhibition with cilnidipine elicits glomerular podocyte protection independent of sympathetic nerve inhibition

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Ablation of the N-type calcium channel ameliorates diabetic nephropathy with improved glycemic control and reduced blood pressure.Angiotensin-converting enzyme inhibitor does not suppress renal angiotensin II levels in angiotensin I-infused rats.

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N-type calcium channel inhibition with cilnidipine elicits glomerular podocyte protection independent of sympathetic nerve inhibition

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2012 nî lūn-bûn

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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N-type calcium channel inhibit ...... f sympathetic nerve inhibition

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Akira Nishiyama

http://www.w3.org/2001/XMLSchema#string

Bai Lei

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Daisuke Nakano

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Hirofumi Hitomi

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Hirohito Mori

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Hiroyuki Kobori

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Katsuhiko Asanuma

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Tsutomu Masaki

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Ya Liu

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Yasuhiko Tomino

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P2860

Synaptopodin orchestrates actin organization and cell motility via regulation of RhoA signalling

Angiotensin II increases the intracellular calcium activity in podocytes of the intact glomerulus.

Cilnidipine suppresses podocyte injury and proteinuria in metabolic syndrome rats: possible involvement of N-type calcium channel in podocyte.

Urinary angiotensinogen is correlated with blood pressure in men (Bogalusa Heart Study).

Disease-dependent mechanisms of albuminuria.

Renal sympathetic denervation in patients with treatment-resistant hypertension (The Symplicity HTN-2 Trial): a randomised controlled trial.

Nebivolol attenuates redox-sensitive glomerular and tubular mediated proteinuria in obese rats.

Catheter-based renal sympathetic denervation for resistant hypertension: a multicentre safety and proof-of-principle cohort study.

Salt-induced renal injury in SHRs is mediated by AT1 receptor activation.

The intrarenal renin-angiotensin system: from physiology to the pathobiology of hypertension and kidney disease.

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359-367

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scholarly article

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10.1254/JPHS.12075FP

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119

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2012-07-21T00:00:00Z

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22863666

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3443282

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N-type calcium channel inhibition with cilnidipine elicits glomerular podocyte protection independent of sympathetic nerve inhibition

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N-type calcium channel inhibition with cilnidipine elicits glomerular podocyte protection independent of sympathetic nerve inhibition

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P1476

P2093

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P304

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P2860

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