Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
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Early Repolarization Syndrome; Mechanistic Theories and Clinical CorrelatesPeroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac FibrosisJ Wave Syndromes: History and Current ControversiesElectrophysiological Mechanisms of Brugada Syndrome: Insights from Pre-clinical and Clinical StudiesBrugada Syndrome:Risk Stratification And ManagementJ-Wave syndromes expert consensus conference report: Emerging concepts and gaps in knowledgehiPSC-derived cardiomyocytes from Brugada Syndrome patients without identified mutations do not exhibit clear cellular electrophysiological abnormalitiesLocal Left Ventricular Epicardial J Waves and Late Potentials in Brugada Syndrome Patients with Inferolateral Early Repolarization Pattern.Vinculin variant M94I identified in sudden unexplained nocturnal death syndrome decreases cardiac sodium current.Ion channels, long QT syndrome and arrhythmogenesis in ageing.Is VF an Ablatable Rhythm?Pathogenesis and management of Brugada syndrome.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Towards an integrated understanding of cardiac arrhythmogenesis - Growing roles of experimental pathology.Impact of premature activation of the right ventricle with programmed stimulation in Brugada syndrome.Does Sudden Unexplained Nocturnal Death Syndrome Remain the Autopsy-Negative Disorder: A Gross, Microscopic, and Molecular Autopsy Investigation in Southern China.Unmasking the molecular link between arrhythmogenic cardiomyopathy and Brugada syndrome.Differences in Left Versus Right Ventricular Electrophysiological Properties in Cardiac Dysfunction and Arrhythmogenesis.Recording of isolated very delayed potentials on the right ventricular epicardium in a patient with Brugada syndrome.An East Asian Common Variant Vinculin P.Asp841His Was Associated With Sudden Unexplained Nocturnal Death Syndrome in the Chinese Han Population.Synergism between Enhanced Late Inward Currents and Tissue Fibrosis in the Initiation of Spontaneous Ventricular TachyarrhythmiasAbnormal epicardial electrophysiologic substrate in patients with early repolarization pattern and reduced left ventricular systolic function: A report of two cases.Searching for mechanical abnormalities in subjects with early repolarization pattern: another Holy Grail of cardiac imaging?SCN5A Genetic Polymorphisms Associated With Increased Defibrillator Shocks in Brugada Syndrome.Ajmaline blocks INa and IKr without eliciting differences between Brugada syndrome patient and control human pluripotent stem cell-derived cardiac clusters.Fractionated electrograms with ST-segment elevation recorded from the human right ventricular outflow tract.Normal interventricular differences in tissue architecture underlie right ventricular susceptibility to conduction abnormalities in a mouse model of Brugada syndrome.Fibroblasts and the extracellular matrix in right ventricular disease.Right ventricular scar-related ventricular tachycardia in nonischemic cardiomyopathy: Electrophysiological characteristics, mapping, and ablation of underlying heart disease.The relationship between J waves and contact of lung cancer with the heart.Epicardial ablation utilizing remote magnetic navigation in a patient with Brugada syndrome and inferior early repolarization.Epicardial Brugada syndrome ablation unmasking inferior J waves.Hypothetical "anatomy" of Brugada phenomenon: "Long QT sine Long QT" syndrome implicating morphologically undefined specific "Brugada's myocells".Mechanisms Underlying Epicardial Radiofrequency Ablation to Suppress Arrhythmogenesis in Experimental Models of Brugada Syndrome.Analysis of the High-Frequency Content in Human QRS Complexes by the Continuous Wavelet Transform: An Automatized Analysis for the Prediction of Sudden Cardiac Death.Right ventricular outflow tract high-density endocardial unipolar voltage mapping in patients with Brugada syndrome: evidence for electroanatomical abnormalities.J-Wave syndromes expert consensus conference report: Emerging concepts and gaps in knowledge.Relations between right ventricular morphology and clinical, electrical and genetic parameters in Brugada Syndrome.Calcium in Brugada Syndrome: Questions for Future ResearchAssessment of a conduction-repolarisation metric to predict Arrhythmogenesis in right ventricular disorders
P2860
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P2860
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
@ast
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
@en
type
label
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
@ast
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
@en
prefLabel
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
@ast
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome.
@en
P2093
P2860
P50
P1476
Fibrosis, Connexin-43, and Conduction Abnormalities in the Brugada Syndrome
@en
P2093
Akihiko Nogami
Allard C van der Wal
Arthur A Wilde
Gumpanart Veerakul
Hanno L Tan
Koonlawee Nademanee
Laurence Robinson
Michael Christiansen
Michael Papadakis
Nakorn Boonmee
P2860
P304
P356
10.1016/J.JACC.2015.08.862
P407
P577
2015-11-01T00:00:00Z