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Akt activity protects rheumatoid synovial fibroblasts from Fas-induced apoptosis by inhibition of Bid cleavageUse of Lentiviral Particles As a Cell Membrane-Based mFasL Delivery System for In Vivo Treatment of Inflammatory Arthritis.The microbial capsular polysaccharide galactoxylomannan inhibits IL-17A production in circulating T cells from rheumatoid arthritis patients.Targeting Fas in osteoresorptive disorders.IL-17-mediated Bcl-2 expression regulates survival of fibroblast-like synoviocytes in rheumatoid arthritis through STAT3 activationSmoking and rheumatoid arthritis.Soluble Fas ligand inhibits angiogenesis in rheumatoid arthritisB lymphocyte-typing for prediction of clinical response to rituximab.Expression of high levels of human proteinase inhibitor 9 blocks both perforin/granzyme and Fas/Fas ligand-mediated cytotoxicity.Pro-apoptotic effect of nonsteroidal anti-inflammatory drugs on synovial fibroblasts.Association of systemic and intra-articular osteoclastogenic potential, pro-inflammatory mediators and disease activity with the form of inflammatory arthritis.Expression of TNF-alpha and related signaling molecules in the peripheral blood mononuclear cells of rheumatoid arthritis patients.Negative Regulators of JAK/STAT Signaling in Rheumatoid Arthritis and Osteoarthritis.Increased autophagy in CD4+ T cells of rheumatoid arthritis patients results in T-cell hyperactivation and apoptosis resistance.Keynote lecture in the 13th Japanese Society of Immunotoxicology (JSIT 2006) : -Pathophysiological Development and Immunotoxicology: what we have found from research related to silica and silicate such as asbestos-.Bi(o)communications among peripheral blood fractions: a focus on NK and NKT cell biology in rheumatoid arthritis.Calycopterin, an immunoinhibitory compound from the extract of Dracocephalum kotschyi.Role of cytolytic impairment of natural killer and natural killer T-cell populations in rheumatoid arthritis.Characterization of effector memory CD8+ T cells in the synovial fluid of rheumatoid arthritis.Distinct effects of soluble and membrane-bound fas ligand on fibroblast-like synoviocytes from rheumatoid arthritis patients.B cell resistance to Fas-mediated apoptosis contributes to their ineffective control by regulatory T cells in rheumatoid arthritis.Uncovering Cellular retinoic acid-binding protein 2 as a potential target for rheumatoid arthritis synovial hyperplasia.
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@ast
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@en
type
label
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@ast
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@en
prefLabel
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@ast
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@en
P2860
P356
P1433
P1476
Fas (CD95)-related apoptosis and rheumatoid arthritis.
@en
P2093
P2860
P356
10.1093/RHEUMATOLOGY/KEI113
P577
2005-09-13T00:00:00Z