Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
about
The Apoe(-/-) mouse model: a suitable model to study cardiovascular and respiratory diseases in the context of cigarette smoke exposure and harm reductionPrenatal polycyclic aromatic hydrocarbon, adiposity, peroxisome proliferator-activated receptor (PPAR) γ methylation in offspring, grand-offspring mice.Urban particulate matter triggers lung inflammation via the ROS-MAPK-NF-κB signaling pathway.
P2860
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
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2012 nî lūn-bûn
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name
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@ast
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@en
type
label
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@ast
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@en
prefLabel
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@ast
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@en
P2093
P2860
P921
P1476
Exposure to diesel exhaust upregulates COX-2 expression in ApoE knockout mice.
@en
P2093
Michael E Rosenfeld
Stephan F van Eeden
Terrance J Kavanagh
P2860
P2880
P304
P356
10.3109/08958378.2012.696221
P577
2012-07-01T00:00:00Z