Tandem double CC-->TT mutations are produced by reactive oxygen species.
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Oxidative DNA damage by vitamin A and its derivative via superoxide generationImpaired spermatogenesis and elevated spontaneous tumorigenesis in xeroderma pigmentosum group A gene (Xpa)-deficient mice.Oxyradicals and DNA damage.Oxidative DNA damage induced by copper and hydrogen peroxide promotes CG-->TT tandem mutations at methylated CpG dinucleotides in nucleotide excision repair-deficient cells.A role for ultraviolet A in solar mutagenesis.DNA strand breaks in human nasal respiratory epithelium are induced upon exposure to urban pollution.Molecular epidemiology in environmental health: the potential of tumor suppressor gene p53 as a biomarker.5-Hydroxypyrimidine deoxynucleoside triphosphates are more efficiently incorporated into DNA by exonuclease-free Klenow fragment than 8-oxopurine deoxynucleoside triphosphatesUV signature mutations.p53 mutations in non-small-cell lung cancers occurring in individuals without a past history of active smoking.Distinct effects of ultraviolet B light on antioxidant expression in undifferentiated and differentiated mouse keratinocytes.Specific UV-induced mutation spectrum in the p53 gene of skin tumors from DNA-repair-deficient xeroderma pigmentosum patientsIdentification and quantification of a guanine-thymine intrastrand cross-link lesion induced by Cu(II)/H2O2/ascorbateOccurrence, Biological Consequences, and Human Health Relevance of Oxidative Stress-Induced DNA Damage.Identification and characterization of a novel cross-link lesion in d(CpC) upon 365-nm irradiation in the presence of 2-methyl-1,4-naphthoquinone.High frequency induction of CC to TT tandem mutations in DNA repair-proficient mammalian cells.p53 gene mutations and photocarcinogenesis.One thousand somatic SNVs per skin fibroblast cell set baseline of mosaic mutational load with patterns that suggest proliferative origin.Possible role of oxidative damage in metal-induced carcinogenesis.DNA damages processed by base excision repair: biological consequences.What are the critical attributes for cancer chemopreventive agents?Damage to DNA by reactive oxygen and nitrogen species: role in inflammatory disease and progression to cancerTandem-base mutations occur in mouse liver and adipose tissue preferentially as G:C to T:A transversions and accumulate with age.Ultraviolet Radiations: Skin Defense-Damage Mechanism.Protein oxidation and DNA-protein crosslink induced by sulfur dioxide in lungs, livers, and hearts from mice.Pan-cancer genome and transcriptome analyses of 1,699 paediatric leukaemias and solid tumours.Inhibition of cyclobutane pyrimidine dimer formation in epidermal p53 gene of UV-irradiated mice by alpha-tocopherol.Absence of ras mutations and low incidence of p53 mutations in renal cell carcinomas induced by ferric nitrilotriacetate.Far-UV-lnduced Dimeric Photoproducts in Short Oligonucleotides: Sequence EffectsOrigin of Multiple Mutations in Human Cancers
P2860
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P2860
Tandem double CC-->TT mutations are produced by reactive oxygen species.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@ast
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@en
type
label
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@ast
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@en
prefLabel
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@ast
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@en
P2860
P356
P1476
Tandem double CC-->TT mutations are produced by reactive oxygen species.
@en
P2093
P2860
P304
P356
10.1073/PNAS.90.9.3904
P407
P577
1993-05-01T00:00:00Z