Modulation of the cAMP signaling pathway after traumatic brain injury. - Wikidata - awgldk - TriplyDB

Modulation of the cAMP signaling pathway after traumatic brain injury.

Modulation of the cAMP signaling pathway after traumatic brain injury.

http://www.wikidata.org/entity/Q36276090

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The therapeutic profile of rolipram, PDE target and mechanism of action as a neuroprotectant following spinal cord injury Traumatic brain injury: can the consequences be stopped?Phosphodiesterase Inhibitors as a Therapeutic Approach to Neuroprotection and Repair.Pericontusion axon sprouting is spatially and temporally consistent with a growth-permissive environment after traumatic brain injury.Key role of sulfonylurea receptor 1 in progressive secondary hemorrhage after brain contusion Experimental traumatic brain injury alters ethanol consumption and sensitivity.Post-traumatic seizures exacerbate histopathological damage after fluid-percussion brain injury.Glucagon protects against impaired NMDA-mediated cerebrovasodilation and cerebral autoregulation during hypotension after brain injury by activating cAMP protein kinase A and inhibiting upregulation of tPA.Post-traumatic seizure susceptibility is attenuated by hypothermia therapy.Altered regulation of protein kinase a activity in the medial prefrontal cortex of normal and brain-injured animals actively engaged in a working memory task.A selective phosphodiesterase-4 inhibitor reduces leukocyte infiltration, oxidative processes, and tissue damage after spinal cord injury.Exendin-4 ameliorates traumatic brain injury-induced cognitive impairment in rats.Proinflammatory cytokine regulation of cyclic AMP-phosphodiesterase 4 signaling in microglia in vitro and following CNS injury.Neuron-targeted caveolin-1 protein enhances signaling and promotes arborization of primary neurons.The effect of phosphodiesterase-4-specific inhibitor in the rat model of spinal nerve ligation.Intrathecal injection of adenosine 2A receptor agonists reversed neuropathic allodynia through protein kinase (PK)A/PKC signaling.In vitro stretch injury induces time- and severity-dependent alterations of STEP phosphorylation and proteolysis in neurons.Postinjury treatment with rolipram increases hemorrhage after traumatic brain injury Electrophysiological and behavioral changes by phosphodiesterase 4 inhibitor in a rat model of alcoholic neuropathy.Caveolins: targeting pro-survival signaling in the heart and brain.Therapeutic benefits of phosphodiesterase 4B inhibition after traumatic brain injury.Phosphodiesterase isoform-specific expression induced by traumatic brain injury.Decoding hippocampal signaling deficits after traumatic brain injury Neuroprotective effect of Pycnogenol® following traumatic brain injury.Traumatic Brain Injury Upregulates Phosphodiesterase Expression in the Hippocampus.Age-dependent alterations in cAMP signaling contribute to synaptic plasticity deficits following traumatic brain injury Cyclic adenosine monophosphate and brain-derived neurotrophic factor decreased oxidative stress and apoptosis in developing hypothalamic neuronal cells: role of microglia Chronic Cognitive Dysfunction after Traumatic Brain Injury Is Improved with a Phosphodiesterase 4B Inhibitor.HDAC inhibitor increases histone H3 acetylation and reduces microglia inflammatory response following traumatic brain injury in rats Deficits in ERK and CREB activation in the hippocampus after traumatic brain injury.Adenosine neuromodulation and traumatic brain injury.Glucose-Dependent Insulinotropic Polypeptide Ameliorates Mild Traumatic Brain Injury-Induced Cognitive and Sensorimotor Deficits and Neuroinflammation in Rats.Efficacy of phosphodiesterase-4 inhibitors in juvenile Batten disease (CLN3).Systemic administration of propentofylline, ibudilast, and (+)-naltrexone each reverses mechanical allodynia in a novel rat model of central neuropathic pain.Phosphodiesterase inhibitors as therapeutics for traumatic brain injury.Treatment of traumatic brain injury with anti-inflammatory drugs.PDE4B as a microglia target to reduce neuroinflammation.Pathophysiology Associated with Traumatic Brain Injury: Current Treatments and Potential Novel Therapeutics.Predicting changes in cortical electrophysiological function after in vitro traumatic brain injury.Traumatic brain injury reduces striatal tyrosine hydroxylase activity and potassium-evoked dopamine release in rats

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Modulation of the cAMP signaling pathway after traumatic brain injury.

http://www.wikidata.org/entity/Q36276090

description

2007 nî lūn-bûn

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2007年学术文章

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Modulation of the cAMP signaling pathway after traumatic brain injury.

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Modulation of the cAMP signaling pathway after traumatic brain injury.

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Modulation of the cAMP signaling pathway after traumatic brain injury.

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Modulation of the cAMP signaling pathway after traumatic brain injury.

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Modulation of the cAMP signaling pathway after traumatic brain injury.

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Modulation of the cAMP signaling pathway after traumatic brain injury.

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J.EXPNEUROL.2007.08.011

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Experimental Neurology

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Modulation of the cAMP signaling pathway after traumatic brain injury

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Anthony A Oliva

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Damien D Pearse

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Helen M Bramlett

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Ofelia F Alonso

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W Dalton Dietrich

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P2860

Epac is a Rap1 guanine-nucleotide-exchange factor directly activated by cyclic AMP

Type VIII adenylyl cyclase. A Ca2+/calmodulin-stimulated enzyme expressed in discrete regions of rat brain

Mitogen- and stress-activated protein kinase-1 (MSK1) is directly activated by MAPK and SAPK2/p38, and may mediate activation of CREB

FGF and stress regulate CREB and ATF-1 via a pathway involving p38 MAP kinase and MAPKAP kinase-2

Rolipram, a type IV-specific phosphodiesterase inhibitor, facilitates the establishment of long-lasting long-term potentiation and improves memory

Impact of VIP and cAMP on the regulation of TNF-alpha and IL-10 production: implications for rheumatoid arthritis

Inhibition of microglial inflammatory responses by norepinephrine: effects on nitric oxide and interleukin-1beta production

Transcriptional regulation by the phosphorylation-dependent factor CREB

Coupling of the RAS-MAPK pathway to gene activation by RSK2, a growth factor-regulated CREB kinase

The induction of cyclic nucleotide phosphodiesterase 4 gene (PDE4D) impairs memory in a water maze task

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145-158

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10.1016/J.EXPNEUROL.2007.08.011

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Coleen M. Atkins

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2007-08-29T00:00:00Z

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5929364

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traumatic brain injury

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2141537

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