Modulation of the cAMP signaling pathway after traumatic brain injury.
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The therapeutic profile of rolipram, PDE target and mechanism of action as a neuroprotectant following spinal cord injuryTraumatic brain injury: can the consequences be stopped?Phosphodiesterase Inhibitors as a Therapeutic Approach to Neuroprotection and Repair.Pericontusion axon sprouting is spatially and temporally consistent with a growth-permissive environment after traumatic brain injury.Key role of sulfonylurea receptor 1 in progressive secondary hemorrhage after brain contusionExperimental traumatic brain injury alters ethanol consumption and sensitivity.Post-traumatic seizures exacerbate histopathological damage after fluid-percussion brain injury.Glucagon protects against impaired NMDA-mediated cerebrovasodilation and cerebral autoregulation during hypotension after brain injury by activating cAMP protein kinase A and inhibiting upregulation of tPA.Post-traumatic seizure susceptibility is attenuated by hypothermia therapy.Altered regulation of protein kinase a activity in the medial prefrontal cortex of normal and brain-injured animals actively engaged in a working memory task.A selective phosphodiesterase-4 inhibitor reduces leukocyte infiltration, oxidative processes, and tissue damage after spinal cord injury.Exendin-4 ameliorates traumatic brain injury-induced cognitive impairment in rats.Proinflammatory cytokine regulation of cyclic AMP-phosphodiesterase 4 signaling in microglia in vitro and following CNS injury.Neuron-targeted caveolin-1 protein enhances signaling and promotes arborization of primary neurons.The effect of phosphodiesterase-4-specific inhibitor in the rat model of spinal nerve ligation.Intrathecal injection of adenosine 2A receptor agonists reversed neuropathic allodynia through protein kinase (PK)A/PKC signaling.In vitro stretch injury induces time- and severity-dependent alterations of STEP phosphorylation and proteolysis in neurons.Postinjury treatment with rolipram increases hemorrhage after traumatic brain injuryElectrophysiological and behavioral changes by phosphodiesterase 4 inhibitor in a rat model of alcoholic neuropathy.Caveolins: targeting pro-survival signaling in the heart and brain.Therapeutic benefits of phosphodiesterase 4B inhibition after traumatic brain injury.Phosphodiesterase isoform-specific expression induced by traumatic brain injury.Decoding hippocampal signaling deficits after traumatic brain injuryNeuroprotective effect of Pycnogenol® following traumatic brain injury.Traumatic Brain Injury Upregulates Phosphodiesterase Expression in the Hippocampus.Age-dependent alterations in cAMP signaling contribute to synaptic plasticity deficits following traumatic brain injuryCyclic adenosine monophosphate and brain-derived neurotrophic factor decreased oxidative stress and apoptosis in developing hypothalamic neuronal cells: role of microgliaChronic Cognitive Dysfunction after Traumatic Brain Injury Is Improved with a Phosphodiesterase 4B Inhibitor.HDAC inhibitor increases histone H3 acetylation and reduces microglia inflammatory response following traumatic brain injury in ratsDeficits in ERK and CREB activation in the hippocampus after traumatic brain injury.Adenosine neuromodulation and traumatic brain injury.Glucose-Dependent Insulinotropic Polypeptide Ameliorates Mild Traumatic Brain Injury-Induced Cognitive and Sensorimotor Deficits and Neuroinflammation in Rats.Efficacy of phosphodiesterase-4 inhibitors in juvenile Batten disease (CLN3).Systemic administration of propentofylline, ibudilast, and (+)-naltrexone each reverses mechanical allodynia in a novel rat model of central neuropathic pain.Phosphodiesterase inhibitors as therapeutics for traumatic brain injury.Treatment of traumatic brain injury with anti-inflammatory drugs.PDE4B as a microglia target to reduce neuroinflammation.Pathophysiology Associated with Traumatic Brain Injury: Current Treatments and Potential Novel Therapeutics.Predicting changes in cortical electrophysiological function after in vitro traumatic brain injury.Traumatic brain injury reduces striatal tyrosine hydroxylase activity and potassium-evoked dopamine release in rats
P2860
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P2860
Modulation of the cAMP signaling pathway after traumatic brain injury.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Modulation of the cAMP signaling pathway after traumatic brain injury.
@ast
Modulation of the cAMP signaling pathway after traumatic brain injury.
@en
type
label
Modulation of the cAMP signaling pathway after traumatic brain injury.
@ast
Modulation of the cAMP signaling pathway after traumatic brain injury.
@en
prefLabel
Modulation of the cAMP signaling pathway after traumatic brain injury.
@ast
Modulation of the cAMP signaling pathway after traumatic brain injury.
@en
P2093
P2860
P1476
Modulation of the cAMP signaling pathway after traumatic brain injury
@en
P2093
Anthony A Oliva
Damien D Pearse
Helen M Bramlett
Ofelia F Alonso
W Dalton Dietrich
P2860
P304
P356
10.1016/J.EXPNEUROL.2007.08.011
P407
P577
2007-08-29T00:00:00Z