Transgenic APP expression during postnatal development causes persistent locomotor hyperactivity in the adult.
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Toward more predictive genetic mouse models of Alzheimer's diseaseMonoacylglycerol lipase inhibitor JZL184 improves behavior and neural properties in Ts65Dn mice, a model of down syndromeCerebral vascular leak in a mouse model of amyloid neuropathology.Genetic modulation of soluble Aβ rescues cognitive and synaptic impairment in a mouse model of Alzheimer's diseaseEffect size of memory deficits in mice with adult-onset P301L tau expression.Impairments in experience-dependent scaling and stability of hippocampal place fields limit spatial learning in a mouse model of Alzheimer's disease.Anti-Aβ antibodies incapable of reducing cerebral Aβ oligomers fail to attenuate spatial reference memory deficits in J20 mice.Role of cystatin C in amyloid precursor protein-induced proliferation of neural stem/progenitor cells.Reversible pathologic and cognitive phenotypes in an inducible model of Alzheimer-amyloidosis.Wild-type neural progenitors divide and differentiate normally in an amyloid-rich environmentGenetic suppression of transgenic APP rescues Hypersynchronous network activity in a mouse model of Alzeimer's disease.Amyloid β Enhances Typical Rodent Behavior While It Impairs Contextual Memory Consolidation.Assessment of Spontaneous Alternation, Novel Object Recognition and Limb Clasping in Transgenic Mouse Models of Amyloid-β and Tau NeuropathologyDiscrete pools of oligomeric amyloid-β track with spatial learning deficits in a mouse model of Alzheimer amyloidosis.Practical considerations for choosing a mouse model of Alzheimer's disease.Alzheimer's Disease: The Role of Microglia in Brain Homeostasis and Proteopathy.Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease.Early-Life Stress Does Not Aggravate Spatial Memory or the Process of Hippocampal Neurogenesis in Adult and Middle-Aged APP/PS1 Mice.Combination anti-Aβ treatment maximizes cognitive recovery and rebalances mTOR signaling in APP mice.
P2860
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P2860
Transgenic APP expression during postnatal development causes persistent locomotor hyperactivity in the adult.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
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2012年學術文章
@zh-hant
name
Transgenic APP expression duri ...... or hyperactivity in the adult.
@ast
Transgenic APP expression duri ...... or hyperactivity in the adult.
@en
type
label
Transgenic APP expression duri ...... or hyperactivity in the adult.
@ast
Transgenic APP expression duri ...... or hyperactivity in the adult.
@en
prefLabel
Transgenic APP expression duri ...... or hyperactivity in the adult.
@ast
Transgenic APP expression duri ...... or hyperactivity in the adult.
@en
P2093
P2860
P356
P1476
Transgenic APP expression duri ...... or hyperactivity in the adult.
@en
P2093
Heather A Born
Joanna L Jankowsky
Pritam Das
Shaefali P Rodgers
P2860
P2888
P356
10.1186/1750-1326-7-28
P577
2012-06-18T00:00:00Z
P5875
P6179
1011981107