The great escape: when cancer cells hijack the genes for chemotaxis and motility.
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Targeting migration inducting gene-7 inhibits carcinoma cell invasion, early primary tumor growth, and stimulates monocyte oncolytic activityOverexpression of carcinoma and embryonic cytotrophoblast cell-specific Mig-7 induces invasion and vessel-like structure formationA glimpse of the ERM proteinsCell polarity signaling in the plasticity of cancer cell invasivenessTraversing the basement membrane in vivo: a diversity of strategiesTumor associated macrophage: a review on the phenotypes, traits and functionsCorrelated immunohistochemical and cytological assays for the prediction of hematogenous dissemination of breast cancerAn improved chamber for direct visualisation of chemotaxisMelanoma cells break down LPA to establish local gradients that drive chemotactic dispersalStudy of the Chemotactic Response of Multicellular Spheroids in a Microfluidic DeviceThe mechanics of metastasis: insights from a computational modelA multi-paradigm modeling framework to simulate dynamic reciprocity in a bioreactor.Rapid 3D Extrusion of Synthetic Tumor Microenvironments.Quantification of cell edge velocities and traction forces reveals distinct motility modules during cell spreadingThe role of filament-packing dynamics in powering amoeboid cell motilityA stochastic description of Dictyostelium chemotaxis.Alternatively activated macrophages and collagen remodeling characterize the postpartum involuting mammary gland across speciesEmergent Stratification in Solid Tumors Selects for Reduced Cohesion of Tumor Cells: A Multi-Cell, Virtual-Tissue Model of Tumor Evolution Using CompuCell3DN-ethylmaleimide-sensitive factor attachment protein α (αSNAP) regulates matrix adhesion and integrin processing in human epithelial cellsDual actin-bundling and protein kinase C-binding activities of fascin regulate carcinoma cell migration downstream of Rac and contribute to metastasisLocally controlled inhibitory mechanisms are involved in eukaryotic GPCR-mediated chemosensing.A hybrid microfluidic-vacuum device for direct interfacing with conventional cell culture methodsComputer simulation of glioma growth and morphology.Transforming growth factor beta controls the directional migration of hepatocyte cohorts by modulating their adhesion to fibronectin.Epidermal growth factor-induced enhancement of glioblastoma cell migration in 3D arises from an intrinsic increase in speed but an extrinsic matrix- and proteolysis-dependent increase in persistence.Open access microfluidic device for the study of cell migration during chemotaxis.Supervillin reorganizes the actin cytoskeleton and increases invadopodial efficiency.Systematic quantification of negative feedback mechanisms in the extracellular signal-regulated kinase (ERK) signaling network.Two-photon intravital multicolor imaging combined with inducible gene expression to distinguish metastatic behavior of breast cancer cells in vivo.Microfluidic kit-on-a-lid: a versatile platform for neutrophil chemotaxis assaysThe Hippo pathway polarizes the actin cytoskeleton during collective migration of Drosophila border cells.αE-catenin actin-binding domain alters actin filament conformation and regulates binding of nucleation and disassembly factorsSILAC-based proteomic quantification of chemoattractant-induced cytoskeleton dynamics on a second to minute timescaleChemotaxis in cancerThe microfluidic multitrap nanophysiometer for hematologic cancer cell characterization reveals temporal sensitivity of the calcein-AM efflux assay.Interplay between chemotaxis and contact inhibition of locomotion determines exploratory cell migrationComputational model of mesenchymal migration in 3D under chemotaxis.Actomyosin-dependent dynamic spatial patterns of cytoskeletal components drive mesoscale podosome organization.Intravital characterization of tumor cell migration in pancreatic cancer.Mass homozygotes accumulation in the NCI-60 cancer cell lines as compared to HapMap Trios, and relation to fragile site location.
P2860
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P2860
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@ast
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@en
type
label
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@ast
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@en
prefLabel
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@ast
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@en
P2093
P1476
The great escape: when cancer cells hijack the genes for chemotaxis and motility.
@en
P2093
Jeffrey E Segall
John Condeelis
Robert H Singer
P304
P356
10.1146/ANNUREV.CELLBIO.21.122303.120306
P577
2005-01-01T00:00:00Z