Redox mechanisms of muscle dysfunction in inflammatory disease.
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Investigating activity in hospitalized patients with chronic obstructive pulmonary disease: a pilot study.Exercise-induced oxidative stress: cellular mechanisms and impact on muscle force productionSlow recovery of the impaired fatigue resistance in postunloading mouse soleus muscle corresponding to decreased mitochondrial function and a compensatory increase in type I slow fibers.Free radical-mediated skeletal muscle dysfunction in inflammatory conditions.Reactive oxygen species: impact on skeletal muscle.The role of inflammation in ICU-acquired weaknessMitochondria dysfunction in lung cancer-induced muscle wasting in C2C12 myotubes.The Ang-(1-7)/Mas-1 axis attenuates the expression and signalling of TGF-β1 induced by AngII in mouse skeletal muscle.Detrimental effects of reloading recovery on force, shortening velocity, and power of soleus muscles from hindlimb-unloaded rats.Reply from Tyler Barker and Maret G. Traber
P2860
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P2860
Redox mechanisms of muscle dysfunction in inflammatory disease.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
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2005年學術文章
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name
Redox mechanisms of muscle dysfunction in inflammatory disease.
@ast
Redox mechanisms of muscle dysfunction in inflammatory disease.
@en
type
label
Redox mechanisms of muscle dysfunction in inflammatory disease.
@ast
Redox mechanisms of muscle dysfunction in inflammatory disease.
@en
prefLabel
Redox mechanisms of muscle dysfunction in inflammatory disease.
@ast
Redox mechanisms of muscle dysfunction in inflammatory disease.
@en
P2093
P1476
Redox mechanisms of muscle dysfunction in inflammatory disease.
@en
P2093
C William Balke
Francisco H Andrade
Karyn A Esser
Michael B Reid
P304
925-49, ix
P356
10.1016/J.PMR.2005.08.016
P577
2005-11-01T00:00:00Z