Preferential Iron Trafficking Characterizes Glioblastoma Stem-like Cells - Wikidata - awgldk - TriplyDB

Preferential Iron Trafficking Characterizes Glioblastoma Stem-like Cells

Preferential Iron Trafficking Characterizes Glioblastoma Stem-like Cells

http://www.wikidata.org/entity/Q36282985

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β-Elemene Selectively Inhibits the Proliferation of Glioma Stem-Like Cells Through the Downregulation of Notch1.Targeting iron metabolism in drug discovery and delivery Prognostic value of FOXM1 in solid tumors: a systematic review and meta-analysis.Low oxygen tension reverses antineoplastic effect of iron chelator deferasirox in human glioblastoma cells.Aberrations in the Iron Regulatory Gene Signature Are Associated with Decreased Survival in Diffuse Infiltrating Gliomas.Integration of MicroRNA, mRNA, and Protein Expression Data for the Identification of Cancer-Related MicroRNAs.Iron and cancer: recent insights Nuclear GSK3β promotes tumorigenesis by phosphorylating KDM1A and inducing its deubiquitylation by USP22.MiR-146b-5p overexpression attenuates stemness and radioresistance of glioma stem cells by targeting HuR/lincRNA-p21/β-catenin pathway.Enhancement of 5-aminolevulinic acid-based fluorescence detection of side population-defined glioma stem cells by iron chelation.Quantitative Proteomic Profiling of Tachyplesin I Targets in U251 Gliomaspheres.The roles of NRF2 in modulating cellular iron homeostasis.m6A Demethylase ALKBH5 Maintains Tumorigenicity of Glioblastoma Stem-like Cells by Sustaining FOXM1 Expression and Cell Proliferation Program.Glioblastoma Cancer Stem Cells Evade Innate Immune Suppression of Self-Renewal through Reduced TLR4 Expression.Cancer-associated isocitrate dehydrogenase mutations induce mitochondrial DNA instability Lysosomes as Oxidative Targets for Cancer Therapy BRAF mutations are associated with increased iron regulatory protein-2 expression in colorectal tumorigenesis.Iron addiction: a novel therapeutic target in ovarian cancer Transferrin receptor-1 and ferritin heavy and light chains in astrocytic brain tumors: Expression and prognostic value.L-Ferritin targets breast cancer stem cells and delivers therapeutic and imaging agents.Pharmacological Targeting of the Histone Chaperone Complex FACT Preferentially Eliminates Glioblastoma Stem Cells and Prolongs Survival in Preclinical Models Organotypic brain explant culture as a drug evaluation system for malignant brain tumors.Fumarate Mediates a Chronic Proliferative Signal in Fumarate Hydratase-Inactivated Cancer Cells by Increasing Transcription and Translation of Ferritin Genes.MYC-Regulated Mevalonate Metabolism Maintains Brain Tumor-Initiating Cells.Salinomycin kills cancer stem cells by sequestering iron in lysosomes.Dysregulation of Iron Metabolism in Cholangiocarcinoma Stem-like Cells.Targeting transferrin receptor delivery of temozolomide for a potential glioma stem cell-mediated therapy.A molecular view of the radioresistance of gliomas.Cell surface Thomsen-Friedenreich proteome profiling of metastatic prostate cancer cells reveals potential link with cancer stem cell-like phenotype.Assessing multiparametric drug response in tissue engineered tumor microenvironment models.Transferrin conjugated nontoxic carbon dots for doxorubicin delivery to target pediatric brain tumor cells.Differential regulation of hepcidin in cancer and non-cancer tissues and its clinical implications.Hepcidin, an emerging and important player in brain iron homeostasis.Temporal DNA-PK activation drives genomic instability and therapy resistance in glioma stem cells.Gallium Maltolate Disrupts Tumor Iron Metabolism and Retards the Growth of Glioblastoma by Inhibiting Mitochondrial Function and Ribonucleotide Reductase.Multiple functions of m6A RNA methylation in cancer.SuperQuant-assisted comparative proteome analysis of glioblastoma subpopulations allows for identification of potential novel therapeutic targets and cell markers.Six-Transmembrane Epithelial Antigen of Prostate 3 Predicts Poor Prognosis and Promotes Glioblastoma Growth and Invasion.Alterations in Cellular Iron Metabolism Provide More Therapeutic Opportunities for Cancer.

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Preferential Iron Trafficking Characterizes Glioblastoma Stem-like Cells

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Preferential Iron Trafficking Characterizes Glioblastoma Stem-like Cells

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Ann Mari Rosager

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Anne M Nixon

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Awad M Jarrar

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Jeremy N Rich

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P2860

Coordinated regulation of iron-controlling genes, H-ferritin and IRP2, by c-MYC

Phosphorylation of EZH2 activates STAT3 signaling via STAT3 methylation and promotes tumorigenicity of glioblastoma stem-like cells

FoxM1 promotes β-catenin nuclear localization and controls Wnt target-gene expression and glioma tumorigenesis

Interleukin-6 induces hepcidin expression through STAT3

Glioma stem cell proliferation and tumor growth are promoted by nitric oxide synthase-2

PIAS3 interacts with ATF1 and regulates the human ferritin H gene through an antioxidant-responsive element

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

Identification of human brain tumour initiating cells

Multiple knockout mouse models reveal lincRNAs are required for life and brain development

The transferrin receptor part I: Biology and targeting with cytotoxic antibodies for the treatment of cancer

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