Age dependent increase in the levels of osteopontin inhibits skeletal muscle regeneration.
about
The ins and outs of muscle stem cell agingThe central role of muscle stem cells in regenerative failure with agingA single heterochronic blood exchange reveals rapid inhibition of multiple tissues by old bloodStem cells as vehicles for youthful regeneration of aged tissues.Osteopontin, inflammation and myogenesis: influencing regeneration, fibrosis and size of skeletal muscleSerum OPN expression for identification of gastric cancer and atrophic gastritis and its influencing factorsRegenerative capacity of old muscle stem cells declines without significant accumulation of DNA damageRespiratory syncytial virus (RSV) infection in elderly mice results in altered antiviral gene expression and enhanced pathologyThe altered fate of aging satellite cells is determined by signaling and epigenetic changes.Extracellular matrix regulation in the muscle satellite cell niche.Systemic attenuation of the TGF-β pathway by a single drug simultaneously rejuvenates hippocampal neurogenesis and myogenesis in the same old mammalOxytocin is an age-specific circulating hormone that is necessary for muscle maintenance and regenerationSarcospan: a small protein with large potential for Duchenne muscular dystrophyhESC-secreted proteins can be enriched for multiple regenerative therapies by heparin-bindingAge-dependent alteration in muscle regeneration: the critical role of tissue niche.Cellular dynamics in the muscle satellite cell niche.OPN-a induces muscle inflammation by increasing recruitment and activation of pro-inflammatory macrophages.Changes in Communication between Muscle Stem Cells and their Environment with Aging.Functional dysregulation of stem cells during aging: a focus on skeletal muscle stem cells.Systemic Problems: A perspective on stem cell aging and rejuvenation.Application of bio-orthogonal proteome labeling to cell transplantation and heterochronic parabiosis.Increased Adipocyte Area in Injured Muscle With Aging and Impaired Remodeling in Female Mice.Absence of CCR2 results in an inflammaging environment in young mice with age-independent impairments in muscle regeneration.Genetic modifiers of muscular dystrophy act on sarcolemmal resealing and recovery from injury.SPP1 genotype and glucocorticoid treatment modify osteopontin expression in Duchenne muscular dystrophy cells.The Basis of Muscle Regeneration
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P2860
Age dependent increase in the levels of osteopontin inhibits skeletal muscle regeneration.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
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2012年學術文章
@zh-hant
name
Age dependent increase in the ...... skeletal muscle regeneration.
@ast
Age dependent increase in the ...... skeletal muscle regeneration.
@en
type
label
Age dependent increase in the ...... skeletal muscle regeneration.
@ast
Age dependent increase in the ...... skeletal muscle regeneration.
@en
prefLabel
Age dependent increase in the ...... skeletal muscle regeneration.
@ast
Age dependent increase in the ...... skeletal muscle regeneration.
@en
P2093
P2860
P356
P1433
P1476
Age dependent increase in the ...... skeletal muscle regeneration.
@en
P2093
Denny Wijaya
Irina M Conboy
Novalia Pishesha
Preeti Paliwal
P2860
P304
P356
10.18632/AGING.100477
P577
2012-08-01T00:00:00Z