What does tumour necrosis factor excess do to the immune system long term? - Wikidata - awgldk - TriplyDB

What does tumour necrosis factor excess do to the immune system long term?

What does tumour necrosis factor excess do to the immune system long term?

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Gammadelta T cells are reduced and rendered unresponsive by hyperglycemia and chronic TNFalpha in mouse models of obesity and metabolic disease Psychoneuroimmunology meets neuropsychopharmacology: translational implications of the impact of inflammation on behavior An extensive phenotypic characterization of the hTNFalpha transgenic mice.Adenovirus-mediated CD40L gene transfer increases Teffector/Tregulatory cell ratio and upregulates death receptors in metastatic melanoma patients.Maturation-dependent licensing of naive T cells for rapid TNF production.De novo assembly and transcriptome characterization: novel insights into the natural resistance mechanisms of Microtus fortis against Schistosoma japonicum Immune suppression and immune activation in depression Contrasting effects of TNF and anti-TNF on the activation of effector T cells and regulatory T cells in autoimmunity.Serum from exercising humans suppresses t-cell cytokine production.Role of TNFR1 in the innate airway hyperresponsiveness of obese mice.The evolutionary significance of depression in Pathogen Host Defense (PATHOS-D).Perinatal tumor necrosis factor-α production, influenced by maternal pregnancy weight gain, predicts childhood asthma.TNFR2 expression by CD4 effector T cells is required to induce full-fledged experimental colitis The phenotypic and functional consequences of tumour necrosis factor receptor type 2 expression on CD4(+) FoxP3(+) regulatory T cells.Immunomodulation at epithelial sites by obesity and metabolic disease.TNF activity and T cells.Pathogen-Host Defense in the Evolution of Depression: Insights into Epidemiology, Genetics, Bioregional Differences and Female Preponderance.TNF receptor 1 signaling is critically involved in mediating angiotensin-II-induced cardiac fibrosis Targeting α- and β-Adrenergic Receptors Differentially Shifts Th1, Th2, and Inflammatory Cytokine Profiles in Immune Organs to Attenuate Adjuvant Arthritis.The role of aberrant expression of T cell miRNAs affected by TNF-α in the immunopathogenesis of rheumatoid arthritis.Sustained T cell Rap1 signaling is protective in the collagen-induced arthritis model of rheumatoid arthritis.Infliximab associated with faster symptom resolution compared with corticosteroids alone for the management of immune-related enterocolitis

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What does tumour necrosis factor excess do to the immune system long term?

http://www.wikidata.org/entity/Q36292657

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

What does tumour necrosis factor excess do to the immune system long term?

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What does tumour necrosis factor excess do to the immune system long term?

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What does tumour necrosis factor excess do to the immune system long term?

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What does tumour necrosis factor excess do to the immune system long term?

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ARD.2005.042523

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Annals of the Rheumatic Diseases

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What does tumour necrosis factor excess do to the immune system long term?

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Clark J

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Panesar M

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Vagenas P

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P2860

CD25brightCD4+ regulatory T cells are enriched in inflamed joints of patients with chronic rheumatic disease

To be, or not to be: NF-kappaB is the answer--role of Rel/NF-kappaB in the regulation of apoptosis

RANK-L and RANK: T cells, bone loss, and mammalian evolution

The TNF and TNF receptor superfamilies: integrating mammalian biology

Co-stimulatory members of the TNFR family: keys to effective T-cell immunity?

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment.

Tumor necrosis factor receptor and Fas signaling mechanisms.

A domain in TNF receptors that mediates ligand-independent receptor assembly and signaling.

Assessment of antibodies to double-stranded DNA induced in rheumatoid arthritis patients following treatment with infliximab, a monoclonal antibody to tumor necrosis factor alpha: findings in open-label and randomized placebo-controlled trials.

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iv70-6

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10.1136/ARD.2005.042523

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64 Suppl 4

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