Hypothesis: the humoral immune response to oral bacteria provides a stimulus for the development of rheumatoid arthritis.
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Interventions for periodontal disease in people with rheumatoid arthritisMicrobiota and chronic inflammatory arthritis: an interwoven linkThe Link Between Periodontitis and Rheumatoid Arthritis: A Periodontist's PerspectiveCould early rheumatoid arthritis resolve after periodontitis treatment only?: case report and review of the literatureUnique Structure and Stability of HmuY, a Novel Heme-Binding Protein of Porphyromonas gingivalisDetection of oral bacterial DNA in synovial fluid.Using Big Data to Evaluate the Association between Periodontal Disease and Rheumatoid ArthritisMicrobiome and mucosal inflammation as extra-articular triggers for rheumatoid arthritis and autoimmunityThe CO donor CORM-2 inhibits LPS-induced vascular cell adhesion molecule-1 expression and leukocyte adhesion in human rheumatoid synovial fibroblasts.Microbiota in T-cell homeostasis and inflammatory diseasesMatrix Metalloproteinase Gene Activation Resulting from Disordred Epigenetic Mechanisms in Rheumatoid Arthritis.Porphyromonas gingivalis and the pathogenesis of rheumatoid arthritis: analysis of various compartments including the synovial tissue.Porphyromonas gingivalis peptidylarginine deiminase, a key contributor in the pathogenesis of experimental periodontal disease and experimental arthritisB cells expressing the IgA receptor FcRL4 participate in the autoimmune response in patients with rheumatoid arthritis.Periodontitis and Porphyromonas gingivalis in patients with rheumatoid arthritis.Periodontal disease and subgingival microbiota as contributors for rheumatoid arthritis pathogenesis: modifiable risk factors?Bacterial and human peptidylarginine deiminases: targets for inhibiting the autoimmune response in rheumatoid arthritis?A prospective study of periodontal disease and risk of rheumatoid arthritisPeriodontitis, Porphyromonas, and the pathogenesis of rheumatoid arthritis.The association between rheumatoid arthritis and periodontal diseaseAnti-HmuY antibodies specifically recognize Porphyromonas gingivalis HmuY protein but not homologous proteins in other periodontopathogens.Differential roles of tryptophan residues in conformational stability of Porphyromonas gingivalis HmuY hemophoreDefining the role of Porphyromonas gingivalis peptidylarginine deiminase (PPAD) in rheumatoid arthritis through the study of PPAD biology.Periodontitis and Porphyromonas gingivalis in preclinical stage of arthritis patientsGerms and joints: the contribution of the human microbiome to rheumatoid arthritis.Association between anti-Porphyromonas gingivalis or anti-α-enolase antibody and severity of periodontitis or rheumatoid arthritis (RA) disease activity in RA.Rheumatoid arthritis and periodontitis - inflammatory and infectious connections. Review of the literature.Rheumatoid Arthritis Risk Associated with Periodontitis Exposure: A Nationwide, Population-Based Cohort Study.Periodontal disease and rheumatoid arthritis: the evidence accumulates for complex pathobiologic interactions.Rheumatoid arthritis-associated autoantibodies in non-rheumatoid arthritis patients with mucosal inflammation: a case-control study.Minocycline and doxycycline therapy in community patients with rheumatoid arthritis: prescribing patterns, patient-level determinants of use, and patient-reported side effects.Prevalence of Periodontitis in Patients with Established Rheumatoid Arthritis: A Swedish Population Based Case-Control Study.The peptidylarginine deiminase gene is a conserved feature of Porphyromonas gingivalis.Periodontal disease, tooth loss and incident rheumatoid arthritis: results from the First National Health and Nutrition Examination Survey and its epidemiological follow-up study.Porphyromonas gingivalis and disease-related autoantibodies in individuals at increased risk of rheumatoid arthritis.Periodontitis increases rheumatic factor serum levels and citrullinated proteins in gingival tissues and alter cytokine balance in arthritic rats.Antibodies to Porphyromonas gingivalis Indicate Interaction Between Oral Infection, Smoking, and Risk Genes in Rheumatoid Arthritis Etiology.Periodontitis in established rheumatoid arthritis patients: a cross-sectional clinical, microbiological and serological studyBronchiectasis is a Model for Chronic Bacterial Infection Inducing Autoimmunity in Rheumatoid Arthritis.Release of Active Peptidyl Arginine Deiminases by Neutrophils Can Explain Production of Extracellular Citrullinated Autoantigens in Rheumatoid Arthritis Synovial Fluid
P2860
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P2860
Hypothesis: the humoral immune response to oral bacteria provides a stimulus for the development of rheumatoid arthritis.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@ast
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@en
type
label
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@ast
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@en
prefLabel
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@ast
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@en
P2093
P1433
P1476
Hypothesis: the humoral immune ...... pment of rheumatoid arthritis.
@en
P2093
Elliot D Rosenstein
Gerald Weissmann
Laura J Kushner
Robert A Greenwald
P2888
P304
P356
10.1007/S10753-004-6641-Z
P577
2004-12-01T00:00:00Z