Ceftriaxone normalizes nucleus accumbens synaptic transmission, glutamate transport, and export following cocaine self-administration and extinction training.
about
Glial abnormalities in substance use disorders and depression: does shared glutamatergic dysfunction contribute to comorbidity?Astrocytic dysfunction and addiction: consequences of impaired glutamate homeostasisGlutamate Transporter GLT-1 as a Therapeutic Target for Substance Use DisordersPotential role of N-acetylcysteine in the management of substance use disordersImbalances in prefrontal cortex CC-Homer1 versus CC-Homer2 expression promote cocaine preference.Effects of repeated cocaine exposure on habit learning and reversal by N-acetylcysteineClavulanic acid reduces rewarding, hyperthermic and locomotor-sensitizing effects of morphine in rats: a new indication for an old drug?Effects of ceftriaxone on chronic ethanol consumption: a potential role for xCT and GLT1 modulation of glutamate levels in male P rats.Effects of ceftriaxone on systemic and central expression of anti- and pro-inflammatory cytokines in alcohol-preferring (P) rats exposed to ethanol.Effects of ceftriaxone on ethanol intake: a possible role for xCT and GLT-1 isoforms modulation of glutamate levels in P rats.Effects of MS-153 on chronic ethanol consumption and GLT1 modulation of glutamate levels in male alcohol-preferring rats.Effectiveness of Ceftriaxone Treatment in Preventing Relapse-like Drinking Behavior Following Long-term Ethanol Dependence in P RatsRepeated cycles of chronic intermittent ethanol exposure increases basal glutamate in the nucleus accumbens of mice without affecting glutamate transport.Ceftriaxone and cefazolin attenuate the cue-primed reinstatement of alcohol-seekingGlutamate transporter GLT-1 mediates N-acetylcysteine inhibition of cocaine reinstatement.Effects of ceftriaxone on GLT1 isoforms, xCT and associated signaling pathways in P rats exposed to ethanol.Glial Expression of the Caenorhabditis elegans Gene swip-10 Supports Glutamate Dependent Control of Extrasynaptic Dopamine SignalingCeftriaxone attenuates acquisition and facilitates extinction of cocaine-induced suppression of saccharin intake in C57BL/6J miceCeftriaxone attenuates ethanol drinking and restores extracellular glutamate concentration through normalization of GLT-1 in nucleus accumbens of male alcohol-preferring ratsReversal of morphine-induced cell-type-specific synaptic plasticity in the nucleus accumbens shell blocks reinstatement.Clavulanic acid enhances glutamate transporter subtype I (GLT-1) expression and decreases reinforcing efficacy of cocaine in miceCeftriaxone attenuates cocaine relapse after abstinence through modulation of nucleus accumbens AMPA subunit expression.Glutamatergic mechanisms of comorbidity between acute stress and cocaine self-administrationSignals from the Fourth Dimension Regulate Drug RelapseThe Nucleus Accumbens: Mechanisms of Addiction across Drug Classes Reflect the Importance of Glutamate HomeostasisBiotic acts of antibioticsEffects of Ceftriaxone on Glial Glutamate Transporters in Wistar Rats Administered Sequential Ethanol and MethamphetamineEffects of the beta-lactam antibiotic ceftriaxone on nicotine withdrawal and nicotine-induced reinstatement of preference in mice.Alterations of the Host Microbiome Affect Behavioral Responses to Cocaine.Disruption of glutamate receptor-interacting protein in nucleus accumbens enhances vulnerability to cocaine relapse.Synaptic glutamate spillover due to impaired glutamate uptake mediates heroin relapseCeftriaxone attenuates locomotor activity induced by acute and repeated cocaine exposure in miceGlutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease.Main path and byways: non-vesicular glutamate release by system xc(-) as an important modifier of glutamatergic neurotransmission.Glutamate Transport: A New Bench to Bedside Mechanism for Treating Drug AbuseContrasting the Role of xCT and GLT-1 Upregulation in the Ability of Ceftriaxone to Attenuate the Cue-Induced Reinstatement of Cocaine Seeking and Normalize AMPA Receptor Subunit Expression.Corticostriatal circuitry in regulating diseases characterized by intrusive thinking.Uncoupling DAPK1 from NMDA receptor GluN2B subunit exerts rapid antidepressant-like effects.Alcohol consumption increases basal extracellular glutamate in the nucleus accumbens core of Sprague-Dawley rats without increasing spontaneous glutamate release.Ceftriaxone attenuates acute cocaine-evoked dopaminergic neurotransmission in the nucleus accumbens of the rat.
P2860
Q26829556-155D9696-2306-4E59-9A85-192673D8E7D1Q26996339-6013B12B-9DD1-4B86-8D50-863BF8C52477Q28081726-5CF944CC-71D8-4F8E-B0C6-DB755D2B1A0EQ28306130-776A7F0D-C7B3-440A-8002-35A5E83A49DDQ30447403-4339FE3E-32FF-44FC-92BA-46F39EB857EFQ33764257-48B31E94-23D7-4633-9F17-21FF2D44088AQ34021956-5D3DF5D6-15FB-4FDB-9789-2BCF2DED4D3BQ34022203-1E5F4169-03F8-4F4C-A966-FBB44A0EC3ECQ34048165-B8184CE1-E5F9-400F-83CC-65A0984299CDQ34250509-1A638BAF-F3EC-4B84-BC73-4D6100A5881BQ34426947-0D859256-DED0-49EA-93FC-FB745E4D230BQ35077751-7B2DA66A-8B37-40EA-B27B-1A541E985B7AQ35110324-4326E7D6-931E-41B3-BB8E-AE5AA22FA826Q35161340-C7971338-7670-4A36-8C42-9301FA7F0CF6Q35623912-E7BB4BE4-3E3C-4B70-B562-6A096DB3E1FBQ35738010-257C679A-F0CB-4560-9D5F-520C212A702BQ35772934-9BF57C6D-E29F-4F37-A5EE-6B9B494111B5Q35864748-59B9FCCA-1744-43E7-9EC4-9FDADF927801Q35956322-071CE4A3-0A54-4661-AD9F-93560AB6FBD0Q36498162-EB8686F6-624C-4583-8195-DD0B3BEC15CCQ36581692-0EEF8595-7D57-42D0-BD85-55BFBEDC9CAFQ36604607-75D9CE46-F8ED-4AD0-AFB5-505ECA8441D2Q36772392-05DCBF1F-4A38-4894-89DA-A1BE70122FDBQ37059772-C6910786-E4CB-4243-B899-68DC13660ED2Q37062547-D157B117-53CA-4F6A-827F-A94150429DA2Q37101851-D5006D87-4150-41C5-8480-E8099E417944Q37273362-7180C085-AC5C-47C0-9C5D-662A25304668Q37291940-856F2DB0-5DE7-4275-B710-64875440B157Q37345533-7770D5F5-6A46-482D-96CC-D659264719A8Q37491638-B4D15B4F-3ACE-47CB-8940-3B3E2CA87FDAQ37705513-F43AE6EC-E8D1-4611-AB61-875F5DEE5C0CQ37731456-B8653AC9-7C8F-40E5-A91F-00008E31C643Q38512084-B34C0BC8-6196-4BB0-9DC1-B0BF74EB65C9Q38580123-9ABAD903-700E-4FB1-811C-9AB580A415F3Q38729846-3F41E45F-FF18-4B2B-8543-91AC8FD73544Q38790892-9E260078-E127-4162-886E-74D538AF2605Q38805967-19CD32C5-EE41-4D1C-A12C-961F190F99ADQ40229544-5051D245-FB7A-4EE3-842D-F2511441082DQ41664989-849926AA-323A-48BA-8618-6312DEEB0CA8Q41817045-DEDDA8AB-F80A-42BE-8D30-11A0100BE411
P2860
Ceftriaxone normalizes nucleus accumbens synaptic transmission, glutamate transport, and export following cocaine self-administration and extinction training.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@ast
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@en
type
label
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@ast
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@en
prefLabel
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@ast
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@en
P2093
P2860
P1476
Ceftriaxone normalizes nucleus ...... ation and extinction training.
@en
P2093
Heather Trantham-Davidson
Kathryn J Reissner
Lori A Knackstedt
Peter W Kalivas
Ryan T LaLumiere
P2860
P304
12406-12410
P356
10.1523/JNEUROSCI.1976-12.2012
P407
P577
2012-09-01T00:00:00Z