Knockdown of EMMPRIN improves adverse remodeling mediated by IL-18 in the post-infarcted heart

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Extracellular matrix metalloproteinase inducer enhances host resistance against pseudomonas aeruginosa infection through MAPK signaling pathway.Basigin rs8259 Polymorphism Confers Decreased Risk of Chronic Heart Failure in a Chinese Population.

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Knockdown of EMMPRIN improves adverse remodeling mediated by IL-18 in the post-infarcted heart

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2015 nî lūn-bûn

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2015年学术文章

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Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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P1476

Knockdown of EMMPRIN improves ...... 18 in the post-infarcted heart

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Haifeng Zhang

http://www.w3.org/2001/XMLSchema#string

Jingfeng Wang

http://www.w3.org/2001/XMLSchema#string

Rongjie Lin

http://www.w3.org/2001/XMLSchema#string

Ruqiong Nie

http://www.w3.org/2001/XMLSchema#string

Shuanglun Xie

http://www.w3.org/2001/XMLSchema#string

Xiaorong Shu

http://www.w3.org/2001/XMLSchema#string

Yuyang Chen

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Zizhuo Su

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Involvement of HAb18G/CD147 in T cell activation and immunological synapse formation.

Interleukin-18 induces EMMPRIN expression in primary cardiomyocytes via JNK/Sp1 signaling and MMP-9 in part via EMMPRIN and through AP-1 and NF-kappaB activation

EMMPRIN activates multiple transcription factors in cardiomyocytes, and induces interleukin-18 expression via Rac1-dependent PI3K/Akt/IKK/NF-kappaB andMKK7/JNK/AP-1 signaling.

CD147 overexpression on synoviocytes in rheumatoid arthritis enhances matrix metalloproteinase production and invasiveness of synoviocytes

Interleukin 18 in acute myocardial infarction.

Neutralization of interleukin-18 ameliorates ischemia/reperfusion-induced myocardial injury.

Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging mice.

Resveratrol blocks interleukin-18-EMMPRIN cross-regulation and smooth muscle cell migration.

IL-18 binding protein-expressing mesenchymal stem cells improve myocardial protection after ischemia or infarction.

Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction

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2015-10-15T00:00:00Z

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Knockdown of EMMPRIN improves adverse remodeling mediated by IL-18 in the post-infarcted heart

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P2860

P2860

Knockdown of EMMPRIN improves adverse remodeling mediated by IL-18 in the post-infarcted heart

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P1433

P1476

P2093

P2860

P304

P31

P407

P433

P478

P577

P698

P932

P1433

P1476

P2093

P2860

P304

P31

P407

P433

P478

P577

P698

P932