Toll-like receptor 2 controls the gamma interferon response to Francisella tularensis by mouse liver lymphocytes.
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Immunotherapy for tularemiaFine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensisNasal Acai polysaccharides potentiate innate immunity to protect against pulmonary Francisella tularensis and Burkholderia pseudomallei InfectionsTLR-dependent control of Francisella tularensis infection and host inflammatory responsesIdentification of an essential Francisella tularensis subsp. tularensis virulence factor.Adaptation of Francisella tularensis to the mammalian environment is governed by cues which can be mimicked in vitro.Glycogen synthase kinase-3beta (GSK3beta) inhibition suppresses the inflammatory response to Francisella infection and protects against tularemia in miceIKKβ in myeloid cells controls the host response to lethal and sublethal Francisella tularensis LVS infection.Discordant results obtained with Francisella tularensis during in vitro and in vivo immunological studies are attributable to compromised bacterial structural integrity.Interleukin-17 is required for T helper 1 cell immunity and host resistance to the intracellular pathogen Francisella tularensis.IL12Rβ1: the cytokine receptor that we used to know.Francisella tularensis LVS-induced Interleukin-12 p40 cytokine production mediates dendritic cell migration through IL-12 Receptor β1.IL-23 in infections, inflammation, autoimmunity and cancer: possible role in HIV-1 and AIDSPhenotypic, morphological, and functional heterogeneity of splenic immature myeloid cells in the host response to tularemia.Francisella tularensis live vaccine strain folate metabolism and pseudouridine synthase gene mutants modulate macrophage caspase-1 activation.Francisella tularensis invasion of lung epithelial cellsRequirement of TLR4 and CD14 in dendritic cell activation by Hemagglutinin B from Porphyromonas gingivalis.Francisella tularensis induces extensive caspase-3 activation and apoptotic cell death in the tissues of infected mice.Mitogen-activated protein kinases (MAPKs) are modulated during Francisella tularensis infection, but inhibition of extracellular-signal-regulated kinases (ERKs) is of limited therapeutic benefitServing the new masters - dendritic cells as hosts for stealth intracellular bacteria.Delineation of the role of Toll-like receptor signaling during peritonitis by a gradually growing pathogenic Escherichia coli.Innate Immune Recognition: Implications for the Interaction of Francisella tularensis with the Host Immune System.Complement Receptor 3-Mediated Inhibition of Inflammasome Priming by Ras GTPase-Activating Protein During Francisella tularensis Phagocytosis by Human Mononuclear Phagocytes.Decreased TLR2 signal expression in peripheral blood mononuclear cell from patients with cryptococcal meningitis.
P2860
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P2860
Toll-like receptor 2 controls the gamma interferon response to Francisella tularensis by mouse liver lymphocytes.
description
2007 nî lūn-bûn
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2007年の論文
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2007年学术文章
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2007年学术文章
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2007年学术文章
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2007年学术文章
@zh-my
2007年学术文章
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2007年學術文章
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2007年學術文章
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name
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@ast
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@en
type
label
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@ast
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@en
prefLabel
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@ast
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@en
P2093
P2860
P356
P1476
Toll-like receptor 2 controls ...... is by mouse liver lymphocytes.
@en
P2093
Hung-Wen Yeh
Jason R Wickstrum
Kee-Jong Hong
Michael J Parmely
P2860
P304
P356
10.1128/IAI.00561-07
P407
P577
2007-09-04T00:00:00Z