Inhibition of in vitro natural killer activity by the third component of complement: role for the C3a fragment
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Rituximab: mechanism of actionRecombinant acylation stimulating protein administration to C3-/- mice increases insulin resistance via adipocyte inflammatory mechanisms.Human platelet activation by C3a and C3a des-arg.Complement regulates conventional DC-mediated NK-cell activation by inducing TGF-β1 in Gr-1+ myeloid cellsHuman natural killer cells, activated lymphocyte killer cells, and monocytes possess similar cytotoxic mechanisms.A Complement-Optimized EGFR Antibody Improves Cytotoxic Functions of Polymorphonuclear Cells against Tumor Cells.Cross-talk between α7 nAChR-mediated cholinergic pathway and acylation stimulating protein signaling in 3T3-L1 adipocytes: role of NFκB and STAT3.
P2860
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P2860
Inhibition of in vitro natural killer activity by the third component of complement: role for the C3a fragment
description
1982 nî lūn-bûn
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1982年の論文
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1982年学术文章
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1982年学术文章
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1982年学术文章
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1982年学术文章
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1982年学术文章
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1982年學術文章
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1982年學術文章
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1982年學術文章
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name
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@ast
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@en
type
label
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@ast
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@en
prefLabel
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@ast
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@en
P2093
P2860
P356
P1476
Inhibition of in vitro natural ...... ent: role for the C3a fragment
@en
P2093
P2860
P304
P356
10.1073/PNAS.79.19.6003
P407
P577
1982-10-01T00:00:00Z