Matrix metalloproteinase 9 and vascular endothelial growth factor are essential for osteoclast recruitment into developing long bones.
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KMUP-1 suppresses RANKL-induced osteoclastogenesis and prevents ovariectomy-induced bone loss: roles of MAPKs, Akt, NF-κB and calcium/calcineurin/NFATc1 pathwaysConnective tissue growth factor coordinates chondrogenesis and angiogenesis during skeletal developmentTRE17/USP6 oncogene translocated in aneurysmal bone cyst induces matrix metalloproteinase production via activation of NF-kappaBRecruitment of stem and progenitor cells from the bone marrow niche requires MMP-9 mediated release of kit-ligandMatrix metalloproteinases: old dogs with new tricksCartilage to bone transitions in health and diseaseAirway Remodeling in Chronic Obstructive Pulmonary Disease and Asthma: the Role of Matrix Metalloproteinase-9Matrix metalloproteinase-9 activates TGF-β and stimulates fibroblast contraction of collagen gelsProteolytic excision of a repressive loop domain in tartrate-resistant acid phosphatase by cathepsin K in osteoclastsRole of matrix metalloproteinase 13 in both endochondral and intramembranous ossification during skeletal regenerationCritical roles for collagenase-3 (Mmp13) in development of growth plate cartilage and in endochondral ossificationHyaluronan production by means of Has2 gene expression in chondrocytes is essential for long bone developmentGalectin-3 is a downstream regulator of matrix metalloproteinase-9 function during endochondral bone formation.How matrix metalloproteinases regulate cell behaviorA physical mechanism for coupling bone resorption and formation in adult human bone.Hck contributes to bone homeostasis by controlling the recruitment of osteoclast precursors.Matrix metalloproteinase-9 contributes to choroidal neovascularization.Endostatin inhibits VEGF-A induced osteoclastic bone resorption in vitroTemporal changes in MMP mRNA expression in the lens epithelium during anterior subcapsular cataract formation.Disease modifying and antiangiogenic activity of 2-methoxyestradiol in a murine model of rheumatoid arthritis.Malfunction of bone marrow-derived osteoclasts and the delay of bone fracture healing in diabetic mice.Molecular pathology of vertebral deformities in hyperthermic Atlantic salmon (Salmo salar)Tissue inhibitor of metalloproteinase-3 (TIMP-3) regulates hematopoiesis and bone formation in vivo.Matrix metalloproteinase (MMP)-13 regulates mammary tumor-induced osteolysis by activating MMP9 and transforming growth factor-beta signaling at the tumor-bone interface.Complementary interplay between matrix metalloproteinase-9, vascular endothelial growth factor and osteoclast function drives endochondral bone formationGenetic disorders of the skeleton: a developmental approachFgf-9 is required for angiogenesis and osteogenesis in long bone repair.Vascular endothelial growth factor stimulates bone repair by promoting angiogenesis and bone turnover.Intermittent hypoxia effect on osteoclastogenesis stimulated by neuroblastoma cells.Choline kinase beta is required for normal endochondral bone formationOsteoclast-derived matrix metalloproteinase-9 directly affects angiogenesis in the prostate tumor-bone microenvironment.Endogenous retinoids in mammalian growth plate cartilage: analysis and roles in matrix homeostasis and turnover2-Methoxystypandrone represses RANKL-mediated osteoclastogenesis by down-regulating formation of TRAF6-TAK1 signalling complexesMMP9 is protective against lethal inflammatory mass lesions in the mouse colon.Physiology and pathophysiology of matrix metalloproteases.Sequential morphological characteristics of murine fetal liver hematopoietic microenvironment in Swiss Webster mice.Matrix metalloproteinase-9 in lung remodeling.Matrix metalloproteinase activity and osteoclasts in experimental prostate cancer bone metastasis tissue.(+)-Vitisin A inhibits osteoclast differentiation by preventing TRAF6 ubiquitination and TRAF6-TAK1 formation to suppress NFATc1 activationThe N-terminal cleavage of chondromodulin-I in growth-plate cartilage at the hypertrophic and calcified zones during bone development
P2860
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P2860
Matrix metalloproteinase 9 and vascular endothelial growth factor are essential for osteoclast recruitment into developing long bones.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh
2000年學術文章
@zh-hant
name
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@ast
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@en
type
label
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@ast
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@en
prefLabel
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@ast
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@en
P2093
P2860
P356
P1476
Matrix metalloproteinase 9 and ...... nt into developing long bones.
@en
P2093
A C Pedersen
B Therkidsen
J M Delaissé
K Henriksen
M T Engsig
P2860
P304
P356
10.1083/JCB.151.4.879
P407
P577
2000-11-01T00:00:00Z