Oligodendroglial modulation of fast axonal transport in a mouse model of hereditary spastic paraplegia. - Wikidata - awgldk - TriplyDB

Oligodendroglial modulation of fast axonal transport in a mouse model of hereditary spastic paraplegia.

Oligodendroglial modulation of fast axonal transport in a mouse model of hereditary spastic paraplegia.

http://www.wikidata.org/entity/Q36322059

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Alzheimer's disease as homeostatic responses to age-related myelin breakdown Disrupted axo-glial junctions result in accumulation of abnormal mitochondria at nodes of ranvier Myelin damage and repair in pathologic CNS: challenges and prospects A putative functional role for oligodendrocytes in mood regulation Time-lapse imaging of the dynamics of CNS glial-axonal interactions in vitro and ex vivo Delving into the complexity of hereditary spastic paraplegias: how unexpected phenotypes and inheritance modes are revolutionizing their nosology Uncoupling of neuroinflammation from axonal degeneration in mice lacking the myelin protein tetraspanin-2 Proteolipid protein is required for transport of sirtuin 2 into CNS myelin Oligodendrocytes: biology and pathology Animal Models of Cerebral Palsy: Hypoxic Brain Injury in the Newborn Axon-myelin interactions during a viral infection of the central nervous system Zika virus tropism and interactions in myelinating neural cell cultures: CNS cells and myelin are preferentially affected.Dicer ablation in oligodendrocytes provokes neuronal impairment in mice Heterogeneity in oligodendroglia: Is it relevant to mouse models and human disease?Myelin loss does not lead to axonal degeneration in a long-lived model of chronic demyelination The Olig family affects central nervous system development and disease Defects in mitochondrial axonal transport and membrane potential without increased reactive oxygen species production in a Drosophila model of Friedreich ataxia Axon-glial interaction in the CNS: what we have learned from mouse models of Pelizaeus-Merzbacher disease.Neuroinflammation by cytotoxic T-lymphocytes impairs retrograde axonal transport in an oligodendrocyte mutant mouse Proteolipid protein cannot replace P0 protein as the major structural protein of peripheral nervous system myelin.Axonal pathology precedes demyelination in a mouse model of X-linked demyelinating/type I Charcot-Marie Tooth neuropathy.Role of ERK1/2 MAPK signaling in the maintenance of myelin and axonal integrity in the adult CNS.Multivesicular bodies in neurons: distribution, protein content, and trafficking functions.Functional genomic and proteomic analysis reveals disruption of myelin-related genes and translation in a mouse model of early life neglect Maternal infection and white matter toxicity.Activated microglia mediate axoglial disruption that contributes to axonal injury in multiple sclerosis Myelination and axonal electrical activity modulate the distribution and motility of mitochondria at CNS nodes of Ranvier.Myelin basic protein-dependent plasma membrane reorganization in the formation of myelin Astroglial-axonal interactions during early stages of myelination in mixed cultures using in vitro and ex vivo imaging techniques T-cell-mediated disruption of the neuronal microtubule network: correlation with early reversible axonal dysfunction in acute experimental autoimmune encephalomyelitis.Differential expression of sPLA2 following spinal cord injury and a functional role for sPLA2-IIA in mediating oligodendrocyte death Glia Disease and Repair-Remyelination Modeling the natural history of Pelizaeus-Merzbacher disease.Demyelination and axonal preservation in a transgenic mouse model of Pelizaeus-Merzbacher disease.Evolution of a neuroprotective function of central nervous system myelin.Evidence for Cooperative Selection of Axons for Myelination by Adjacent Oligodendrocytes in the Optic Nerve.Stem cells, progenitors and myelin repair.Study of αB-crystallin expression in Gerbil BCAO model of transient global cerebral ischemia.NG2-expressing cells in the nervous system: role of the proteoglycan in migration and glial-neuron interaction.Diversity Matters: A Revised Guide to Myelination.

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Oligodendroglial modulation of fast axonal transport in a mouse model of hereditary spastic paraplegia.

http://www.wikidata.org/entity/Q36322059

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Donald Yool

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Ian D Duncan

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Ian R Griffiths

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James Garbern

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Jennifer A Barrie

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Jill H Fowler

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Klaus A Nave

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Mark McLaughlin

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Paul Montague

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P2860

Cloning and characterization of two cytoplasmic dynein intermediate chain genes in mouse and human

Unstable mutants in the peripheral endosomal membrane component ALS2 cause early-onset motor neuron disease

A kinesin heavy chain (KIF5A) mutation in hereditary spastic paraplegia (SPG10)

Charcot-Marie-Tooth disease type 2A caused by mutation in a microtubule motor KIF1Bbeta

Mutations in a newly identified GTPase gene cause autosomal dominant hereditary spastic paraplegia

Molecular composition of the node of Ranvier: identification of ankyrin-binding cell adhesion molecules neurofascin (mucin+/third FNIII domain-) and NrCAM at nodal axon segments

Paranodin, a glycoprotein of neuronal paranodal membranes

An isoform of ankyrin is localized at nodes of Ranvier in myelinated axons of central and peripheral nerves

Disruption of Cnp1 uncouples oligodendroglial functions in axonal support and myelination

Axonal degeneration in paraplegin-deficient mice is associated with abnormal mitochondria and impairment of axonal transport

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