A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel. - Wikidata - awgldk - TriplyDB

A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel.

A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel.

http://www.wikidata.org/entity/Q36323554

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Calcium antagonists for Duchenne muscular dystrophy International Union of Basic and Clinical Pharmacology. LXXVI. Current progress in the mammalian TRP ion channel family Mechanosensitive channel properties and membrane mechanics in mouse dystrophic myotubes Molecular candidates for cardiac stretch-activated ion channels TRP channels and analgesia Early pathogenesis of Duchenne muscular dystrophy modelled in patient-derived human induced pluripotent stem cells.Inhibitory control over Ca(2+) sparks via mechanosensitive channels is disrupted in dystrophin deficient muscle but restored by mini-dystrophin expression Cardiac Mechano-Gated Ion Channels and Arrhythmias.Tissue-specific expression of TRP channel genes in the mouse and its variation in three different mouse strains Critical role of TRPC1-mediated Ca²⁺ entry in decidualization of human endometrial stromal cells.TRPV2 is critical for the maintenance of cardiac structure and function in mice.Absence of γ-sarcoglycan alters the response of p70S6 kinase to mechanical perturbation in murine skeletal muscle.Our trails and trials in the subsarcolemmal cytoskeleton network and muscular dystrophy researches in the dystrophin era.Mechanosensitive channels in striated muscle and the cardiovascular system: not quite a stretch anymore.Role of TRP channels in the cardiovascular system.Understanding the cellular function of TRPV2 channel through generation of specific monoclonal antibodies.A multidisciplinary evaluation of the effectiveness of cyclosporine a in dystrophic mdx mice.TRP vanilloid 2 knock-out mice are susceptible to perinatal lethality but display normal thermal and mechanical nociception.Newly emerging Ca2+ entry channel molecules that regulate the vascular tone.Non-selective cationic channels of smooth muscle and the mammalian homologues of Drosophila TRP.Mutation of delta-sarcoglycan is associated with Ca(2+) -dependent vascular remodeling in the Syrian hamster.Probenecid: novel use as a non-injurious positive inotrope acting via cardiac TRPV2 stimulation Antiaging gene Klotho enhances glucose-induced insulin secretion by up-regulating plasma membrane levels of TRPV2 in MIN6 β-cells Enhanced Na+/H+ exchange activity contributes to the pathogenesis of muscular dystrophy via involvement of P2 receptors.Mechanisms of stretch-induced muscle damage in normal and dystrophic muscle: role of ionic changes.TRP channels in disease.Nerve Growth Factor Regulates Transient Receptor Potential Vanilloid 2 via Extracellular Signal-Regulated Kinase Signaling To Enhance Neurite Outgrowth in Developing Neurons Unusual localization and translocation of TRPV4 protein in cultured ventricular myocytes of the neonatal rat.Capsaicin, transient receptor potential (TRP) protein subfamilies and the particular relationship between capsaicin receptors and small primary sensory neurons.TRPpathies.The emerging role of TRP channels in mechanisms of temperature and pain sensation Axial stretch-dependent cation entry in dystrophic cardiomyopathy: Involvement of several TRPs channels Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophy Cancer cachexia causes skeletal muscle damage via transient receptor potential vanilloid 2-independent mechanisms, unlike muscular dystrophy ThermoTRP channels in nociceptors: taking a lead from capsaicin receptor TRPV1 What do we know about the transient receptor potential vanilloid 2 (TRPV2) ion channel?Activation of TRPV2 and BKCa channels by the LL-37 enantiomers stimulates calcium entry and migration of cancer cells.Mechanobiology in cardiac physiology and diseases.Novel role of transient receptor potential vanilloid 2 in the regulation of cardiac performance.Calcium and the damage pathways in muscular dystrophy.

P2860

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P2860

A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel.

http://www.wikidata.org/entity/Q36323554

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

A novel mechanism of myocyte d ...... owth factor-regulated channel.

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A novel mechanism of myocyte d ...... owth factor-regulated channel.

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type

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A novel mechanism of myocyte d ...... owth factor-regulated channel.

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A novel mechanism of myocyte d ...... owth factor-regulated channel.

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prefLabel

A novel mechanism of myocyte d ...... owth factor-regulated channel.

@ast

A novel mechanism of myocyte d ...... owth factor-regulated channel.

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A novel mechanism of myocyte d ...... owth factor-regulated channel.

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P2093

Kazuo Komamura

http://www.w3.org/2001/XMLSchema#string

Kunio Miyatake

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Munekazu Shigekawa

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Yuji Arai

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Yuki Katanosaka

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Yuko Iwata

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P2860

A capsaicin-receptor homologue with a high threshold for noxious heat

The capsaicin receptor: a heat-activated ion channel in the pain pathway

A new generation of Ca2+ indicators with greatly improved fluorescence properties

A novel topology model of the human Na(+)/H(+) exchanger isoform 1

The TRP channels, a remarkably functional family

The failing heart

Translocation of a calcium-permeable cation channel induced by insulin-like growth factor-I

Duchenne dystrophy: electron microscopic findings pointing to a basic or early abnormality in the plasma membrane of the muscle fiber.

Alteration in calcium handling at the subcellular level in mdx myotubes.

Insulin-like growth factor-1 enhances ventricular hypertrophy and function during the onset of experimental cardiac failure

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