JIL-1, a chromosomal kinase implicated in regulation of chromatin structure, associates with the male specific lethal (MSL) dosage compensation complex.
about
A human protein complex homologous to the Drosophila MSL complex is responsible for the majority of histone H4 acetylation at lysine 16Molecular basis for the discrimination of repressive methyl-lysine marks in histone H3 by Polycomb and HP1 chromodomainsInteraction of OKL38 and p53 in regulating mitochondrial structure and functionThe chromosomal proteins JIL-1 and Z4/Putzig regulate the telomeric chromatin in Drosophila melanogasterSU(VAR)3-7 links heterochromatin and dosage compensation in DrosophilaAn evolutionary consequence of dosage compensation on Drosophila melanogaster female X-chromatin structure?Phosphorylation of SU(VAR)3-9 by the chromosomal kinase JIL-1.Genome-wide analysis of regulation of gene expression and H3K9me2 distribution by JIL-1 kinase mediated histone H3S10 phosphorylation in DrosophilaHistone H3S10 phosphorylation by the JIL-1 kinase in pericentric heterochromatin and on the fourth chromosome creates a composite H3S10phK9me2 epigenetic mark.Modulation of ISWI function by site-specific histone acetylation.Global analysis of the relationship between JIL-1 kinase and transcription.Association and spreading of the Drosophila dosage compensation complex from a discrete roX1 chromatin entry site.The roX genes encode redundant male-specific lethal transcripts required for targeting of the MSL complexThe amino-terminal region of Drosophila MSL1 contains basic, glycine-rich, and leucine zipper-like motifs that promote X chromosome binding, self-association, and MSL2 binding, respectivelyInteraction study of the male specific lethal (MSL) complex and trans-acting dosage effects in metafemales of Drosophila melanogaster.JIL-1 kinase, a member of the male-specific lethal (MSL) complex, is necessary for proper dosage compensation of eye pigmentation in Drosophila.Mutations in the transcription elongation factor SPT5 disrupt a reporter for dosage compensation in DrosophilaGene expression analysis of the function of the male-specific lethal complex in Drosophila.Functional redundancy within roX1, a noncoding RNA involved in dosage compensation in Drosophila melanogaster.The Drosophila GAGA factor is required for dosage compensation in males and for the formation of the male-specific-lethal complex chromatin entry site at 12DE.X-chromosome-wide profiling of MSL-1 distribution and dosage compensation in Drosophila.Chromosome-wide gene-specific targeting of the Drosophila dosage compensation complex.Non-coding roX RNAs prevent the binding of the MSL-complex to heterochromatic regions.Evidence against a role for the JIL-1 kinase in H3S28 phosphorylation and 14-3-3 recruitment to active genes in Drosophila.Loss-of-function alleles of the JIL-1 kinase are strong suppressors of position effect variegation of the wm4 allele in Drosophila.Drosophila dosage compensation: males are from Mars, females are from VenusroX RNAs are required for increased expression of X-linked genes in Drosophila melanogaster males.Dosage compensation in Drosophila.The epigenetic H3S10 phosphorylation mark is required for counteracting heterochromatic spreading and gene silencing in Drosophila melanogasterThe right dose for every sexLifting a chromosome: dosage compensation in Drosophila melanogaster.Modulation of Heterochromatin by Male Specific Lethal Proteins and roX RNA in Drosophila melanogaster Males.Cumulative contributions of weak DNA determinants to targeting the Drosophila dosage compensation complex.Finding a balance: how diverse dosage compensation strategies modify histone h4 to regulate transcriptionCaenorhabditis elegans dosage compensation regulates histone H4 chromatin state on X chromosomesProximity ligation assays of protein and RNA interactions in the male-specific lethal complex on Drosophila melanogaster polytene chromosomes.Identification of chromatin-associated regulators of MSL complex targeting in Drosophila dosage compensation.Targeting dosage compensation to the X chromosome of Drosophila males.Pervasive and largely lineage-specific adaptive protein evolution in the dosage compensation complex of Drosophila melanogaster.Transcriptional adaptor ADA3 of Drosophila melanogaster is required for histone modification, position effect variegation, and transcription
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P2860
JIL-1, a chromosomal kinase implicated in regulation of chromatin structure, associates with the male specific lethal (MSL) dosage compensation complex.
description
2000 nî lūn-bûn
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2000年学术文章
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name
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@ast
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@en
type
label
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@ast
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@en
prefLabel
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@ast
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@en
P2093
P2860
P356
P1476
JIL-1, a chromosomal kinase im ...... ) dosage compensation complex.
@en
P2093
P2860
P304
P356
10.1083/JCB.149.5.1005
P407
P577
2000-05-01T00:00:00Z