Mitochondrial disease in mouse results in increased oxidative stress. - Wikidata - awgldk - TriplyDB

Mitochondrial disease in mouse results in increased oxidative stress.

Mitochondrial disease in mouse results in increased oxidative stress.

http://www.wikidata.org/entity/Q36331851

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Reactive oxygen generated by Nox1 triggers the angiogenic switch ARL2 and BART enter mitochondria and bind the adenine nucleotide transporter Twinkle and POLG defects enhance age-dependent accumulation of mutations in the control region of mtDNA Role of telomere dysfunction in cardiac failure in Duchenne muscular dystrophy Oxidative stress, mitochondrial dysfunction, and aging Redox Characterization of Functioning Skeletal Muscle Mitochondrial involvement and oxidative stress in temporal lobe epilepsy Sal1p, a calcium-dependent carrier protein that suppresses an essential cellular function associated With the Aac2 isoform of ADP/ATP translocase in Saccharomyces cerevisiae.Alternative antimicrobial approach: nano-antimicrobial materials Mitochondrial protein quality control: the mechanisms guarding mitochondrial health Overexpression of semicarbazide-sensitive amine oxidase in human myopathies Dual mutations reveal interactions between components of oxidative phosphorylation in Kluyveromyces lactis Toxicity of nanomaterials Effects of hydrogen peroxide on mitochondrial gene expression of intestinal epithelial cells Association of mitochondrial antioxidant enzymes with mitochondrial DNA as integral nucleoid constituents Mitochondrial oxidative stress in mice lacking the glutathione peroxidase-1 gene Recurrent De Novo Dominant Mutations in SLC25A4 Cause Severe Early-Onset Mitochondrial Disease and Loss of Mitochondrial DNA Copy Number A mitochondrial paradigm of metabolic and degenerative diseases, aging, and cancer: a dawn for evolutionary medicine Cardiac involvement in mitochondrial DNA disease: clinical spectrum, diagnosis, and management.Endothelial mitochondria--less respiration, more integration.Phenotypic rescue of a Drosophila model of mitochondrial ANT1 disease.Multiple-stress analysis for isolation of Drosophila longevity genes.Protective effect of selenium-enriched Lactobacillus on CCl4-induced liver injury in mice and its possible mechanisms.The role of mitochondrial DNA mutations in mammalian aging Oxygen toxicity and the health and survival of eukaryote cells: a new piece is added to the puzzle.Mitochondrial DNA haplogroup T is associated with coronary artery disease and diabetic retinopathy: a case control study.Neonatal exendin-4 treatment reduces oxidative stress and prevents hepatic insulin resistance in intrauterine growth-retarded rats.Adenine nucleotide translocase 1 deficiency results in dilated cardiomyopathy with defects in myocardial mechanics, histopathological alterations, and activation of apoptosis.Oxidative stress in early stage Bipolar Disorder and the association with response to lithium mtDNA mutations increase tumorigenicity in prostate cancer.Degree of glutathione deficiency and redox imbalance depend on subtype of mitochondrial disease and clinical status.Impact of Aging and Exercise on Mitochondrial Quality Control in Skeletal Muscle.Mitochondrial disorders of the nuclear genome The age-related accumulation of a mitochondrial DNA control region mutation in muscle, but not brain, detected by a sensitive PNA-directed PCR clamping based method.The neurogenic basic helix-loop-helix transcription factor NeuroD6 confers tolerance to oxidative stress by triggering an antioxidant response and sustaining the mitochondrial biomass.Catalytic antioxidants and neurodegeneration Early induction of oxidative stress in mouse model of Alzheimer disease with reduced mitochondrial superoxide dismutase activity.Mouse heat shock transcription factor 1 deficiency alters cardiac redox homeostasis and increases mitochondrial oxidative damage Muscle-specific loss of apoptosis-inducing factor leads to mitochondrial dysfunction, skeletal muscle atrophy, and dilated cardiomyopathy.Association of mitochondrial DNA variations with lung cancer risk in a Han Chinese population from southwestern China

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P2860

Mitochondrial disease in mouse results in increased oxidative stress.

http://www.wikidata.org/entity/Q36331851

description

1999 nî lūn-bûn

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1999年の論文

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年學術文章

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1999年學術文章

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1999年學術文章

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name

Mitochondrial disease in mouse results in increased oxidative stress.

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Mitochondrial disease in mouse results in increased oxidative stress.

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type

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Mitochondrial disease in mouse results in increased oxidative stress.

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Mitochondrial disease in mouse results in increased oxidative stress.

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prefLabel

Mitochondrial disease in mouse results in increased oxidative stress.

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Mitochondrial disease in mouse results in increased oxidative stress.

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Mitochondrial disease in mouse results in increased oxidative stress.

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P2093

A Panov

http://www.w3.org/2001/XMLSchema#string

B A Cottrell

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D C Wallace

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L A Esposito

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S Melov

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P2860

Radicals r'aging.

Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase

A novel neurological phenotype in mice lacking mitochondrial manganese superoxide dismutase

Mitochondrial disease in superoxide dismutase 2 mutant mice

The mitochondrial generation of hydrogen peroxide. General properties and effect of hyperbaric oxygen

Sequence and gene organization of mouse mitochondrial DNA

The Fenton reagents.

Multi-organ characterization of mitochondrial genomic rearrangements in ad libitum and caloric restricted mice show striking somatic mitochondrial DNA rearrangements with age

Mice with a homozygous null mutation for the most abundant glutathione peroxidase, Gpx1, show increased susceptibility to the oxidative stress-inducing agents paraquat and hydrogen peroxide.

Aconitase is a sensitive and critical target of oxygen poisoning in cultured mammalian cells and in rat lungs.

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4820-4825

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scholarly article

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10.1073/PNAS.96.9.4820

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9

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96

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13078801

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10220377

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21775

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