Translational inhibition of colonic epithelial heat shock proteins by IFN-gamma and TNF-alpha in intestinal inflammation.
about
Mitochondrial dysfunction in inflammatory bowel diseaseRole of endoplasmic reticulum stress and autophagy as interlinking pathways in the pathogenesis of inflammatory bowel diseaseIncreased susceptibility to dextran sulfate sodium-induced colitis in the endoplasmic reticulum stress transducer OASIS deficient miceHeat shock protein-derived T-cell epitopes contribute to autoimmune inflammation in pediatric Crohn's disease.Stress granule formation mediates the inhibition of colonic Hsp70 translation by interferon-gamma and tumor necrosis factor-alpha.Endoplasmic reticulum stress and oxidative stress in cell fate decision and human disease.Regional differences in colonic mucosa-associated microbiota determine the physiological expression of host heat shock proteins.Tumor necrosis factor alpha and inflammation disrupt the polarity complex in intestinal epithelial cells by a posttranslational mechanismHeat Shock Proteins: Intestinal Gatekeepers that Are Influenced by Dietary Components and the Gut MicrobiotaDifferential expression of HSPA1 and HSPA2 proteins in human tissues; tissue microarray-based immunohistochemical study.Correlation between depression, anxiety, and polymorphonuclear cells' resilience in ulcerative colitis: the mediating role of heat shock protein 70.High-dose green tea polyphenols induce nephrotoxicity in dextran sulfate sodium-induced colitis mice by down-regulation of antioxidant enzymes and heat-shock protein expressions.Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation.Gut epithelial inducible heat-shock proteins and their modulation by diet and the microbiota.Distinct and Synergistic Contributions of Epithelial Stress and Adaptive Immunity to Functions of Intraepithelial Killer Cells and Active Celiac Disease.Quantitative proteomics and bioinformatic analysis provide new insight into the dynamic response of porcine intestine to Salmonella Typhimurium.Loss of stress response as a consequence of viral infection: implications for disease and therapy.Dual Role of Endogenous Serotonin in 2,4,6-Trinitrobenzene Sulfonic Acid-Induced Colitis.The Helicobacter pylori cytotoxin CagA is essential for suppressing host heat shock protein expression.PKR protects colonic epithelium against colitis through the unfolded protein response and prosurvival signalingThe unfolded protein response and chemical chaperones reduce protein misfolding and colitis in mice.Novel mechanism of cytokine-induced disruption of epithelial barriers: Janus kinase and protein kinase D-dependent downregulation of junction protein expressionInflammation-induced, 3'UTR-dependent translational inhibition of Hsp70 mRNA impairs intestinal homeostasis.Inducible heat shock protein 70 prevents multifocal flat dysplastic lesions and invasive tumors in an inflammatory model of colon cancereIF2α phosphorylation controls thermal nociception.Glutamine enhances heat shock protein 70 expression via increased hexosamine biosynthetic pathway activity.Therapeutic treatment with a novel hypoxia-inducible factor hydroxylase inhibitor (TRC160334) ameliorates murine colitis.Integrated stress response-altered pro-inflammatory signals in mucosal immune-related cells.Gut microbiota imbalance and chaperoning system malfunction are central to ulcerative colitis pathogenesis and can be counteracted with specifically designed probiotics: a working hypothesis.Toll-like receptor regulation of intestinal development and inflammation in the pathogenesis of necrotizing enterocolitis.Endoplasmic reticulum stress in intestinal epithelial cell function and inflammatory bowel diseaseGut mucosal DAMPs in IBD: from mechanisms to therapeutic implications.Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.Disruption of the epithelial barrier during intestinal inflammation: Quest for new molecules and mechanisms.Endoplasmic Reticulum Stress and Oxidative Stress: A Vicious Nexus Implicated in Bowel Disease PathophysiologyTraffic control of bacteria-derived molecules: a new system of host-bacterial crosstalk.Enterococcus faecium NCIMB 10415 modulates epithelial integrity, heat shock protein, and proinflammatory cytokine response in intestinal cellsSchistosoma japonicum attenuates dextran sodium sulfate-induced colitis in mice via reduction of endoplasmic reticulum stress.Proteome profiling of spinal cord and dorsal root ganglia in rats with trinitrobenzene sulfonic acid-induced colitis.Do reciprocal interactions between cell stress proteins and cytokines create a new intra-/extra-cellular signalling nexus?
P2860
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P2860
Translational inhibition of colonic epithelial heat shock proteins by IFN-gamma and TNF-alpha in intestinal inflammation.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Translational inhibition of co ...... ha in intestinal inflammation.
@ast
Translational inhibition of co ...... ha in intestinal inflammation.
@en
type
label
Translational inhibition of co ...... ha in intestinal inflammation.
@ast
Translational inhibition of co ...... ha in intestinal inflammation.
@en
prefLabel
Translational inhibition of co ...... ha in intestinal inflammation.
@ast
Translational inhibition of co ...... ha in intestinal inflammation.
@en
P2093
P2860
P1433
P1476
Translational inhibition of co ...... ha in intestinal inflammation.
@en
P2093
Eugene B Chang
Lev Lichtenstein
Mae J Ciancio
Maor Lahav
Mark W Musch
Mikihiro Fujiya
Shrikant Anant
P2860
P304
P356
10.1053/J.GASTRO.2007.09.026
P407
P577
2007-09-25T00:00:00Z