Effects of increased systolic Ca(2+) and β-adrenergic stimulation on Ca(2+) transient decline in NOS1 knockout cardiac myocytes.
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Diesterified nitrone rescues nitroso-redox levels and increases myocyte contraction via increased SR Ca(2+) handlingNitric Oxide Synthase 1 Modulates Basal and β-Adrenergic-Stimulated Contractility by Rapid and Reversible Redox-Dependent S-Nitrosylation of the HeartModulation of myocardial contraction by peroxynitriteNeuronal nitric oxide synthase is indispensable for the cardiac adaptive effects of exercise.Tri-modal regulation of cardiac muscle relaxation; intracellular calcium decline, thin filament deactivation, and cross-bridge cycling kinetics.Designing proteins to combat disease: Cardiac troponin C as an example.Kcnj11 Ablation Is Associated With Increased Nitro-Oxidative Stress During Ischemia-Reperfusion Injury: Implications for Human Ischemic Cardiomyopathy.Moving into a new neighborhood: NOS goes nuclear.
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P2860
Effects of increased systolic Ca(2+) and β-adrenergic stimulation on Ca(2+) transient decline in NOS1 knockout cardiac myocytes.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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2012年學術文章
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name
Effects of increased systolic ...... OS1 knockout cardiac myocytes.
@ast
Effects of increased systolic ...... OS1 knockout cardiac myocytes.
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type
label
Effects of increased systolic ...... OS1 knockout cardiac myocytes.
@ast
Effects of increased systolic ...... OS1 knockout cardiac myocytes.
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prefLabel
Effects of increased systolic ...... OS1 knockout cardiac myocytes.
@ast
Effects of increased systolic ...... OS1 knockout cardiac myocytes.
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P2860
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Effects of increased systolic ...... OS1 knockout cardiac myocytes.
@en
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Brandon J Biesiadecki
Jonathan P Davis
Mark T Ziolo
Paul M L Janssen
Steve R Roof
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P304
P356
10.1016/J.NIOX.2012.08.077
P577
2012-08-30T00:00:00Z