Cardiac mast cell regulation of matrix metalloproteinase-related ventricular remodeling in chronic pressure or volume overload.
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Gender differences in non-ischemic myocardial remodeling: are they due to estrogen modulation of cardiac mast cells and/or membrane type 1 matrix metalloproteinaseRight ventricular failure and pathobiology in patients with congenital heart disease - implications for long-term follow-upMast cell inhibition attenuates myocardial damage, adverse remodeling, and dysfunction during fulminant myocarditis in the ratAdenosine A2A receptor activation reduces infarct size in the isolated, perfused mouse heart by inhibiting resident cardiac mast cell degranulation.Estrogen attenuates chronic volume overload induced structural and functional remodeling in male rat hearts.Extracellular matrix remodeling during the progression of volume overload-induced heart failure.Cardiac and renal function are progressively impaired with aging in Zucker diabetic fatty type II diabetic ratsCD117-positive cells of the heart: progenitor cells or mast cells?Temporal evaluation of cardiac myocyte hypertrophy and hyperplasia in male rats secondary to chronic volume overload.Preclinical research into basic mechanisms of radiation-induced heart disease.Structural and mechanical adaptations of right ventricle free wall myocardium to pressure overloadOpposing roles of PARP-1 in MMP-9 and TIMP-2 expression and mast cell degranulation in dyslipidemic dilated cardiomyopathy.Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient ratsPotential targets for intervention in radiation-induced heart disease.Lack of beta3 integrin signaling contributes to calpain-mediated myocardial cell loss in pressure-overloaded myocardiumEstrogenic modulation of inflammation-related genes in male rats following volume overloadResident cardiac mast cells: are they the major culprit in the pathogenesis of cardiac hypertrophy?Cardioprotective effects of lysyl oxidase inhibition against volume overload-induced extracellular matrix remodeling.Mast cells and epsilonPKC: a role in cardiac remodeling in hypertension-induced heart failure.The pathogenesis of cardiac fibrosis.Nampt secreted from cardiomyocytes promotes development of cardiac hypertrophy and adverse ventricular remodeling.ETA selective receptor antagonism prevents ventricular remodeling in volume-overloaded rats.Remodeling of the guinea pig intrinsic cardiac plexus with chronic pressure overload.Calpain inhibition preserves myocardial structure and function following myocardial infarction.Progressive right ventricular functional and structural changes in a mouse model of pulmonary arterial hypertension.Merits of non-invasive rat models of left ventricular heart failure.Mechanisms of cardiac radiation injury and potential preventive approaches.Efficacy of antihistamines on mortality in patients receiving maintenance hemodialysis: an observational study using propensity score matching.Hypertension-induced remodelling: on the interactions of cardiac risk factors.Estrogen receptor antagonism exacerbates cardiac structural and functional remodeling in female rats.Exposure to chronic alcohol accelerates development of wall stress and eccentric remodeling in rats with volume overload.The kallikrein-kinin system in experimental Chagas disease: a paradigm to investigate the impact of inflammatory edema on GPCR-mediated pathways of host cell invasion by Trypanosoma cruzi.Defects in ryanodine receptor function are associated with systolic dysfunction in rats subjected to volume overload.High-salt intake induces cardiomyocyte hypertrophy in rats in response to local angiotensin II type 1 receptor activation.Potential markers and metabolic processes involved in the mechanism of radiation-induced heart injury.Defective intracellular Ca2+ homeostasis contributes to myocyte dysfunction during ventricular remodelling induced by chronic volume overload in rats.Effects of nonselective endothelin-1 receptor antagonism on cardiac mast cell-mediated ventricular remodeling in rats.The Non-cardiomyocyte Cells of the Heart. Their Possible Roles in Exercise-Induced Cardiac Regeneration and Remodeling.Inhibition of matrix metalloproteinase activity by ACE inhibitors prevents left ventricular remodeling in a rat model of heart failure.Long-term MMP inhibition by doxycycline exerts divergent effect on ventricular extracellular matrix deposition and systolic performance in stroke-prone spontaneously hypertensive rats.
P2860
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P2860
Cardiac mast cell regulation of matrix metalloproteinase-related ventricular remodeling in chronic pressure or volume overload.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Cardiac mast cell regulation o ...... c pressure or volume overload.
@ast
Cardiac mast cell regulation o ...... c pressure or volume overload.
@en
type
label
Cardiac mast cell regulation o ...... c pressure or volume overload.
@ast
Cardiac mast cell regulation o ...... c pressure or volume overload.
@en
prefLabel
Cardiac mast cell regulation o ...... c pressure or volume overload.
@ast
Cardiac mast cell regulation o ...... c pressure or volume overload.
@en
P2093
P1476
Cardiac mast cell regulation o ...... c pressure or volume overload.
@en
P2093
Amanda L Chancey
David B Murray
Gregory L Brower
James A Stewart
Jason D Gardner
Joseph S Janicki
Mary F Forman
P304
P356
10.1016/J.CARDIORES.2005.10.020
P577
2005-12-22T00:00:00Z