Presynaptic Deletion of GIT Proteins Results in Increased Synaptic Strength at a Mammalian Central Synapse.
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Molecular Machines Determining the Fate of Endocytosed Synaptic Vesicles in Nerve TerminalsMolecular Machines Regulating the Release Probability of Synaptic Vesicles at the Active ZoneSuperpriming of synaptic vesicles as a common basis for intersynapse variability and modulation of synaptic strength.A novel region in the CaV2.1 α1 subunit C-terminus regulates fast synaptic vesicle fusion and vesicle docking at the mammalian presynaptic active zone.Age-related defects in short-term plasticity are reversed by acetyl-L-carnitine at the mouse calyx of Held.GIT1 regulates synaptic structural plasticity underlying learning.
P2860
Presynaptic Deletion of GIT Proteins Results in Increased Synaptic Strength at a Mammalian Central Synapse.
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name
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@ast
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@en
type
label
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@ast
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@en
prefLabel
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@ast
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@en
P2093
P2860
P50
P1433
P1476
Presynaptic Deletion of GIT Pr ...... t a Mammalian Central Synapse.
@en
P2093
Debbie Guerrero-Given
Mónica S Montesinos
Naomi Kamasawa
Rachel Satterfield
Robert Schmalzigaug
P2860
P304
P356
10.1016/J.NEURON.2015.10.042
P407
P577
2015-12-01T00:00:00Z