Contraction stimulates muscle glucose uptake independent of atypical PKC.

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Contraction stimulates muscle glucose uptake independent of atypical PKC.

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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Contraction stimulates muscle glucose uptake independent of atypical PKC.

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Contraction stimulates muscle glucose uptake independent of atypical PKC.

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Item

label

Contraction stimulates muscle glucose uptake independent of atypical PKC.

@ast

Contraction stimulates muscle glucose uptake independent of atypical PKC.

@en

prefLabel

Contraction stimulates muscle glucose uptake independent of atypical PKC.

@ast

Contraction stimulates muscle glucose uptake independent of atypical PKC.

@en

P1476

Contraction stimulates muscle glucose uptake independent of atypical PKC

@en

P2093

Ding An

http://www.w3.org/2001/XMLSchema#string

Haiyan Yu

http://www.w3.org/2001/XMLSchema#string

Laurie J Goodyear

http://www.w3.org/2001/XMLSchema#string

Michael F Hirshman

http://www.w3.org/2001/XMLSchema#string

Michael Leitges

http://www.w3.org/2001/XMLSchema#string

Nobuharu L Fujii

http://www.w3.org/2001/XMLSchema#string

Robert V Farese

http://www.w3.org/2001/XMLSchema#string

Taro Toyoda

http://www.w3.org/2001/XMLSchema#string

P2860

Role of AMP-activated protein kinase in mechanism of metformin action

Skeletal muscle-selective knockout of LKB1 increases insulin sensitivity, improves glucose homeostasis, and decreases TRB3

A role for AMP-activated protein kinase in contraction- and hypoxia-regulated glucose transport in skeletal muscle

Dissociation of AMPK activity and ACCbeta phosphorylation in human muscle during prolonged exercise

Restoring systemic GDF11 levels reverses age-related dysfunction in mouse skeletal muscle

Regulation of dishevelled and beta-catenin in rat skeletal muscle: an alternative exercise-induced GSK-3beta signaling pathway

Atypical PKC: a target for treating insulin-resistant disorders of obesity, the metabolic syndrome and type 2 diabetes mellitus.

Disconnecting mitochondrial content from respiratory chain capacity in PGC-1-deficient skeletal muscle

AMPK alpha1 activation is required for stimulation of glucose uptake by twitch contraction, but not by H2O2, in mouse skeletal muscle

AICAR and metformin, but not exercise, increase muscle glucose transport through AMPK-, ERK-, and PDK1-dependent activation of atypical PKC.

P31

scholarly article

P356

10.14814/PHY2.12565

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Joram D Mul

P577

2015-11-01T00:00:00Z

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P5875

283792313

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P698

26564060

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P921

glycobiology

P932

4673624

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